Interactive Transcript
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Okay, we're talking about the six key components of the
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rotator cuff complex: the supraspinatus,
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the infraspinatus, the teres minor, the subscapularis,
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the capsule, and the coracohumeral ligament.
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We've shown you, in other vignettes, injuries of all of
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them and how to assess them in different projections.
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But I want to focus just for a moment on the
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surrounding anatomy.
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Specifically, I want to focus on the subacromial arch.
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This case of a massive rotator cuff tear,
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there is no supraspinatus.
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There is massive retraction with the fibers seen medially,
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was instigated, at least in part,
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by this funny-looking acromion.
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The acromion has been a subject of
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much debate and much discussion.
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There's even a categorization of the acromion called
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the Bigliani subtypes of the acromion.
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I hardly ever use that.
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What I do use is the proper descriptors.
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I'd like to see if the acromion is downsloping
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in the coronal projection.
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There is a sloping angle,
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but that's a little bit beyond the discussion today.
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How often do I use it? Never.
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I rarely measure things,
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but I do look at them.
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So downsloping in the coronal projection is a little
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different than downsloping in the sagittal projection,
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but I pay attention to both.
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Then there is the shape of the acromion.
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Very often,
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people that have anatomic impingement have an acromion
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that has a little shelf on it, that looks a little
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bit like the end of a telephone receiver.
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You'll also hear this called a keel-like acromion
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I often refer to it as a telephone receiver acromion.
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Now, when you're younger,
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the acromion plays a scant role in impingement,
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unless you have a humeral head that won't stay down,
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in other words,
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it starts floating up when you pick your arm up.
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In other words, there's microinstability.
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The humeral head doesn't rotate
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in the center of the cup.
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And if you pick your arm up and the humeral head
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goes up, what does it bump into? Your acromion.
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As you get a little bit older, the acromion remodels,
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and it becomes like this,
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keel-like.
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And now you've got a self-perpetuating prophecy.
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The humeral head is up. The acromion is in the way.
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The acromion further damages the rotator cuff.
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The humeral head comes up higher, and eventually,
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and unfortunately, they meet bone to bone.
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There is another structure that participates in this
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process, and that is the coracoacromial ligament.
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The coracoacromial ligament comes off the acromion
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and heads towards the coracoid.
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Now, this patient's coracoacromial ligament
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is not that well seen.
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It's right here
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and it's not that thick. It's a little bit thick,
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but most of the time,
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if you went to decide whether the ligament or the bone
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is a bigger contributor to anatomic encroachment,
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it's more often the ligament than the bone.
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It's about 60-40, 70-30, but either one can do it,
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and sometimes it is both. In this case,
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it's more bony than it is ligamentous.
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Let's take a quick look at the sagittal projection for a moment
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and see what our acromion looks like
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in the sagittal projection.
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When we say that the acromion is downsloping,
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here's what we mean.
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Here's our humeral head,
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not a very good drawing of it.
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And if our acromion is tilted down from posterocraniad
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to anterocaudad, it's positively downsloping.
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If it's straight in line with the humeral head,
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something like this, then it's neutral.
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And if it's angled up, then it's negative slope.
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We don't pay too much attention to that.
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We are more interested in positive sloping,
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but especially
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whether the acromion has a hook
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or a spur associated with it.
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And it often looks quite different in the sagittal projection.
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It often looks like it has a little snout,
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and that snout is where the coracoacromial ligament attaches.
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So you have a fat acromion that has a snout or a spur.
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And if it's downsloping sagittally,
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which this one really is not.
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And in the coronal projection,
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you have a kell-like or telephone receiver acromion,
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which in this case it is.
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And if you have a thick coracoacromial ligament,
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which in this case it's a little bit thickened,
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but not very much.
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All of those components are part of the analysis of
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impingement syndrome and contributors
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to rotator cuff pathology.
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