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Wk 5, Case 1 - Review

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This is a 51-year-old complaining

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of Achilles pain for two weeks.

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So when I hear Achilles, the first thing

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I do is I pull down all my sagittals.

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So that's what we're going to do.

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Sagittal number one, sagittal number two.

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I think I've only got two.

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So let's pull down those first two.

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And I can usually figure out in the

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Achilles what's going on from just this.

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Now there is another interesting finding here.

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The patient's 51 and has

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somehow escaped into adulthood.

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with a fibrocartilaginous coalition.

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Why is this a coalition as opposed

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to just simply the Chopart joint?

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And the answer is it's too broad.

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In other words, the snout of the anterior

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process of the calcaneus normally is pointed.

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This one is rectangular.

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And then you see the cartilaginous interface

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between the anterior process and the navicular.

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Now, some of these patients, we don't

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have the resolution to tell, will have

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deficiency of the bifurcate ligament

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as part of a congenital abnormality.

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Those that have such a deficiency will often have

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More pronounced arthrosis of the Chopart joint.

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So by inference, the fact that there is so

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little arthritis of the Chopart joint, which

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is the talonavicular and calcaneal cuboid

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articulation tells you that you probably do

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not have displaces or absence of the ligaments.

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As you keep scrolling you see just

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how irregular though that coalition

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is there's some fragmentation.

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On the navicular side of the articulation.

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You also see a little swelling of the subtalar

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lateral sinus Tarsi with the extensive retinaculum

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and that is a side finding to the case.

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Now let's turn our attention.

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Back to the Achilles.

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So get out your pen.

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Let's go down the Achilles checklist.

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First question.

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Gender.

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It's a man.

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How do I ask that?

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But women wear high heels.

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They're more likely not exclusively, but

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they're more likely to have a pump bump or a

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Haglund deformity, which is found right here.

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Posterior superior.

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It looks like this,

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but a piece of bone.

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This patient doesn't have it.

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What if it was a man?

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In a man, in certain settings, I might

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consider something like reactive arthritis,

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formerly known as Reiter's syndrome.

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I might also consider gout.

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We had one last week, gouty

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infiltration of the Achilles tendon.

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So, that colors my judgment

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and my differential diagnosis.

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And I look at the age. This patient's 51.

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So I'm not thinking about,

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you know, congenital things.

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I'm thinking about acquired things.

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I'm thinking about overuse syndromes.

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And the next thing I do is I look

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at the shape of the calcaneus.

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I look for that pump bumper Haglund deformity.

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I check Kager's fat space.

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These are all part of my Achilles checklist.

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And there's a little bit of scarring there,

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nothing too dramatic in Kager's fat space.

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And then I look at the Achilles.

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I first start out up high, I look at the size of

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the muscles, I look at the myotendinous junction.

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And by the way, I do this very quickly,

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within a matter of 15 to 30 seconds.

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Then I work my way down and I look for focal

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architectural distortion of the fibers,

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which are normally parallel fibers that are

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hard to see unless the Achilles is inflamed.

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Okay.

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But they should run in parallel.

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There should be no focal high signal

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intensity defects, not even little ones.

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And I really don't see any defects so far.

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Now I do see something that is defect-like

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right here that's more focal, but when I look at

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the T1, it's got the signal intensity of bone.

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So I've got some heterotopic bone that

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is forming in the superficial Achilles.

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And the next thing I do is

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I look at the Achilles size.

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I'll allow the Achilles to be

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8 to 10 millimeters A to P.

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This one's well under 8 millimeters.

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Next on my checklist is the Supra Calcaneal Bursa.

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And this one has a tiny amount of fluid,

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nothing that I would even comment on.

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It's too small to even bother with.

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The next thing I do is I look

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at the footplate or footprint.

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This is the footprint of the Achilles.

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It has a high attachment, a mid

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attachment, and a low attachment.

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There's the low attachment right here.

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So you got to look all the way up and down and

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make sure that the Achilles is not delaminated.

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I look for concealed interstitial under surface

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delaminations, which would occur right here, here.

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here and they might or might not insert some

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insight, some periosteitis or marrow swelling.

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Now I don't have these little small

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under surface concealed tears, but I

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do have reactive bone marrow swelling

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right there on the water-weighted image.

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Hard to see on the T1, but it is there.

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It's real.

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It's not a coil artifact right there.

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So that is an indicator that

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the patient has periosteitis

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from traction from the Achilles.

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But if this was a man, and it is, I might

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also consider the diagnosis of reactive

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arthritis, formerly known as Reiter's syndrome.

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The next thing I do is I look at the

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peritenon, the outer covering of the Achilles.

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Now I can't see the peritenon, but I want

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to see fat circumscribing the Achilles.

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And I don't see that in this locus right here.

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Move it around a little bit.

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Not seeing good fat right there.

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Remember, that's where we saw that piece of bone.

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I look at the superficial aspect of the Achilles.

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So what do you call that?

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A superficial tear or fraying of

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the Achilles with heterotopic bone.

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Now, let's say there was a tear.

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Let's say there was a tear right here.

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I would tell the clinician in my report

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that the tear begins two centimeters

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above the superior calcaneal protuberance.

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The tear is two centimeters long.

