Interactive Transcript
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Okay, the next patient is a 56-year-old man.
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He's got right shoulder pain
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and decreased range of motion.
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All right, so let's go three up.
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We have an axial GRE on the left.
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Remember, this is your joint cartilage fibrocartilage
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hyaline cartilage sequence.
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Great for finding calcium,
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great for finding blood, great for finding metal.
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Let's throw up a T1 spin echo
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anatomic fat-weighted image.
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And we'll do a little scrolling of that.
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And let's put up the T2-weighted image.
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Shortly,
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we will substitute the heavily water-weighted
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sequences for the axial gradient echo image.
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So right now we have a T1 scrolling together with
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a T2. Why don't you say a high-grade tear
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of the supraspinatus and Terry's?
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All right,
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the supraspinatus does have a fair amount of
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intermediate signal intensity
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along its undercarriage.
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This area right here is known
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as the rotator cuff table.
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It's a little hard to appreciate because it's
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all gray. It's this condensed, thick,
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dark area right here.
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I'm going to draw over it,
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and I'm going to take my little yellow.
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Mark away, and you'll see it right there.
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Right there. And that area is infiltrated.
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There's some of the cable right here.
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So this is diffusely thickened tendinotic rotator
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cuff. And let's scroll back and forth on it.
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And there's a little bit of linear signal here
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and there, little bits and knits of signal,
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really too small
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to put much stock in.
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And some of you reasonably might say, well,
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I happen to know, Dr.
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Pomerance,
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that T2 is not that effective in picking up
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rotator cuff tears unless they're acute.
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If they're chronic,
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the rotator cuff tear may fill in with scar,
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which is dark, and old blood and hemosiderin,
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which is dark,
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and make the rotator cuff look normal.
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So let's take a look at the proton
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density fat suppression sequence,
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which is the most sensitive rotator cuff sequence.
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Right. Before we do that,
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let's just scroll our gradient
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echo before we dispose of it.
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And I think you can all appreciate some
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irregularity of the posterior capsule,
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some narrowing of the anterior joint space.
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And granted, this patient did move a little bit.
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The biceps sits a little bit in a medial position.
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And the relationship,
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the actual conformity of a round structure
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fitting perfectly in a cup.
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In other words, here's.
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Your cup
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is a little off.
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It looks like the humeral head is doing this.
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It's kind of lurching back a little bit.
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Let me take that away.
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And here's the lurching right there.
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So we're seeing the humeral head dislocate
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posteriorly, and the labrum is detaching,
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and the periosteum is detaching.
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That's the periosteum.
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It should be right on there.
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And the capsule is very irregular.
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So this patient has what we call posterior
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glenoid deficiency syndrome, which is a.
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Common accompaniment of osteoarthritis.
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Now, there are some big spurs here.
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And why is this important?
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Here's some more labor tearing.
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This is important because the amount of glenoid
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cup deformity and insufficiency is going to
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dictate whether you put in a standard arthroplasty
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or reverse hemiarthroplasty or arthroplasty.
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And this patient's posterior glenoid
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does look insufficient.
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If you actually look at the slope of the glenoid,
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it slopes back a little bit.
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Now, normally, the glenoid,
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if you draw a line along the spine,
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the glenoid should make a line that's perfectly
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perpendicular to that line of the scapular spine.
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That is not the case here.
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There's an angle right here,
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and this is known as the retroversion angle.
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So the patient has glenoid retroversion.
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You don't have to measure it necessarily.
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Has glenoid retroversion,
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has a posterior laboral tear,
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has posterior capsuler disease,
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has posterior glenoid deficiency syndrome as
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a manifestation of chronic Oa with
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large hypertrophic spurring.
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And now let's put up our proton
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density fat suppression image.
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Oops.
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It's not very proton density ish.
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It's certainly fat suppressed, though.
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But I like the Te to be around 40.
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Let's see if the sagittal has a better t.
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It doesn't. So it's a little T2 ish for my taste,
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but still the fat suppression is here and not on
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the T1 So it is a much more sensitive sequence
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for looking at the rotator cuff tear.
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In fact, it's pretty good.
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You can see there is a little tear here.
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There's a little bone penetration,
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which I refer to as rim rent.
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Bone penetration.
