Interactive Transcript
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There's a 20-year-old male with
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"syndesmosis widening".
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And so, unfortunately, the history is a
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dead giveaway, but that's the real history.
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Let's evaluate the way I would normally
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evaluate if I was sitting by myself,
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and I put up the sagittals, usually hand
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in hand, fat-weighted, water-weighted.
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And I focus mostly, initially, on the water
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weighted image because it's like a bone scan.
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We're looking for hot spots.
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We've got an effusion.
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And in that effusion is a glob.
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The glob is not sharply defined or
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dark enough to represent the body.
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That glob, in a young person with
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an injury history, should tell
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you you got blood in the joint.
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So you're immediately suspicious
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that something serious has happened.
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Then as we scroll back and forth, the
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next thing that should strike you is this
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edema in the posterior malleolar region.
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And now, game over.
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Now you know you've got a high ankle
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injury, and this is known as Volkman's sign.
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So now your job is to assess the
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type and nature of the ankle injury.
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Are there other fractures?
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But clearly, what type of
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ligamentous injury do you have?
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And as soon as you see this Volkmann's
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sign, you know you have at least a
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component of a high ankle injury.
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Now that may be accompanied by a low
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ankle injury, but almost certainly, with
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virtually 99 to 100 percent certainty,
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you've got an element of a high ankle injury.
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Let's keep scrolling and see if there
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are any other ancillary findings.
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And the answer is, not really.
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You know, there are no other
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fractures to really tip you off.
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And what types of fractures am I looking for?
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I'm looking for fractures of the anterior
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fibula, or fractures of the anterior tibia,
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where the other syndesmotic ligaments are
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found. This area is known as the Chaput,
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tubercle, and this area is known on
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the fibular side as the Wagstaff tubercle.
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So if we had avulsion fragments or edema
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in these areas, this would further lead
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us into the realm of high ankle injuries.
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But this is the most reliable, and this one
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already tells you you've got a high ankle problem.
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So my next maneuver is to go to the axials.
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Now, I want to point out that the way this
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exam is done is in neutral positioning.
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So it wasn't specifically done with
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the foot plantar flex to look for
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the tendons, and that's appropriate.
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Although, you have to be aware that your
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tendon analysis is going to be a little
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bit hampered by magic angle effect because
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the tendon is going to curve a little more.
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But that's not necessarily
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why we're doing this study.
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We're doing it to assess the complex severity
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and complications of an injury to the ankle.
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So now let's look axially and let's start
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out low where the tail is kind of looks
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like an egg or it looks like an egg.
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You're right in the heart of the anterior
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talofibular ligament, which is the most
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common ligament to tear in a low ankle sprain.
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And it is clearly not normal.
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It's too gray.
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It's too fat.
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It's even gray on the water-weighted image.
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Is it ruptured?
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No.
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Is it sliced and retracted?
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No.
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Is the posterior talofibular ligament torn?
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No, it never tears.
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You have to get hit by a truck and
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dislocate your ankle to tear it.
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It doesn't tear.
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Okay, so we know we've had a low ankle injury.
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Let's keep scrolling.
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We'll get up a little higher into
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the higher fibers of the low ankle
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of the anterior talofibular ligament.
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And they still look a little rugged,
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especially as we get towards the Wagstaff
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tubercle, the anterior tubercle of the fibula.
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Still very, very swollen
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with lots of edema and fluid.
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Now, let's keep working our way up higher.
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And now we're really in the heart of the Wagstaff
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tubercle where the syndesmosis, the anterior
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tib fib ligament, has begun, right there.
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And where is the connection between the high
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ankle bones, between the talus and the fibula?
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It's not present.
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It's filled with shredded ligament and blood.
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That's all we see.
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Normally, we should see a
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very discreet line here now.
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It looks something like this.
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Easy to spot.
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Let's keep going up higher.
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Let's keep working our way up higher.
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We get up higher, and there is a
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little bit of interosseous tissue here.
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Some of that may be related to some
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crossing vessels or some residual ligament.
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But the higher we go, the more swollen
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it gets, and the wider it gets.
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Now, normally, this space up higher
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should be about 2 millimeters.
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I'm not going to measure it right now, but
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it's clearly more than two millimeters,
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but the measurement doesn't really matter.
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Because you're able to directly visualize
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this area, as I'll show you coronally in a
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minute, and see that the interosseous is torn.
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Now, the tears of the high ankle
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occur in an intercalary fashion.
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In other words, they occur
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from anterior to posterior.
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So you can't tear the posterior
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without tearing the anterior.
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So the anterior tib fib is torn.
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They're interosseous.
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Is torn and swollen.
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There again is our Volkmann sign or
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our Volkmann fracture in the back.
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And then the posterior tib fib ligament,
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which should also be a nice line.
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It ends as a little stub.
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So all three components of the high ankle kaput,
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not shepherd kaput, they're gone.
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Volkmann sign.
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Anterior tib fib tear, interosseous
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tear, posterior tib fib tear.
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Now let's go back to the low ankle so we can
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complete our responsibilities in this case.
