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Wk 1, Case 5 - Review

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Okay, the next patient is a 56-year-old man.

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He's got right shoulder pain

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and decreased range of motion.

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All right, so let's go three up.

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We have an axial GRE on the left.

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Remember, this is your joint cartilage fibrocartilage

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hyaline cartilage sequence.

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Great for finding calcium,

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great for finding blood, great for finding metal.

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Let's throw up a T1 spin echo

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anatomic fat-weighted image.

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And we'll do a little scrolling of that.

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And let's put up the T2-weighted image.

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Shortly,

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we will substitute the heavily water-weighted

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sequences for the axial gradient echo image.

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So right now we have a T1 scrolling together with

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a T2. Why don't you say a high-grade tear

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of the supraspinatus and Terry's?

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All right,

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the supraspinatus does have a fair amount of

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intermediate signal intensity

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along its undercarriage.

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This area right here is known

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as the rotator cuff table.

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It's a little hard to appreciate because it's

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all gray. It's this condensed, thick,

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dark area right here.

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I'm going to draw over it,

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and I'm going to take my little yellow.

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Mark away, and you'll see it right there.

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Right there. And that area is infiltrated.

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There's some of the cable right here.

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So this is diffusely thickened tendinotic rotator

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cuff. And let's scroll back and forth on it.

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And there's a little bit of linear signal here

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and there, little bits and knits of signal,

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really too small

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to put much stock in.

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And some of you reasonably might say, well,

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I happen to know, Dr.

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Pomerance,

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that T2 is not that effective in picking up

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rotator cuff tears unless they're acute.

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If they're chronic,

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the rotator cuff tear may fill in with scar,

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which is dark, and old blood and hemosiderin,

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which is dark,

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and make the rotator cuff look normal.

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So let's take a look at the proton

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density fat suppression sequence,

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which is the most sensitive rotator cuff sequence.

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Right. Before we do that,

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let's just scroll our gradient

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echo before we dispose of it.

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And I think you can all appreciate some

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irregularity of the posterior capsule,

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some narrowing of the anterior joint space.

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And granted, this patient did move a little bit.

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The biceps sits a little bit in a medial position.

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And the relationship,

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the actual conformity of a round structure

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fitting perfectly in a cup.

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In other words, here's.

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Your cup

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is a little off.

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It looks like the humeral head is doing this.

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It's kind of lurching back a little bit.

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Let me take that away.

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And here's the lurching right there.

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So we're seeing the humeral head dislocate

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posteriorly, and the labrum is detaching,

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and the periosteum is detaching.

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That's the periosteum.

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It should be right on there.

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And the capsule is very irregular.

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So this patient has what we call posterior

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glenoid deficiency syndrome, which is a.

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Common accompaniment of osteoarthritis.

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Now, there are some big spurs here.

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And why is this important?

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Here's some more labor tearing.

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This is important because the amount of glenoid

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cup deformity and insufficiency is going to

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dictate whether you put in a standard arthroplasty

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or reverse hemiarthroplasty or arthroplasty.

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And this patient's posterior glenoid

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does look insufficient.

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If you actually look at the slope of the glenoid,

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it slopes back a little bit.

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Now, normally, the glenoid,

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if you draw a line along the spine,

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the glenoid should make a line that's perfectly

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perpendicular to that line of the scapular spine.

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That is not the case here.

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There's an angle right here,

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and this is known as the retroversion angle.

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So the patient has glenoid retroversion.

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You don't have to measure it necessarily.

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Has glenoid retroversion,

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has a posterior laboral tear,

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has posterior capsuler disease,

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has posterior glenoid deficiency syndrome as

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a manifestation of chronic Oa with

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large hypertrophic spurring.

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And now let's put up our proton

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density fat suppression image.

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Oops.

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It's not very proton density ish.

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It's certainly fat suppressed, though.

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But I like the Te to be around 40.

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Let's see if the sagittal has a better t.

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It doesn't. So it's a little T2 ish for my taste,

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but still the fat suppression is here and not on

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the T1 So it is a much more sensitive sequence

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for looking at the rotator cuff tear.

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In fact, it's pretty good.

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You can see there is a little tear here.

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There's a little bone penetration,

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which I refer to as rim rent.

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Bone penetration.

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The term rim rent has sort of fallen

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into disfavor, so I still use it.

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But if you want to say there's

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a small under surface,

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humeral sided rotator cuff tear with a little bit

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of bone penetration, that would be reasonable.

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The rest of the cuff demonstrates generalized

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swelling and hypertrophy and what we

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would call generalized tendinosis.

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We don't use the term tendinitis anymore.

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There's a little bit of fluid in the subacromial,

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subdeltoid space.

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There's a little bit of fluid in the joint.

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Clinicians like to know that.

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And you can see how these penetrating cysts and

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pseudocysts are quite large in this humeral head.

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Now,

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this rotator cuff tear has really not a

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lot of significance unless it's large.

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clinicians absolutely like to know

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when they're doing a repair,

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whether the rotator cuff is intact or not.

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But most of these degenerated shoulders have

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small tears, as this patient does.

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And for the most part the.

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Cuff is still there other than this focal defect.

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Now, why do I say it's not as important?

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Because this patient isn't moving their arm.

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This patient's arm is somewhat frozen.

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They can't pick their arm up beyond

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the 90 degree position.

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And the reason is they've got this goat beard

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deformity. So when they try and pick their arm up,

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the goat beard deformity slams into the labrum.

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Look what it's doing to the glenoid.

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It's hollowing out the glenoid.

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So this osteoarthropathy is producing decreased

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range of motion, and in some ways, it.

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Has protected

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the rotator cuff from further.

