Interactive Transcript
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On to the next case.
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So this is a 49-year-old woman who presented with infertility.
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Again, I start with my sagittal T2 non fat saturated images.
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Anterior compartments looking okay.
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In my middle compartment, my uterus is anteverted but retroflexed.
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So my brain is already telling me that there's something going on.
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There's this big mass back here that is involving both the middle compartment
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and tethering back to the posterior compartment back here.
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So we're going to need to dive into that.
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If I look at my axials here,
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so we have one ovary back here on the left.
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We have our right ovary back here.
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They're kind of displaced.
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They're very posterior in location and they're kind of medialized.
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So that's going to be something important for us to discuss as well.
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Then I switch and pull up my T1 fat saturated images
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next to the T2s, so that I can get a sense of both
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the T1 and the T2 character of things at the same time.
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So these
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T2 hyperintense lesions that I'm seeing in both ovaries are more
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T1 hyperintense than they are T2 hyperintense.
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So these are some bilateral endometriomas.
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This area of abnormality along the posterior uterine serosa
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is predominantly T2 and T1 hypointense.
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Although, I'm also seeing areas of T1 hyperintensity
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within it and little cystic areas of T2 hyperintensity within it.
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That's going to tell us what type
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of morphology of deep infiltrating endometriosis we're dealing with.
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I apologize if this is getting a little grainy as I scroll through too quickly.
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And then we're also seeing this kind
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of very T2 dark stellate scarring sort of appearance.
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Okay, so let's put this all together.
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And we'll do so with the sagittal images.
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So here along the posterior uterine serosa,
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at the location of what's called the torus uterinus, which is the junction
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of the uterine body and this uterine cervix along the posterior serosa.
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So right here. It's called the torus uterinus.
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There is a big endometriotic implant,
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and it is both active glandular and chronic stromal fibrotic in morphology.
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So the active glandular part are these
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T1 hyperintense glands that are hemorrhagic and the cystic spaces.
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And the chronic stromal fibrotic component is the T2 dark stellate
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component that is tethering the rectum to the uterus,
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obliterating the rectouterine space here,
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and then extending along both uterosacral ligaments.
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So the uterosacral ligaments extend
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from the torus uterinus on both sides, posteriorly and laterally
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and insert along the sacrum
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on both sides, and help to suspend the uterus and the pelvis.
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If we look at the axial T2s, we can appreciate the morphology
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of that chronic stromal fibrotic component much better.
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And we can see from the torus uterinus, which is right here,
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there's extension of T2 hypointensity, posterolaterally on both sides.
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That's involvement of the very proximal uterosacral ligament.
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Right.
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So I think that was everything that I wanted to point out on this case.
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So we can go to the next question, please.
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So which of the features of this case
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that we've talked about has the biggest impact on surgical planning?
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Is it the presence of both active glandular and chronic stromal fibrotic morphologies?
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The Rectouterine space obliteration?
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Retroflexion of the uterus or the bilateral endometriomas?
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Perfect. Everybody got it right.
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It is rectouterine space obliteration.
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And again, that's really important
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to the surgeons because they do these surgeries laparoscopically and if
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the space is scarred down, they literally cannot get their scope beyond it.
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The active glandular and chronic stromal fibrotic morphologies,
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the presence of endometriomas,
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the retroflexion of the uterus,
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all of those are just findings of that endometriosis,
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but they don't have specific implications for the surgery itself.
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