Interactive Transcript
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This is a 14-year-old girl.
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This case is dramatically different from
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the one I showed you, which was extremely
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subtle, in which the diagnosis of complex
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regional pain syndrome was raised as
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a possibility along with apophysitis.
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It turned out not to be the case.
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There's another patient with a, with a
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dysplastic calcaneonavicular articulation.
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The snout is too broad, and that might
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be something that you would latch onto.
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this person, the interpreter of
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this case did and also said that the
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patient had multiple stress injuries.
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Let's scroll it.
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It's a case I never want you to forget.
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It's the most important case in the deck.
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Now, children run around a lot.
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And so I don't mind if they have this
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sort of patchy dotted pattern of edema.
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What do I call that?
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I call that overused syndrome or high
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bone turnover syndrome of the juvenile.
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A lot of them complain of vague amorphous pain.
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And I dismiss them 99 percent of the time.
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But when don't I dismiss them?
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When I see a focal area of linearity.
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Like this that looks a little bit like atrial
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fibrillation, then I start to, you know, then then
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I call it a stress fraction or it's coalesced in
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one spot, but I start to get really nervous when
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I see a demon and a child that borders the cortex.
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That favors the cortex and if you zoom out, look
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at how marbly looking the marrow is and look at
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where, why would you get a stress injury here?
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You don't put any stress on the dorsal navicular.
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That would be a really crazy odd place.
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So you have to use good old
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fashioned Aussie common sense.
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Why would you get a stress injury here?
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Very bizarre.
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Oh, this is complex regional
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pain syndrome in a child.
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And the T1 findings are usually
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nondescript or nonexistent.
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They're very minimal.
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Uh, we've got a coronal T1, you
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can see it, it almost looks normal.
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You can barely see the pattern of
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maroedema, and most of the time
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they don't have soft tissue changes.
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There is a soft tissue Pseudoect form
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of neurogenic reaction or neurovascular
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reaction, um, but that is uncommon.
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Most of the time, it starts out in
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the skeleton, and this patient has it.
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The T2 is also unimpressive.
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So without the proton density fat suppression,
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suppression image, TE50, you would, you
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would miss the diagnosis completely.
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Look at this subcortical edema
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here in the back of the calcaneus.
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That is a really weird
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pattern for stress phenomenon.
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Doesn't make any sense.
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She's also got an effusion.
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Now, what are some other causes of juxta
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articular juxtacortical edema was one
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major one that is inflammatory arthritis,
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but we can totally rule that out.
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One reason why we can partially rule
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it out is there's no sign of itis.
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But the reason we can totally
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rule it out is right here.
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There's no synovium on the
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dorsum of the navicular.
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That would be a weird place to have a
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synovial inflammatory skeletal reaction.
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You get it here where the joint is.
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It's non-articular.
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You know, you can even look
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here, not articular or here.
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So the fact that you have nonarticular peripheral
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window framing subcortical subperiosteal edema
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in a juvenile with an effusion, the diagnosis
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of complex regional pain syndrome is clinched
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and the patient needs a lumbar block and
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physical therapy, moving the extremity, right?
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As much as possible and Children who
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have this are frequently hospitalized so
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that they can get this movement returned.
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Uh, and they don't end up with a frozen
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foot again in the axial projection.
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Look at the peripheral nature
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of the edema right there.
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That's real.
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That's real.
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So is that.
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So is that.
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So is that there isn't a great
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correlation between the MRI and the
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and and the improvement of the patient.
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I know this well because someone in my family
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suffered from this condition for over a year.
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So I studied it extensively.
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And there is not a good correlation between the
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regression of edema and how the patient feels.
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But I'll tell you this, when they're treated
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and they get a lumbar block, their pain
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instantly decreases and the vasculature to
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the extremity changes within 20 seconds.
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You can see the foot actually
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go from blue to pink.
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So the patient knows that they're getting
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better and they'll be able to tell you the
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clinician, you know, my, my symptoms are
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coming back or my symptoms are improving.
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So we manage them in terms of symptoms.
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We don't typically use radiography unless the
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patient has taken a negative turn clinically.
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to decide whether they're getting better or not.
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You will know, and so will the clinician.
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What non-contrast features do you
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look for to diagnose synovitis?
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Um, one, juxta-articular edema.
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Not juxta-cortical, juxta-articular.
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Two, joint space narrowing.
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Three, if it's inflammatory,
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auto-digestion of cartilage.
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The cartilage is too thin.
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Four, Erosions.
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Those erosions could be, let's pull up a coronal.
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If we have one, we do.
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Those erosions could be central,
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marginal, or peripheral and para-articular.
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You might see such a weird erosion in gout.
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Number five, the capsule.
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It's not just distended.
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It's floppy.
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It's redundant.
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It doesn't look like it's just filled with fluid.
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It looks like it's elastic.
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You'll get capsules that look like this next.
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Within the capsule, you see synovial
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hypertrophy, which some people have
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described as little rice bodies.
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If those bodies get too big,
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you got to worry about synovial
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chondromatosis or osteochondromatosis.
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If they get really big, you got
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to worry about PVNS or localized
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giant cell tumor of tendon sheath.
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Pannus in the soft tissues, rheumatoid
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nodules, polyarticular involvement.
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symmetry where both extremities
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are involved, soft tissue swelling.
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Those are the features I look
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for to diagnose synovitis.
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