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Labyrinthitis, Secondary to Otomastoiditis

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There are different causes of labyrinthitis.

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The most common actually is secondary

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spread from a middle ear infection.

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I'm showing you the post-contrast fat suppression scan of

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a patient who is being evaluated for

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hearing loss on the left side.

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We immediately see that there is opacification and

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enhancement of the mastoid air cells on the

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left side, and also into the middle ear cavity.

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On the post-gadolinium enhanced scans,

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where I'd like to stop is on this image

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and demonstrate the pathology as it relates to

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the inner ear spread of middle ear infection.

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I have to admit that at 3 Tesla,

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evaluating the cochlear and vestibule and semicircular

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canals for enhancement is a little bit problematic

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because they naturally look a little bit brighter

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than the adjacent soft tissues.

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So if we look on the right side,

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we have the cochlear basal turn, we have the vestibule,

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we have the lateral semicircular canal.

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So vestibule, cochlea, lateral semicircular canal.

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And here is the apical turn of the cochlea.

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Although there is slight hyperintensity to the basal

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turn of the cochlea and the vestibule

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and the lateral semicircular canal,

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those are normal phenomena on a post-gad three

1:26

tesla fat-suppressed scan. However,

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if we look on the contralateral side,

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I think that you can see pretty strikingly that the

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basal turn of the cochlea on the left side is enhancing

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much more so than its contralateral normal and the

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anatomic structure. So let's just erase some of this

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so it's a little easier to see the areas of enhancement.

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Not only that,

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but you can also see that the vestibule

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is also showing abnormal enhancement compared

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to the contralateral vestibule.

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So let me erase everything so you can sort of look at

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this on your own. And hopefully, you can see that there is

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abnormal enhancement here,

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an abnormal enhancement here.

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Here's the lateral semicircular canal on the left side,

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and that is secondary to spread of this infection from

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the middle ear structures to the inner ear structures.

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And this is what we would call labyrinthine enhancement,

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secondary to labyrinthitis,

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secondary to otomastoiditis.

Report

Description

Faculty

David M Yousem, MD, MBA

Professor of Radiology, Vice Chairman and Associate Dean

Johns Hopkins University

Tags

Temporal bone

Non-infectious Inflammatory

Neuroradiology

MRI

Infectious

Head and Neck

Brain

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