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Wk 1, Case 2 - Review

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25-year-old with right shoulder pain and

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decreased range of motion for seven years.

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History of labral repair eight years ago.

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So a young and at 25 years of age.

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Okay, this time it's a little bit intimidating.

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All the images that we have, we have

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pre-contrast and post-contrast imaging.

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And, um, let's take a look at what we've got.

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I think we'll start with the axial this time.

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So the first thing I look at on the axial, and when I have

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a lot of images like this I usually will start with the

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axial just to get my feel for the shape of things, you know,

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does the humerus not only does it fit into the glenoid.

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And these are very simple but valuable things that

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you guys can extrapolate from CT and from radiography.

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How's the humeral head sitting in the cup.

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In other words, is the center of the humeral head right here,

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centered with the center of the glenoid, and it's primo.

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It's perfect.

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So then we scroll up and down, and I look at the shape.

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This happens to be a gradient echo, not particularly, um,

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effective for tendons, but I look at the tendons anyway.

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Um, why isn't it effective for tendons?

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It produces a lot of gray signal

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due to magic angle effect or the 55.

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4-degree artifact or the anisotropic

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phenomenon artifact with a short TE.

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So, but but I do use it for cartilage, and I

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can see the hyaline cartilage pretty well.

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In fact, this little gray slit here, you see cartilage

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on one side capsule collapsed, and then cartilage on the

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other side so you can really get into the joint space.

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joint mice, loose bodies.

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This is really the role.

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Articular surface evaluation for the GRE.

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What else does the GRE, uh, help you recognize?

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Well, first, what is, what, what does a GRE look like?

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Well, the muscles appear kind of shiny.

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The cartilage, and if there's fluid, is

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white, and the bones are typically gray.

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case all the fat will be black.

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So that's one way to identify a GRE.

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R.

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E.

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Another way to identify a GRE.

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R.

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E.

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Is it says T2 star T2 asterisk and then it says G.

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E.

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Not for general electric for gradient echo.

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So that's another easy way to figure out it's a GRE.

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What else does G.

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R.

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E.

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Do it shows you calcium It shows you blood.

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It shows you ossification.

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It shows you metal.

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So let's scroll up and down and see if we have

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any blood ossification or metal or even iron.

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And we don't until we do.

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I'm gonna give you five seconds to identify it right there.

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There's something up there that doesn't belong.

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So right away, we haven't even looked at the case yet,

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and we have come up with a diagnosis in a 25-year-old.

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Now you might say, well, how do you have a diagnosis?

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Well, is it a hematoma?

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Well, why would it be a hematoma

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in a 25-year-old with that history?

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Plus there's no soft tissue swelling.

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Ossification, I suppose it's possible,

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but pretty rare to ossify spontaneously.

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Um, you know, you can get conditions that give you

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ossification, um, like heterotopic bone, often from trauma.

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So that wouldn't make a lot of sense.

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And, um, you're pretty much left with it being

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something common, so-called calcific tendonitis, which is

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really known as hydroxyapatite deposition disease or HADD.

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Now I don't want you to go off half-cocked and call

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this pseudo gout because this is not pseudo gout.

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This is just trophic calcification from impingement.

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And how do you get this kind of impingement.

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Swimming.

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When you're stroking, when you're overhead and you're

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in the finishing position of either freestyle or

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butterfly, you are in a position where the anterior

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shoulder comes in contact with the anterior acromion.

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Let's keep going, shall we?

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I have one coronal T1 and you can see somebody went to

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great lengths in this study, none of which was necessary.

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I don't do a lot of arthrography I do about five of them a day.

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That's five out of maybe 100.

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And the main reason I do it is to appease the clinician.

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I don't need it.

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So here's your T1.

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I'm going to put up a sagittal non-contrast.

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T2.

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So the T2 looks a lot like the GRE that you saw earlier, but

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the muscles are a little duller and the bones are also duller.

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And you can tell by looking at the parameters,

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the TR is very long and the TE is 60 or above.

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Whereas the TE in a gradient echo is very short and then

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you look up here and it says T2 FSE with fat suppression.

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So now let's scroll around and see if we can spot our

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calcific tendonitis or hydroxyapatite deposition and we can.

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It's right there on the coronal T1 and

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right there on the sagittal, sagittal T2.

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So Now, some of you are wondering why this

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coronal T1 doesn't look like this coronal T1.

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And that's because many people will choose on their

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arthrographically injected studies to fat suppress.

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So this is a fat-suppressed T1-weighted

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image, which makes the fluid look white.

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It also brings forth hydroxyapatite deposition.

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I like to perform a pre-injection

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image and a post-injection image.

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The reason this is only a post

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injection image is economics and time.

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When you get really sophisticated and you've done a lot of

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these, you can probably omit the pre-contrast injection.

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I've been doing this a long time.

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And the reason that I still hold on to that is because after you

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inject, you cannot tell what is swollen and what is not swollen.

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So if you have 10 or 15 different findings, And

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you're trying to figure out what is the critical

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finding, doing a post-arthrographic injection

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only can obscure where the real swelling is.

