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Wk 3, Case 2 - Review

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Next is a 24 year old patient with

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medial ankle pain following trauma.

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On T1 weighted images, there is a focal

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osteochondral defect in the posterior

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medial aspect of the talar dome.

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The defect is outlined by marrow edema on

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corresponding fluid sensitive sequences with focal

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non shoulder indentation of the subchondral plate.

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At the donor site, we see a focal area of ossific

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signal intensity that is located within the

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defect corresponding to an osteochondral fragment.

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Sagittal images allow us to see the extent,

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the anteroposterior extent of this osteocondral defect.

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Visualize to a better extent a posterior

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migrated fragment in the posterior ankle

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joint recess seen here on T1 weighted images.

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So, if we use the classification of

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osteochondral lesions of the talar dome,

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this would be a fragment that is completely

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detached from the donor side, and it's also

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displaced into the posterior ankle gutter.

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Stage four of osteochondral lesion.

Report

Patient History
24-year-old man complaining of left ankle pain after lifting

Findings
SKELETAL/BONES:
Large chronic left medial talar osteochondral defect, measuring 2.9 cm AP by 1.1 cm transverse by 0.9 cm deep. Osseous remodeling with moderate-to-marked flattening of the affected talar dome. Stress-related adjacent osteoedema throughout the talar dome. No T2 hyperintense fluid cleft to suggest potentially unstable fragment. No displaced osteochondral body identified.

Scuffing of the posterior aspect of the talar dome, with an adjacent small to moderate-sized os trigonum. Low-grade osteoedema through the os trigonum. Prominent synovitis throughout the posterior tibiotalar recess.

Moderate talofibular arthrosis with stress-related osteoedema of the distal fibula and lateral talus and a small subcortical pseudocyst of the distal fibula.

No further pattern of reactive osteoedema. No micro- or macro-trabecular fracture. No aggressive osseous abnormality.

Incidental pes planus deformity noted.

ARTICULATIONS:
Tibiotalar joint/talar dome: Large osteochondral defect/erosion of the talar dome as described above. Moderate secondary tibiotalar arthrosis with spurring of the anterior tibial plafond and and anterior talar dome. Prominent diffuse capsulitis with prominent posterior tibiotalar synovitis, extending out to the adjacent large osteochondral defect.

Ankle mortise/syndesmosis: The ankle mortise is in anatomic alignment. No syndesmosis widening.

Chopart joint: Unremarkable.

Midfoot/hindfoot: No fracture or injury of the anterior calcaneal process. No prominent midfoot or hindfoot arthrosis.

LIGAMENTS:
High ankle: Remote high ankle sprain with thickened, scarred anterior tibiofibular ligament. Intact interosseous ligament and posterior tibiofibular ligament. No syndesmotic diastasis.

Low ankle: Intact.

Subtalar/Chopart: Attenuated cervical and interosseous ligaments. Spring ligaments intact and unremarkable.

TENDONS:
Intact.

GENERAL:
Sinus tarsi: Unremarkable.

Muscles: No traumatic muscle injury. No volumetric muscle atrophy.

Soft tissue: Unremarkable.

Plantar fascia: Intact.

Neurovascular complex/tarsal tunnel: No evidence of entrapment neuropathy.

Loose bodies: Tiny mm sized mineralized fragments/osseous bodies are seen scattered through the posterior tibiotalar synovitis.

Impressions
1. Large chronic “NON-SHOULDERED” medial talar dome osteochondral defect/erosion with flattening of the articular surface and moderate subjacent stress reaction. No unstable or displaced osteochondral fragment.
2. Moderate secondary tibiotalar arthrosis with active capsulitis. Posterior tibiotalar synovitis with tiny metaplastic mineralized bodies.
3. Moderate talofibular arthrosis.

Case Discussion

Faculty

Stephen J Pomeranz, MD

Chief Medical Officer, ProScan Imaging. Founder, MRI Online

ProScan Imaging

Jenny T Bencardino, MD

Vice-Chair, Academic Affairs Department of Radiology

Montefiore Radiology

Edward Smitaman, MD

Clinical Associate Professor

University of California San Diego

Tags

Musculoskeletal (MSK)

MRI

Foot & Ankle

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