Interactive Transcript
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This next patient is a 58-year-old female who presents with
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microscopic hematuria for which a CT scan was performed.
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As we look at our CT images, we do them in multiple
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phases as per a routine hematuria protocol.
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Now we can see that in the lateral left hepatic
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lobe, There is an indeterminate liver lesion.
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On the non-contrast portion of the
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study, the lesion is hard to see.
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If you window it, you'll probably see that there's
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a faintly hypodense lesion in this location.
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On the nephrographic phase, we can see that
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the lesion enhances pretty homogeneously.
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And finally, in our delayed phases, the lesion is very,
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very difficult to see, very tough to know where it is.
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So this was deemed to be indeterminate, and
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an MRI was suggested for further evaluation.
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We'll start off our MR evaluation
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looking at the T2-weighted sequence.
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We'll scroll down to see if we can find the lesion.
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We can see the lesion over here.
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And on this T2-weighted sequence, we can see
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that the lesion has hyperintense signal.
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Again, the signal is similar, but perhaps a
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little bit less bright than the CSF down here.
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But it's bright nonetheless, and it generally demonstrates
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a signal that, by and large, I would not be worried about.
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However, need to look at the remaining
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sequences to further evaluate this.
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The next set of sequences are going to be
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the T1 performed in phase and out of phase.
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And we'll scroll down to see what this lesion looks like.
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In the out-of-phase sequence over here, you can see the
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lesion in the left hepatic lobe on the in-phase sequence.
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Again, we see this lesion in the left hepatic lobe.
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The overall T1 signal is hypointense, pretty low signal.
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There's no loss of signal in the out-of-
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phase imaging to suggest hepatic steatosis.
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And no areas of, uh, loss of signal in the
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in-phase images to suggest that it contains
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anything that causes increased susceptibility.
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On this, we can see the lesion again in the left
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hepatic lobe over here, and we can see that,
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once again, it has this hypointense T1 signal.
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We'll have to see how it enhances after giving contrast
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to make a final assessment of what we think this is.
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So finally, we now have our dynamic
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post-contrast imaging sequences.
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We'll have a look and see if and how this lesion enhances.
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Once again, we can see it in the lateral left hepatic lobe.
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On the arterial phase images, we notice
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that the lesion homogeneously enhances.
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I'll draw a schematic of this lesion, and it enhances
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very homogeneously on the arterial phase images.
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On the portal venous phase image
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over here, the lesion stays enhanced.
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However, the brightness of it is not as
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bright as it was in the arterial phase image.
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Finally, on the equilibrium phase image,
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once again, this lesion stays enhanced.
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So overall, we have a T2 hyperintense mass,
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homogeneously enhances.
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and remains enhanced or hyperintense on
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the remaining contrast-enhanced phases.
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If you notice, on the arterial phase images,
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the lesion is as bright as the predominant.
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These imaging features are characteristic
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of a flash-filling hemangioma.
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Now, these are a subset of hemangiomas where you have
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homogeneous enhancement with the lesion remaining hyper
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intense on the remaining post-contrast images, and the
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enhancement pattern generally follows the blood pool.
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We tend to see it in hemangiomas that
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are less than two centimeters in size.
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This is, uh, a little bit probably at the cutoff
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for that border, but generally when they're
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less than two centimeters in size, they can
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have this sort of flash-filling appearance.
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Sometimes with these lesions, you may also
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see areas of perilesional enhancement.
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phase, you may sometimes see areas
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of, uh, non-mass-like arterial hyperenhancement
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that subsequently become isointense to the
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remaining liver parenchyma on the remaining phases.
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So these findings are characteristic
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of a flash-filling hemangioma.
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