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Wk 4, Case 2 - Review

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The first case we're going to tackle today

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is a 54- year-old woman with complaints of slight pain,

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popping, decreased range of motion

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that's been ongoing for years.

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Now, the purpose of this meeting is not

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only to review these cases,

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but also to evaluate the comments that have been

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provided to me regarding some of

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your diagnoses and questions.

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So, in my approach to a case like this,

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I usually start out with either a

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set of coronals or the axial.

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I would say more commonly than not,

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I begin with the axial because it's often

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the first image that you're shown.

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And just for expediency,

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I put up what I'm given first.

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However, to get comfortable,

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to see the architecture of the shoulder,

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the conformity,

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the best fit of the humeral head circle

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relative to the glenoid cup.

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Nothing could beat the coronal projection

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as an initial foray into that.

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Now, one of your first comments was that there

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was an anteroinferior labral tear.

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And as you look at this study,

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yes, there is inflammation of the rotator cuff,

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both deep to it, superficial to it, and here,

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I think you would be totally appropriate in

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using the term generalized tendinopathy.

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What you'll also notice, though,

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is the coronal slope of the acromion, which,

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by the way, I never measure.

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It's the relationship of the architecture and

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angle of the clavicle and the acromion,

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which you can see is this way right here.

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Let me get my drawing tool out.

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I'm going to draw it for you.

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So it's clear.

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So this way versus the orientation of the glenoid

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cup, which you can see is this way.

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And that should not be a 90-degree angle.

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Typically, that should be about a 60 or 70-degree angle.

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So the fact that the glenoid cup...

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Let's look at it on the T1. I'm going

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to blow up the T1 for a moment,

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and let's see if I can find my magnifying glass.

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Here we go.

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Going to blow up the T1. Look at the

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architecture of the glenoid.

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And then it has this little dippity-do right

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in the middle, which shouldn't be there.

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Yes, there is a bare area of the glenoid in the middle,

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but there should not be a depression of this size.

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There is a little notch,

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but it shouldn't angle down like this.

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And that little notch is about two to three most.

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This is way too large, way too broad.

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And then it comes back a little bit, but not all the way.

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In other words,

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this glenoid cup should look

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like it's a small pincer.

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It should grab on the bottom and then grab at

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the top. When I take my yellow line away,

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you'll see it's not grabbing the

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humeral head on the bottom.

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I hope you all appreciate that.

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So it's insufficient.

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And there's only two things

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that could really do this.

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Glenoid dysplasia and a dislocation that has

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just worn down and deformed the bone.

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And the patient has no history of a dislocation.

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Yes, she is popping,

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but that's not the same as clinical

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history as a dislocation.

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And then we scroll backwards and we say, well,

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is there a Hill-Sachs?

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No, there's no Hill-Sachs.

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Your volume averaging right here.

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So no Hill-Sachs.

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And let's look at the inferior labral ligamentous

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complex. Now, that looks a little strange.

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If I blow it up on the water-weighted image,

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the band, the glenoid band of the IGHL,

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the inferior glenohumeral ligament.

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There are three bands: an anterior band,

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a posterior band, and an axillary band.

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And this is the glenoid component.

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This is the humeral component.

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This would be the inferior band.

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So when you're anterior, let's go anterior.

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I'll show you the anterior band.

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Anterior band,

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short and stubby.

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Middle band, or axillary band.

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This one's kind of distended.

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And then the posterior band is sort of

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intermediate between the two, right here.

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That one's also distended in the back.

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So the IGHL is stretched out a bit.

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And then we go back to this area right here.

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This entire process,

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which is known as the glenoid labro-ligamentous

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complex, should be one single snug structure.

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I'm going to draw it for you right here.

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Nice and dark.

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I'm making it yellow so you can see it,

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but it should be nice and dark.

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And then you should have the glenoid

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component coming down like this.

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And then the humeral component.

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As you're in the front,

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it's the anterior band in the middle axillary

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band in the back posterior band.

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So that is aberrant.

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There's something wrong with that.

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Now, let's keep going.

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One of you said posterior glenoid dysplasia

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with glenoid retroversion.

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With almost circumferential tearing of the labrum,

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predominantly involving the posterior aspect.

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And that's pretty spot-on, actually.

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So...

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Let's take a look at that.

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Now...

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I think one of the problems that I have with that

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statement is when you say nearly circumferential.

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You're saying that the anterior labrum is torn,

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and I don't think that's the case.

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Let's start up high.

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Start up high.

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And we have this large cord-like structure.

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And what is that?

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That is the conjoint takeoff of the superior

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glenohumeral ligament and then a very thick cord-like

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middle glenohumeral ligament

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with no labrum superiorly.

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And that's a common variation known

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as the Buford variation.

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You can see there's some inflammation around it,

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and that's because the patient has micro

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instability and this glenoid dysplasia.

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Now, let's stop here for a minute.

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We'll take this comment,

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and we'll elaborate on it for a moment.

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We'll get back to the labrum in 1 second.

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But when I look at the glenoid orientation,

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I showed it to you coronally.