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Insult by restricting the patient's motion.

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I don't know if that's an evolutionary thing

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or not, but it's rather interesting.

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We go to the sagittal projection and

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we see a few other findings.

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We see some signal back here in the infraspinatus,

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which suggests that there's a strain back here,

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probably on a more chronic basis.

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And then we also see the region of our rotator

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cuff tear. Once again, small humeral sided.

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Notice I didn't say that it was articular sided

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because there's no articular surface here.

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There's no synovium here.

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There's no highland cartilage here.

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That's an articular surface.

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That's known as the bare area of the articular

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surface where there's no cartilage,

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that is the rest of the articular surface.

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This is a humeral sided,

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concealed delamination tear with

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some bony penetration.

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That is not the main finding in this case.

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The main finding in this case is osteoarthritis,

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hypertrophic,

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with a large goat beard deformity and moderate

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posterior glenoid deficiency syndrome.

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So some of you said high grade rotator

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cuff tear of the supraspinatus.

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I would not call this high grade.

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I'd call it a diminutive concealed,

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interstitial delamination footprint tear that is

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humeral sided. That would go in my report, body.

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And in the conclusion,

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I would say the rotator cuff is mostly intact,

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diffusely tendonotic and described

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as a diminutive tear.

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I would mention in the bile report that there

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is subachromial, subdeltoid fluid.

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And one of you said,

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as a secondary or tertiary finding that

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there is gross DJD of the AC joint.

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And I would say that is absolutely true.

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Very few people don't have DJD of the AC joint.

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How advanced is it? Well, for a 56 year old man,

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I think this is more than I would expect.

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But I try to grade these by how much hypertrophy

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and also how much signal there is on the T2

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weighted image. So there is some signal,

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high signal on the T2 weighted image.

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So I would say that there's mild to modest active

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inflammation of this moderately

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hypertrophic AC joint.

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And I would agree that that is a

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secondary or tertiary finding.

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Some of you said adhesive capsulitis in a man.

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Well,

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I usually don't commingle adhesive capsulitis in

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somebody who can't move their shoulder from OA.

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So it's probably an irrelevant statement

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and not a true statement.

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Some of you said concealed minor tearing of the

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supraspinatus at the insertion is favored

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to be chronic and degenerative,

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and that is absolutely true.

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And there were a few other correct comments

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that were made. The superior labrum,

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amazingly looks pretty good.

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And the reason is can't pick their arm up, right.

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So when you can't pick your arm up,

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you don't get much bouncing behavior of

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the humeral head against the glenoid.

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So the glenoid superior anatomy is preserved.

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And you can see the biceps take off right there.

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Here's the SGHL coming straight at you.

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Here's the coracohumeral ligament right there.

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And there is your tendon of the supraspinatus.

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One, two, three, and four.

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Four layers of hypointensity on the fossa.

Report

Patient History
56-year-old male with right shoulder pain and decreased ROM.
Findings

ROTATOR CUFF: Mild tendinosis of the supraspinatus and infraspinatus followed by teres minor. Interstitial delamination microtear of the subscapularis. No full-thickness or retracted rotator cuff tear.

SUBACROMIAL/SUBDELTOID BURSA: Mild subacromial-subdeltoid peritendinitis.

MUSCLES (ROTATOR CUFF/DELTOID, TRAPEZIUS, PECTORALIS): Mild teres minor strain without tear. No quadrilateral space mass.

BICEPS TENDON: Thin intra-articular segment of the biceps without dislocation or tear. Mild biceps tenosynovitis.

AC JOINT: Mild active acromioclavicular arthropathy, including bony hypertrophy, spurs, periarticular marrow reaction but no separation. Small inferior spurs but no impingement of the traversing cuff.

CORACOCLAVICULAR LIGAMENTS: Intact without tear. Normal coracoclavicular distance.

SUBACROMIAL ARCH/OUTLET: Posterolaterally tilted type 2 acromion without lateral arch stenosis.

SUBCORACOID ARCH: No subcoracoid stenosis.

GLENOHUMERAL JOINT: Posterior decentering of the humeral head. Articular surface flattening on both sides of the joint. Large humeral head spur directed inferiorly. Mostly preserved glenoid bone stock but mild posterior glenoid bone loss and glenoid retroversion noted. Intracapsular debris/bodies including in the subcoracoid bursa.

GLENOID LABRUM: Chronic nondisplaced degenerative posterior and superior labral tear.

BONES: No macrofracture. No infiltrative or destructive bone lesion. Impingement-related pseudocyst formation within the humeral head adjacent to and beneath the infraspinatus insertion. Chronic remodeling of the humeral head with flattening from 12 o’clock to 3 o’clock.

SUBCUTANEOUS SOFT TISSUES: No soft tissue mass.

AXILLA: No lymphadenopathy or vascular abnormality.

Impressions
1. Severe glenohumeral arthropathy including extensive cartilage loss, large humeral head spur, posterior labral tearing, glenoid and humeral head remodeling, complex capsulitis, and debris/bodies in the joint space.
2. Posterior humeral head decentering with greater than 20 degrees of glenoid retroversion.
3. Mild rotator cuff and biceps peritendinitis and tendinosis without tear.
4. Mild active acromioclavicular arthropathy.

Case Discussion

Faculty

Stephen J Pomeranz, MD

Chief Medical Officer, ProScan Imaging. Founder, MRI Online

ProScan Imaging

Jenny T Bencardino, MD

Vice-Chair, Academic Affairs Department of Radiology

Montefiore Radiology

Edward Smitaman, MD

Clinical Associate Professor

University of California San Diego

Tags

Shoulder

Musculoskeletal (MSK)

MRI

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