Interactive Transcript
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This case is a 44-year-old gentleman,
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history is follow-up of liver masses.
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So here we have, uh, the liver MRI for this patient.
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We'll start off looking at our T2-weighted sequences.
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As we scroll through the T2-weighted sequences,
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this is a single-shot, uh, spin echo, uh, sequence.
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We can see that there are numerous liver lesions
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scattered throughout the liver, variable size, some
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larger than others, some have lobulated borders.
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We can see that they're fairly
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hyperintense on this sequence.
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And I'm just going to sort of look at one representative
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lesion in order to describe it a little bit more.
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So if we see this large lesion over here, we can see
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that the internal content is very T2 hyperintense.
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But again, we're not going to use this sequence
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to really judge the T2 signal of any lesion.
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We're going to use the Turbospin echo sequence over here.
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And on this one, indeed, look how bright
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this content is of this, of this mass.
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If we see this content this bright, it
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looks so similar to CSF, we're going to
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be, um, not too worried about this lesion.
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It's probably going to be a cyst, maybe a
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hemangioma, but we're going to look at some of
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the other imaging sequences to sort that out.
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Next, we're going to look at the
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T1 in and out of phase sequence.
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We're looking for areas of signal loss.
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On the out-of-phase sequence
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over here, areas of signal loss.
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On the in-phase sequence over here, and if we sort of take
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a global survey of all these liver lesions, we can see
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that they look pretty similar in all the imaging sequences.
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If we look at that representative lesion in the right
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hepatic lobe, we can see that it has T1 hypointense
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signal, and that doesn't change between the out-of-phase
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sequence over here and the in-phase sequence over here.
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Next up, we're going to look at the
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pre- and post-contrast sequences.
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And again, these lesions all on the pre-contrast
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sequence look like they're hypointense, T1 hypointense.
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We're going to look at that large lesion again.
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This is the arterial phase image over here.
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We see that there's no contrast enhancement.
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Next up, we're going to look at
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the T1 pre-contrast sequence.
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We can see all these lesions again have a T1 hypointense
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signal with respect to the liver parenchyma.
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This large representative lesion, again, very
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homogeneous in its signal and T1 hypointense with
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respect to the remaining liver parenchyma, as are the
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other liver lesions that are present for this patient.
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And finally, we're going to look at the dynamic
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post-contrast imaging sequences to assess if there's
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any internal enhancement within these lesions.
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And again, we'll look at the representative
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lesion in the right hepatic lobe.
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You can see the lesion here on
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the, uh, arterial phase images.
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You can see the lesion here on
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the portal venous phase images.
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And finally here on the equilibrium phase images.
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And it looks jet black on all these sequences.
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Um, no enhancement seen.
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And we can also see that around the rim of it, maybe
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there's a minimal, minimal imperceptible rim enhancement.
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And that's okay.
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But internally, inside of it, there's
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absolutely no enhancement identified.
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These findings are compatible with multiple cysts, and
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given the multiplicity of the lesions, you gotta think
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about autosomal dominant polycystic liver disease.
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Now, this is a, uh, finding that can be seen.
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It's not that common.
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It can be seen with other cysts and organs, such
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as the kidneys, or it can be isolated as well.
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And often patients, uh, start off being
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asymptomatic, so no real symptoms, but as the
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cysts increase in size, you can start to get pain.
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Um, like any other cyst, they can rupture and cause
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hemorrhage, uh, potentially get infected as well.
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If symptomatic, patients will need to
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undergo resection or again, marsupialization.
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So, this is basically, uh, multiple liver cysts
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seen in patients, and typically we're talking
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about more than 10 or 20 liver cysts, um, and you
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gotta start thinking about this, uh, this entity.
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