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Wk 2, Case 4 - Review

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This is a 51-year-old woman who has decreased

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range of motion and pain in her shoulder.

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Oblique coronal fat suppressed situated sequence

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demonstrating small amount of fluid within the joint space.

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But what catches our attention here is this intermediate

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signal intensity tissue interposed between

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the cranial fibers of the subscapularis and the

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anterior leading edge of the supraspinatus tendon.

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So we are right at the area of the rotator

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interval, root of the coracoid process.

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You can see the long head of biceps tendon.

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Uh, going out of the joint space through the rotator

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interval opening into the biceps tendon sheath.

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Now, on oblique sagittal images, these are

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T1 weighted images, we can better see the

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effacement of the fat in the retrocoracoid space.

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However, it's important to note that We should

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check on T2 weighted images because fluid can

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travel into the rotator interval region and it's

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going to look intermediate signal intensity on T1.

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So always, always check back and forth T1 against fluid

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sensitive sequence, in this case a FATSAT T2 weighted sequence.

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And when we look at the retrocoracoid space, it is not fluid.

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It's intermediate signal intensity on

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T1, intermediate signal intensity on T2.

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So this is synovial proliferation in

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the setting of adhesive capsulitis.

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The rotator interval space is the period of proliferation.

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prime location to assess for adhesive capsulitis

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because there are no tendon fibers in that space.

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It's located between the supraspinatus

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superiorly and the psoas scapularis inferiorly.

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Um, so it provides a window to the synovial lining of the joint.

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When there is adhesive capsulitis, we are

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going to have effacement of that space.

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So just to locate you here, supraspinatus.

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Infraspinatus rotator interval, and you see the

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effacement of the fat right behind the coracoid process.

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So this is very, very, uh, compelling

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for the diagnosis of adhesive capsulitis.

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Once we are suspicious of adhesive capsulitis,

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the next step is to go to the axillary recess.

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Why?

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Because in that location, we also have capsule lined

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by synovium without any interposition of tendon fibers.

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And in this patient, we have a very thickened inferior capsule.

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We can see it right here.

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A very thick and inferior capsule, which is also

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edematous, and it can be noted as well in the

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anterior inferior joint recess on the sagittal images.

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So if we cross-reference, we use our cross-referencing

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tool, and we go to the inferior capsule, Right there you see

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the correlation with that area of synovial thickening and

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proliferation with edema in the setting of adhesive capsulitis.

Report

Patient History
51-year-old woman with a 4-month history of right shoulder pain, radiating down her arm, associated with decreased range of motion. Question rotator cuff tendinopathy or tear.

Findings
ROTATOR CUFF:

Supraspinatus: Tendinosis with peritendinitis accompanied by tendon thickening, heterogeneous increased intratendinous signal, and peritendinous edema with scant fluid signal in the distribution of the subacromial bursa. No macro tear.

Infraspinatus: Low-grade tendinosis with peritendinitis, similar to supraspinatus. No macro tear.

Subscapularis: Intact and unremarkable.

Teres minor: Intact and unremarkable.

Biceps tendon and anchor: Intact. Normal anatomic position. Diffuse periligamentous edema through the intra-articular segment. Small to moderate-sized bicipital sheath effusion. No evidence for tenosynovitis.

ACROMIOCLAVICULAR JOINT: Moderate arthrosis accompanied by subcuticular arthropathic cystic change and low-grade distal clavicular periarticular edema/stress response. Moderate capsulosynovial thickening with nominal edema suggesting a low-grade capsulitis.

CORACOCLAVICULAR LIGAMENTS: Intact.

SUBACROMIAL ARCH/OUTLET: Lateral downsloping of a type 2 acromion, combined with a mildly thickened coracoacromial ligament, contribute to mild-to-moderate lateral outlet encroachment.

SUBACROMIAL/SUBDELTOID BURSA: Mildly to moderately thickened, particularly in the subacromial portion. No effusion.

GLENOHUMERAL JOINT: Florid fibro-inflammatory capsulosynovial thickening that is intermediate to high signal on T2 and intermediate signal on T1. Capsular thickening pronounced through the axillary recess/IGHL, rotator interval, and superior capsular labral complex. No joint effusion. No intra-articular loose bodies. No high-grade chondromalacia of the glenohumeral articular surfaces.

GLENOID LABRUM: No traumatic or displaced labral tear.

BONES: No focal osteoedema, micro- or macro-trabecular fracture. No aggressive osseous abnormality. The humeral head is centered within the glenoid.

MUSCLES: Intact. No volumetric muscle atrophy.

SOFT TISSUE: Unremarkable.

AXILLA: Unremarkable.

Impressions
Dominant finding: Florid fibro-inflammatory type glenohumeral capsulosynovial thickening, compatible with clinical adhesive capsulitis.
Mild-to-moderate lateral outlet stenosis due to downsloping type 2 acromion and slightly thickened coracoacromial ligament. Coexisting mild supraspinatus and infraspinatus tendinosis and peritendinobursitis. No rotator cuff tear.
Mild-to-moderate AC joint arthrosis.

Case Discussion

Faculty

Stephen J Pomeranz, MD

Chief Medical Officer, ProScan Imaging. Founder, MRI Online

ProScan Imaging

Jenny T Bencardino, MD

Vice-Chair, Academic Affairs Department of Radiology

Montefiore Radiology

Edward Smitaman, MD

Clinical Associate Professor

University of California San Diego

Tags

Shoulder

Musculoskeletal (MSK)

MRI

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