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The term rim rent has sort of fallen
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into disfavor, so I still use it.
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But if you want to say there's
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a small under surface,
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humeral sided rotator cuff tear with a little bit
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of bone penetration, that would be reasonable.
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The rest of the cuff demonstrates generalized
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swelling and hypertrophy and what we
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would call generalized tendinosis.
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We don't use the term tendinitis anymore.
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There's a little bit of fluid in the subacromial,
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subdeltoid space.
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There's a little bit of fluid in the joint.
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Clinicians like to know that.
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And you can see how these penetrating cysts and
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pseudocysts are quite large in this humeral head.
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Now,
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this rotator cuff tear has really not a
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lot of significance unless it's large.
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clinicians absolutely like to know
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when they're doing a repair,
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whether the rotator cuff is intact or not.
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But most of these degenerated shoulders have
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small tears, as this patient does.
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And for the most part the.
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Cuff is still there other than this focal defect.
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Now, why do I say it's not as important?
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Because this patient isn't moving their arm.
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This patient's arm is somewhat frozen.
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They can't pick their arm up beyond
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the 90 degree position.
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And the reason is they've got this goat beard
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deformity. So when they try and pick their arm up,
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the goat beard deformity slams into the labrum.
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Look what it's doing to the glenoid.
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It's hollowing out the glenoid.
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So this osteoarthropathy is producing decreased
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range of motion, and in some ways, it.
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Has protected
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the rotator cuff from further.
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Insult by restricting the patient's motion.
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I don't know if that's an evolutionary thing
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or not, but it's rather interesting.
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We go to the sagittal projection and
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we see a few other findings.
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We see some signal back here in the infraspinatus,
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which suggests that there's a strain back here,
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probably on a more chronic basis.
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And then we also see the region of our rotator
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cuff tear. Once again, small humeral sided.
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Notice I didn't say that it was articular sided
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because there's no articular surface here.
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There's no synovium here.
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There's no highland cartilage here.
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That's an articular surface.
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That's known as the bare area of the articular
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surface where there's no cartilage,
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that is the rest of the articular surface.
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This is a humeral sided,
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concealed delamination tear with
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some bony penetration.
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That is not the main finding in this case.
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The main finding in this case is osteoarthritis,
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hypertrophic,
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with a large goat beard deformity and moderate
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posterior glenoid deficiency syndrome.
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So some of you said high grade rotator
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cuff tear of the supraspinatus.
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I would not call this high grade.
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I'd call it a diminutive concealed,
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interstitial delamination footprint tear that is
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humeral sided. That would go in my report, body.
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And in the conclusion,
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I would say the rotator cuff is mostly intact,
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diffusely tendonotic and described
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as a diminutive tear.
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I would mention in the bile report that there
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is subachromial, subdeltoid fluid.
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And one of you said,
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as a secondary or tertiary finding that
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there is gross DJD of the AC joint.
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And I would say that is absolutely true.
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Very few people don't have DJD of the AC joint.
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How advanced is it? Well, for a 56 year old man,
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I think this is more than I would expect.
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But I try to grade these by how much hypertrophy
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and also how much signal there is on the T2
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weighted image. So there is some signal,
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high signal on the T2 weighted image.
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So I would say that there's mild to modest active
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inflammation of this moderately
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hypertrophic AC joint.
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And I would agree that that is a
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secondary or tertiary finding.
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Some of you said adhesive capsulitis in a man.
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Well,
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I usually don't commingle adhesive capsulitis in
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somebody who can't move their shoulder from OA.
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So it's probably an irrelevant statement
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and not a true statement.
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Some of you said concealed minor tearing of the
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supraspinatus at the insertion is favored
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to be chronic and degenerative,
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and that is absolutely true.
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And there were a few other correct comments
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that were made. The superior labrum,
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amazingly looks pretty good.
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And the reason is can't pick their arm up, right.
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So when you can't pick your arm up,
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you don't get much bouncing behavior of
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the humeral head against the glenoid.
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So the glenoid superior anatomy is preserved.
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And you can see the biceps take off right there.
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Here's the SGHL coming straight at you.
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Here's the coracohumeral ligament right there.
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And there is your tendon of the supraspinatus.
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One, two, three, and four.
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Four layers of hypointensity on the fossa.
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