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In the axial projection,

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you should be able to put a point

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in the middle of the humerus,

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drop an angle down along the scapular spine,

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and then your glenoid cup should be perpendicular

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to that. Should be a right angle.

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When I take that away,

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I think you'll see that that is not a right angle.

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That there's the angle.

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Now I'm going to take the lines away.

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The angle is more like this.

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Whoops.

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The angle is more like this.

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So you've got a retroverted glenoid cup.

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In other words,

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the anterior portion of the cup is more lateral

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than the posterior portion of the cup.

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Now, I'll allow this about six degrees,

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but this one's more like 15 to 20 degrees.

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So this patient has retroversion

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of the glenoid cup,

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which is forcing the humeral head posteriorly.

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And some of you might appreciate that the humeral

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head almost looks like it bulges a little

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bit more in the back than the front.

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And as we go back up to the top,

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we see this little structure here,

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which is a postero superior labral tear.

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There's also an anterior to posterior labral tear,

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which you can see on the coronal right here.

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This should be snug.

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So your colleague's comment about there being a

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large tear, a nearly circumferential tear,

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is sort of correct. The superior labrum is off.

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The superior labrum is off.

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Again.

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The posterior labrum is beginning to come

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into view. It's still off right there.

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It's still off.

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And now it gets even more profound.

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I'm going to center it a little bit.

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It's still off.

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And now you see it's dissecting over the glenoid

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cup lifting up some capsule periosteal

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tissue right there.

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I'm going to make it a little smaller so

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you can appreciate it a little easier.

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There it is. Still torn.

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Still torn.

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Still too deep and torn.

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Continues, continues, continues.

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So the entire posterior glenoid rim is torn.

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So why am I taking issue with that statement?

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Well, let's go to the front.

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There's a nice triangular-shaped middle glenohumeral

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ligament that's simulating a labrum.

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It's a labral substitute. Let's keep following it.

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There it is. The MGHL is a labral substitute.

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That is not a tear.

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Look at the normal architecture of the bone.

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Look at how smooth everything is.

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That's a fake tear. Let's keep going down.

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Down. That's still a cord.

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That is a cord-like MGHL that is functioning

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as a labrum. Let's come down still.

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We see it again. It is not torn.

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Now, the labrum has taken over.

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The labrum right about here.

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Here's your middle glenohumeral ligament.

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Now, labrum has taken over,

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and the labrum is snug as a bug in a rug.

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Labrum is intact. Intact, intact.

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Now it's starting to get a bit deficient.

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Now you can't really see.

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Now you need your coronal projection to determine

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what's going on in the axillary space where we

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already know that the inferior labroligamentous

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complex, as it is called,

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is peeled away from this dysplastic

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inferior glenoid.

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So your colleague's comment is almost correct.

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Except the anterior labrum, from top to bottom,

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with the exclusion of the anteroaxillary labrum,

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is intact.

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All of that is part of the Buford complex,

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so that is an overstatement.

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Now,

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I said there was something called a notch

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right here called the notch of Ozaki.

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If I was going to draw that in,

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it would be about this big.

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But in this dysplasia, you could say we

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have a notch of Ozaki on steroids.

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It's just way too big and too broad.

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Should be about two or 3 mm right in the barrier.

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So that is an abnormal dysplastic notch.

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So, with regard to the labrum,

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we have an extensive labral tear from the

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superior quadrant all the way to the

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posterior superior quadrant,

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all the way around the back rim,

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extending into the axillary labrum,

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but sparing the entire chunk

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of the anterior labrum.

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So if I was going to draw it

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for you on a sagittal,

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I would say that let's get

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the glenoid cup up here.

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I would say the tear is here.

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I'm not showing you the tear.

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I'm just showing you the architecture

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of where the tear is located.

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And then the whole front end is spared.

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So this part from there to there is spared.

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Okay,

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let's keep commenting now on the remainder

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of your questions on this case,

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because it is a rather extensive case.

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Full-thickness chondral fissuring of the glenoid.

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Well, that's true.

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Here's the cartilage of the glenoid.

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It's not everywhere.

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It just happens to be more central and posterior.

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And when you say full-thickness,

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I assume you mean full depth.

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Yes, it is.

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But when you're in the knee and other structures,

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one of the most important distinctions to make

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is whether a lesion is less than 50% depth,

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more than 50% depth,

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or goes through the osteochondral plate.

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If it goes through the osteochondral plate,

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it's going to be in the bone.

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It's going to be here,

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which so far this patient does not have.

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As far as fissuring goes, well,

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that's a fissure right there.

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Fissure is something linear.

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So I would call this high-grade focal

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chondromalacia due to dysplasia, full

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depth without bone penetration.

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Someone has commented on thickening of

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the coracohumeral ligament. Well,

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let's find that ligament. Here it is.

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And I wouldn't take much issue with that.

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I think it is a bit thickened.

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You see it a little too easily here.

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I'd say it's a minor issue with regard

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to this case, as is the AC joint.

