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Flash Filling Hemangioma

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This next patient is a 58-year-old female who presents with

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microscopic hematuria for which a CT scan was performed.

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As we look at our CT images, we do them in multiple

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phases as per a routine hematuria protocol.

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Now we can see that in the lateral left hepatic

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lobe, There is an indeterminate liver lesion.

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On the non-contrast portion of the

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study, the lesion is hard to see.

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If you window it, you'll probably see that there's

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a faintly hypodense lesion in this location.

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On the nephrographic phase, we can see that

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the lesion enhances pretty homogeneously.

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And finally, in our delayed phases, the lesion is very,

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very difficult to see, very tough to know where it is.

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So this was deemed to be indeterminate, and

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an MRI was suggested for further evaluation.

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We'll start off our MR evaluation

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looking at the T2-weighted sequence.

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We'll scroll down to see if we can find the lesion.

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We can see the lesion over here.

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And on this T2-weighted sequence, we can see

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that the lesion has hyperintense signal.

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Again, the signal is similar, but perhaps a

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little bit less bright than the CSF down here.

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But it's bright nonetheless, and it generally demonstrates

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a signal that, by and large, I would not be worried about.

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However, need to look at the remaining

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sequences to further evaluate this.

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The next set of sequences are going to be

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the T1 performed in phase and out of phase.

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And we'll scroll down to see what this lesion looks like.

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In the out-of-phase sequence over here, you can see the

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lesion in the left hepatic lobe on the in-phase sequence.

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Again, we see this lesion in the left hepatic lobe.

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The overall T1 signal is hypointense, pretty low signal.

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There's no loss of signal in the out-of-

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phase imaging to suggest hepatic steatosis.

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And no areas of, uh, loss of signal in the

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in-phase images to suggest that it contains

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anything that causes increased susceptibility.

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On this, we can see the lesion again in the left

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hepatic lobe over here, and we can see that,

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once again, it has this hypointense T1 signal.

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We'll have to see how it enhances after giving contrast

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to make a final assessment of what we think this is.

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So finally, we now have our dynamic

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post-contrast imaging sequences.

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We'll have a look and see if and how this lesion enhances.

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Once again, we can see it in the lateral left hepatic lobe.

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On the arterial phase images, we notice

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that the lesion homogeneously enhances.

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I'll draw a schematic of this lesion, and it enhances

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very homogeneously on the arterial phase images.

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On the portal venous phase image

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over here, the lesion stays enhanced.

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However, the brightness of it is not as

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bright as it was in the arterial phase image.

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Finally, on the equilibrium phase image,

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once again, this lesion stays enhanced.

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So overall, we have a T2 hyperintense mass,

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homogeneously enhances.

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and remains enhanced or hyperintense on

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the remaining contrast-enhanced phases.

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If you notice, on the arterial phase images,

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the lesion is as bright as the predominant.

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These imaging features are characteristic

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of a flash-filling hemangioma.

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Now, these are a subset of hemangiomas where you have

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homogeneous enhancement with the lesion remaining hyper

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intense on the remaining post-contrast images, and the

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enhancement pattern generally follows the blood pool.

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We tend to see it in hemangiomas that

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are less than two centimeters in size.

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This is, uh, a little bit probably at the cutoff

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for that border, but generally when they're

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less than two centimeters in size, they can

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have this sort of flash-filling appearance.

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Sometimes with these lesions, you may also

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see areas of perilesional enhancement.

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phase, you may sometimes see areas

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of, uh, non-mass-like arterial hyperenhancement

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that subsequently become isointense to the

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remaining liver parenchyma on the remaining phases.

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So these findings are characteristic

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of a flash-filling hemangioma.

Report

Faculty

Mahan Mathur, MD

Associate Professor, Division of Body Imaging; Vice Chair of Education, Dept of Radiology and Biomedical Imaging

Yale School of Medicine

Tags

Vascular

Non-infectious Inflammatory

MRI

Liver

Idiopathic

Gastrointestinal (GI)

CT

Body

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