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So now, another comment that was made by one of

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your colleagues, is that there was loss of bulk of

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the anterior inferior labrum let's check that out.

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we've got an answer, an appropriate

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answer in a swimmer and a 25-year-old.

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And I don't mind if the anterior labrum is a little flat.

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That's a normal variation.

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The labrum can be flat, the labrum can

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be round, the labrum can be pointed.

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So this is not evidentiary of any type of labral pathology.

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You can see the inferior glenohumeral

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ligament right on top of it.

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Beautifully.

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And as we get down low, it becomes

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the labral ligamentous complex.

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Yes, there is a paucity of labral tissue down low.

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That is normal when you get into the axillary space.

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As you come up, it gets strong in the anterior quadrant.

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Look to the right here.

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You can see where you are in the humerus.

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You're in the inferior quadrant.

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And now you're about at the equator.

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Maybe a little bit above the equator.

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Um, actually you're at the equator and

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still a strong labrum still very strong.

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And then as you get up high sulcus, and

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then you're back to the superior labor.

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Now another way to tell that that is not

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pathologic is no contrast is going into the labor.

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None whatsoever.

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There might be a little scar right here, but

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that's probably all you could say about the labrum.

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Let's look at the labrum coronally.

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As you come forward, you got a pretty big sulcus in the front.

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And as you go back, I would say at the sex a little

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bit so I would not object to someone calling

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this some minor fraying of the superior labrum.

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And some of you might be saying well how do you know,

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there are exceptional times when the only answer is

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going to be to look in there and see if there is.

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hemorrhage or inflammation.

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Otherwise, it can be very hard to tell if you are in the mid

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coronal plane and you still see this little bit of fissuring.

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Now, I'll tell you that in swimmers, it's very common to see this

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kind of minor fissuring along the undersurface of the labrum.

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And as long as there isn't clicking and you

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have another answer, then you can play it down.

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This patient did not have arthroscopy for this.

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She had, um, barbitage for her calcific

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tendonitis or had, and she did just fine.

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She was asymptomatic thereafter.

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Um, let's see the rest of the comments on this case.

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Moderate AC joint arthrosis.

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That is way too strong.

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I will say that no person over the

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age of 30 has a normal AC joint.

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It just wasn't meant to last that long.

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But I think for the patient's age and their

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activity level, this is a normal AC joint.

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There's nothing wrong with it whatsoever.

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And somebody read subacromial bursitis.

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We'll have to look at our water-weighted image for that.

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Here's the sagittal water-weighted image.

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There is a little bit of fluid there, so I, I

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wouldn't dispute that there's some minor bursal

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swelling right around the coracoacromial ligament.

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So I'm okay with that particular statement.

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This is just normal wear and tear.

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I don't really get excited

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about the AC joint unless the patient has erosions, capsular

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distension, edema of bone, or it's the only thing on

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the image that explains the patient's clinical syndrome.

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For a repetitive overhead athlete, this is a normal AC joint.

Report

Patient History
25 year-old female with right shoulder pain and decreased range of motion for 7 years. History of labral repair 8 years prior.

Findings
ROTATOR CUFF: Mild supraspinatus and infraspinatus tendinosis without macrotear. Normal subscapularis.

SUBACROMIAL/SUBDELTOID BURSA: Subacromial-subdeltoid peritendinitis surrounding a 1.3 cm signal hypointensity at the anterodistal supraspinatus tendon compatible with calcific tendinitis or hydroxyapatite deposition disease-so called HADD.

MUSCLES (ROTATOR CUFF/DELTOID, TRAPEZIUS, PECTORALIS): No muscle atrophy or tear.

BICEPS TENDON: Normal long head biceps tendon without dislocation or tear.

AC JOINT: Mild acromioclavicular arthropathy without separation or medial arch stenosis.

CORACOCLAVICULAR LIGAMENTS: Normal coracoclavicular ligaments.

SUBACROMIAL ARCH/OUTLET: No lateral arch stenosis.

SUBCORACOID ARCH: No subcoracoid stenosis.

GLENOHUMERAL JOINT: Normal glenohumeral joint without adhesive capsulitis, erosion, or spur.

GLENOID LABRUM: Normal glenoid labrum without tear or cyst.

BONES: Normal without contusion or fracture.

SUBCUTANEOUS SOFT TISSUES: Normal without soft tissue swelling or mass.

AXILLA: Normal without lymphadenopathy or vascular abnormality.

Impressions
1. Calcific tendinitis or hydroxyapatite deposition disease-HADD at the bursal distal surface of the far anterior supraspinatus, surrounded by mild peritendinitis and mild underlying tendinosis, no rotator cuff tear. No internal derangement.
2. Mild acromioclavicular joint arthropathy.

Case Discussion

Faculty

Stephen J Pomeranz, MD

Chief Medical Officer, ProScan Imaging. Founder, MRI Online

ProScan Imaging

Jenny T Bencardino, MD

Vice-Chair, Academic Affairs Department of Radiology

Montefiore Radiology

Edward Smitaman, MD

Clinical Associate Professor

University of California San Diego

Tags

Shoulder

Musculoskeletal (MSK)

MRI

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