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I don't think the AC joint is

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very germane to the case.

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Just center myself up here a little bit.

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But what is germane to the case is this

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huge fissure in the superior labrum.

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Now,

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I don't mind if you had something

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like this on one cut anteriorly,

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but on the next cut, as I move posterior,

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I want it to disappear.

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Especially in a Buford complex,

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you get a lot of large, deep sulci and fissures,

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but they're anterior, so this is anterior.

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That's the biceps laboral anchor complex.

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Now we go posterior. Look at my line.

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We're going posterior.

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Still there's a line there.

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Still there's a line there.

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There's still a line there.

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And the labrum, the superior labrum itself is torn.

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So it's torn here,

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but it's also torn away from the glenoid.

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So it's a rather complex tear,

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and then it rolls right around the back,

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just as we discussed earlier.

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So this is a huge slap lesion that goes all the

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way from antero-superior to postero-superior

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to postero-inferior to antero-inferior.

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Some other comments that were made.

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Downsloping of the lateral acromion with a bony

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spur contributes to bursitis and fraying

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of the bristle surface of the labrum.

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Well, I can't tell you there isn't fraying.

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Perhaps there's a little fraying right there.

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Yes, there is bursitis. Yes,

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there is a little bit of downsloping

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of the acromion.

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I wouldn't take a lot of issue with that.

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However,

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I would bury something like that

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in the body of the report,

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or I probably wouldn't comment on the fraying.

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But I think that's fair.

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It's a fair comment,

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but it certainly wouldn't appear in my conclusion.

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My conclusion would read glenoid dysplasia with a

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complex labral tear sparing only the anterior

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quadrant of the labrum and involving approximately

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220 to 240 degrees of the labral architecture.

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And then I would have mapped it out in the body

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of the report. I will pull up the sagittal.

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You'll see how much swelling there is.

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I'm not sure there's a lot more

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the sagittal has to give.

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I do like to look at the glenoid cup architecture

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and make sure that I have a pear-shaped glenoid.

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Now,

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it's not a perfect pear because this glenoid is

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dysplastic, but this is what I mean by a pear.

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And when you have a dislocator,

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you start to lose bone in the

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front of the glenoid.

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So you get anterior glenoid deficiency syndrome.

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This patient does not have that.

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They have posterior glenoid deficiency syndrome.

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And you can see that right here.

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The posterior glenoid is attenuated in its position.

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Remember, it should look like this,

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not this.

Report

Patient History
54-year-old female with complaint of slight pain, popping and decreased range of motion in shoulder for years.

Findings
ROTATOR CUFF: Mild tendinosis of the supraspinatus and infraspinatus with interstitial microtearing of the insertions, no macrotear. Normal subscapularis and teres minor.

SUBACROMIAL/SUBDELTOID BURSA: Mild subacromial subdeltoid peritendinitis.

MUSCLES (ROTATOR CUFF/DELTOID, TRAPEZIUS, PECTORALIS): No muscle atrophy, strain, or tear.

BICEPS TENDON: Intact biceps long head tendon without dislocation or tear. Mild extra-articular tenosynovitis.

AC JOINT: Moderate acromioclavicular arthropathy without separation or coracoclavicular ligament injury. No medial arch stenosis.

CORACOCLAVICULAR LIGAMENTS: Intact without tear.

SUBACROMIAL ARCH/OUTLET: Posterolaterally tilted type 2 acromion without lateral arch stenosis.

SUBCORACOID ARCH: No subcoracoid stenosis.

GLENOHUMERAL JOINT: Convex anterior glenoid, posterior tilt of the glenoid relative to the long axis of the scapula. Slight depression or coronal downsloping of the inferior one-third of the glenoid consistent with dysplasia.

Blunting of the posterior glenoid rim with retroversion angle of 24 degrees (normal less than 8) also consistent with dysplasia. 1.5 cm osteochondral defect/remodeling of the glenoid cup. No effusion.

GLENOID LABRUM: Essentially circumferential (sometimes referred to as a a SLAP 9) but primarily posterior labral tearing. Tiny noncompressive paralabral cyst.

BONES: No acute macrofracture. No infiltrative or destructive bone lesion.

SUBCUTANEOUS SOFT TISSUES: No soft tissue mass. No muscle atrophy or tear.

AXILLA: No lymphadenopathy or vascular abnormality.

Impressions
Dysplastic glenoid cup with retroversion, posterior glenoid insufficiency, near circumferential but primarily posterior labral tearing, and tiny noncompressive paralabral cyst.
Mild rotator cuff peritendinitis and tendinosis without tear.
Mild acromioclavicular arthropathy.

Case Discussion

Faculty

Stephen J Pomeranz, MD

Chief Medical Officer, ProScan Imaging. Founder, MRI Online

ProScan Imaging

Jenny T Bencardino, MD

Vice-Chair, Academic Affairs Department of Radiology

Montefiore Radiology

Edward Smitaman, MD

Clinical Associate Professor

University of California San Diego

Tags

Shoulder

Musculoskeletal (MSK)

MRI

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