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Let's go down low.
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We've already said that there is an injury
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to the anterior talofibular ligament, the
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most important component of the low ankle.
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It's swollen, transected.
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So there's been a low ankle sprain.
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And remember, this ligament is like a sheet.
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It has length from top to bottom.
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So you can tear the lower fibers,
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or the upper fibers, or all of them.
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But in a high ankle injury, it's generally
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the upper fibers that are injured, and the
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lower fibers tend to look a little better.
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Let's see if that's the
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case here as we go down low.
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Not really.
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The lower fibers are swollen.
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The upper fibers are swollen.
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So the whole anterior talofibular
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ligament has taken a hit.
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But it's not transected and retracted.
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So your next question is, "Okay, well
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what about the calcaneofibular ligament?"
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Well, here's a little tip.
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You can't tear the calcaneofibular ligament
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without tearing the anterior talofibular ligament.
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So if you're having trouble seeing it, you
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might as well just assume that it's intact.
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In other words, if you can't see the
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calcaneofibular ligament, don't say it's torn.
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Say that it's poorly visualized due to
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technical limitations, but likely intact.
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Well, let's see if we can spot it.
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It forms the floor of the peroneus tunnel.
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There it is right there.
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It comes off the calcaneus and
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it goes towards the fibula.
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And it's pretty hard to follow
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it all the way to the fibula.
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Here's the fibula.
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Let's see if we can spot it.
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That's probably as far as we can
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get it, but it's pretty straight.
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It's pretty delicate.
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And if we go to the coronal projection
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now, there's our coronal, T1.
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Here's our coronal water weighted image.
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There is the origin of the CFL.
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That's pretty cool.
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And it's nice and straight and it continues
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to be straight and admittedly I cannot see
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it go on to the fibula as I move posteriorly.
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But there's no swelling there, so I'm just going
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to assume, because the anterior talofibular
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ligament is not ruptured, that it's intact.
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And indeed that is how the case was read.
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Now, oh, there's more.
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You are probably wondering, well, you know that,
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that tib fib area is kind of hard to see axially.
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Yeah, it's swollen.
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Yes, it's fuzzy.
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Yes, it's widened.
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But can I actually see the interosseous
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membrane or the interosseous ligament?
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You sure can.
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There it is.
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It's big.
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It's fat.
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It's bloody.
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It's swollen.
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It's gray.
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It's not black.
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There's edema tracking up
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between the tib and the fib.
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So what if this is widened?
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Okay, it's widened.
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It shouldn't be widened.
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But you now have MRI to directly see the ligament.
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Now, if you just looked at the
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most narrow space on a plain film,
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especially if you were able to look axially, that
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space would be no more than 1 to 2 millimeters.
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This is certainly more than 1 to 2
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millimeters, more like 5 millimeters.
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Now, on the plain film, you were always
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taught that the tibia and fibula overlap
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at the ankle mortis, at, just above the
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ankle mortis, by about 6 millimeters.
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So that's a, a cardinal plain film sign
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you might use for a, for a core exam.
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But with MR, you don't really need
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that anymore, because you have
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direct insight into the ligaments.
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A couple of other things that just are,
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are relevant to conventional radiography.
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There's a little clear space here between the
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plafond and the medial malleolus and the talus.
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That's usually about a millimeter.
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You don't really need to measure that, although
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you can see in this patient with a serious
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ankle sprain that the plafond looks excellent.
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There's no widening here, and
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there's no osteochondral defect.
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Another important aspect of your responsibility.
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In this high ankle sprain case.
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What are some other measurements you might use?
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Well, sometimes on plain film, we look at
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the horizontal axis of the dome of the talus.
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And then we look at the tib
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fib angle relative to that.
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You'd have to bring this one
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over a little bit, say to here.
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Then we'd measure this angle.
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And we'd see if there's any tibio talar
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alteration in the relationship of this angle
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and the relationship of the tibia to the fibula.
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Is the fibula elongated?
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Is the tibia retracted because of
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the loss of interosseous stability?
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That's probably passé for MRI, but
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that's a plain radiographic measurement.
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Finally, the mechanism of injury.
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How does this happen?
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Usually the patient is pushing off.
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The foot is in slight plantar flexion.
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Now in the typical ankle sprain,
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you're not quite as plantar flexed.
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Usually in a neutral position.
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And it's a simple inversion.
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Or you might be slightly plantarflexed,
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and it's a simple inversion.
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The more dorsiflexed you are, the
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more likely you are to have fractures.
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The more plantarflexed you are, the more
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likely you are to have ligamentous injuries.
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The more plantarflexed you are,
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and the more external rotatory.
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In other words, you're plantarflexed,
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your foot's in the turf, and your foot's
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And then you turn, so that there's
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external rotation on the foot and ankle.
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That's when you see high ankle sprains.
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This, an intercalary, three part, high ankle
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sprain, a tif, anterior tib fib ligament,
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interosseous, right here, and posterior
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tib fib ligament, all torn, along with a
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sprain with swelling and bleeding, into the
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low ankle, anterior talofibular ligament.
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