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The tear is not full depth.

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And then I would go on to look at the

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axial and I would decide off the axial.

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What percent from side to side.

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If there were a tear here, let's say

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there was a tear that looked like this.

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I would say there's a 25 percent tear

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in the middle portion of the tendon.

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I would also give a depth.

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I would say less than 25, 25 to 50, or

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greater than 50 percent depth or full depth.

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And then I would also describe

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whether it has involvement of the

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lateral bundle or the medial bundle.

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That is pretty much the

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checklist for Achilles pathology.

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I don't use the coronal all that much.

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They did provide a heavily

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water-weighted coronal sequence.

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And sometimes you can see tears a

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little better here than on the other

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sequences, but most of the time not.

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And you can see that the Achilles itself,

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these wispy linear areas right here.

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They're not normal.

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They are a sign of intra-Achilles inflammation.

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They're parallel to one another.

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That's interstitial swelling between

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the tendon fibrils of the Achilles.

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So the diagnosis in this case would be

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Achilles tendinopathy, chronic, um, but active

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because of the swelling with heterotopic

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bone and a superficial fraying-type tear.

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Of the Achilles along its inferior footprint.

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That would be number one.

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Number two would be incidentally

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noted fibrocartilaginous coalition of

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the calcaneonavicular articulation.

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And if you wanted to, in the body, the

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report, you could throw in this, this

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chronic erosion that is seen in the cuboid.

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I do not use parallel pitch lines.

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Now I understand to diagnose Hagelin deformity.

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Here's what I do.

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I draw.

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I do use.

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I don't ever draw a line.

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I do very little measuring and drawing.

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I will draw a line straight across the

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calcaneus, the superior ridge of the calcaneus.

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And if the superior calcaneal protuberance

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projects more than 8 millimeters above that

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line, I will call it a Hagelin deformity.

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Again, I don't measure it.

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I usually just look.

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People that typically have a symptomatic

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Hagelin deformity will have swelling around it.

Report

Patient History
51-year-old man complaining of Achilles pain for 2 weeks with no inciting event
Findings

SKELETAL/BONES:
Prominent/dysplastic anterior calcaneal process with cortical irregularity and joint space loss at the calcaneonavicular articulation, consistent with a nonosseous calcaneonavicular coalition. Minor subcortical cystic change adjacent to the calcaneal aspect of the coalition. Nominal osteoedema surrounding the coalition.
Concomitant nonosseous talocalcaneal coalition of the middle facet (with joint space loss, cortical irregularity and prominence of the middle subtalar facet). Moderate surrounding stress related osteoedema and subcortical cystic change.
Ill-defined osteoedema surrounding an ill-defined linear area of low signal that contacts the cortex of the posterolateral calcaneus, consistent with a microtrabecular stress fracture.
Mild dorsal calcaneal enthesophyte formation with low-grade osteoedema and periostitis.
No further micro- or macro-trabecular fracture or stress fracture identified. No further pattern of reactive osteoedema. No aggressive osseous abnormality.

ARTICULATIONS:
Tibiotalar joint/talar dome: No osteochondral defect of the talar dome or tibial plafond.
Ankle mortise/syndesmosis: The ankle mortise is in anatomic alignment. No syndesmosis widening.
Chopart joint: Nonosseous Calcaneonavicular and talocalcaneal coalitions as described above. Moderate stress-related osteoedema and fibrocystic change surrounding the talocalcaneal coalition. Moderate posterior subtalar facet osteoarthrosis with osteophytosis and joint space loss.
Midfoot/hindfoot: No fracture or injury of the anterior calcaneal process. No prominent midfoot or hindfoot arthrosis.

LIGAMENTS:
High ankle: Intact.
Low ankle: Intact. Small chronic intrasubstance partial-thickness cystic tear distal ATFL.
Subtalar/Chopart: Intact.

TENDONS:
Intact. Mild peritendinous high-signal edema superficial to the distal Achilles insertion, consistent with insertional enthesitis.

GENERAL:
Sinus tarsi: Unremarkable.
Muscles: No traumatic muscle injury. No volumetric muscle atrophy.
Soft tissue: Unremarkable.
Plantar fascia: Intact.
Neurovascular complex/tarsal tunnel: Unremarkable. No evidence of entrapment neuropathy.
Intra-articular/loose bodies: None.

Impressions
1. Nonosseous talocalcaneal coalition (involving middle facet). Moderate surrounding reactive osteoedema and subcortical degenerative cystic change.
2. Nonosseous calcaneonavicular coalition. Nominal surrounding osteoedema and subcortical cystic change.
3. Microtrabecular stress fracture posterolateral calcaneus (likely due to altered biomechanics of weight-bearing related to the above coalitions).
4. Mild insertional Achilles enthesitis.

Case Discussion

Faculty

Stephen J Pomeranz, MD

Chief Medical Officer, ProScan Imaging. Founder, MRI Online

ProScan Imaging

Jenny T Bencardino, MD

Vice-Chair, Academic Affairs Department of Radiology

Montefiore Radiology

Edward Smitaman, MD

Clinical Associate Professor

University of California San Diego

Tags

Musculoskeletal (MSK)

MRI

Foot & Ankle

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