Interactive Transcript
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So this patient is a 60-year-old
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female, and the history provided
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is elevated liver function tests.
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LFTs are elevated.
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Um, wanted to get an MRI; sounds very reasonable.
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So we do our MRI examination, and we're going
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to go through some sequences to see if we
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can figure out what's causing these problems.
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So here's our T2-weighted sequence,
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non-fat-saturated in the axial plane.
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And we see what probably amounts
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to a small liver cyst over there.
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We can ignore that for the moment.
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And as we go down, we already start to
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see that the bile ducts are dilated.
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Now remember, if you've gone through
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sort of the normal anatomy on these T2
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weighted sequences, you hardly ever get
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to see the intrahepatic bile ducts.
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They're so small, they should
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be less than two millimeters.
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So the fact that I'm seeing the bile
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ducts on my T2-weighted sequences
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here tells me that they’re dilated.
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Doesn't necessarily have to mean there’s
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some abnormality associated with it,
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but it does mean that they are dilated.
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So let’s figure out if we
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can see why they’re dilated.
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You can see here, very, very dilated.
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And again, this is the extrahepatic biliary tree.
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You can see the cystic duct
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over here on this image.
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We start to see a bit of a gallbladder.
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You can see some gallstones here,
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T2 hypointense filling defects.
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But let's ignore that again for the moment.
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Let's focus right over here on
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the extrahepatic biliary tree.
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And already, the next slice downwards, you
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can start to see a filling defect inside
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the common hepatic duct over here.
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At this point, it may even
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be the common bile duct.
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Certainly, as you go lower, it starts
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to become the common bile duct.
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Another filling defect here, another filling
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defect here, T2 hypointense, and quite a
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large one here within the common bile duct.
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Another one here.
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And we can go all the way downwards
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and see that it looks relatively clean.
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Let's look at the same findings, but just
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on a different plane, the coronal plane.
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Again, we can see that the
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intrahepatic tree is dilated.
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Both the right and left hepatic ducts,
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the common hepatic duct is dilated.
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As we go downwards, we can start to
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see that there are numerous filling
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defects inside the common bile duct.
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Various shapes, various sizes, all of them
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are T2 hypointense as we go downwards.
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And so these are going to be
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some filling defects here.
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We can look at the same thing on
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our T2 fat-saturated sequences.
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A little bit more motion here, but we can
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certainly appreciate the filling defects.
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And notice how all these filling
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defects are dependent.
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Right.
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So they're sort of going to the dependent
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portion over here, which is obviously posteriorly
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and they're lying down upon the dependent
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portion of the bile duct for the most part.
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We can look at the same thing
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on our T1 pre-contrast image.
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And we notice that some of these
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filling defects, a lot of
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them really are T1 hyperintense.
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And in this instance, we did an MRCP.
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And so I just want to finish off the
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description by showing you the MRCP images,
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and the images came out reasonably well.
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You can see that the intrahepatic bile ducts are
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dilated; the extrahepatic biliary tree is dilated.
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You can start to see some filling
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defects inside the extrahepatic biliary tree.
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And this is sort of an MIP image showing
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at least one of those filling defects.
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And so again, you know, this is not giving
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me any extra information that I haven't
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gleaned from my other sequences, but, you
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know, nevertheless, this may be useful.
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And so what's going on here?
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You may have guessed it already.
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This is an example of choledocholithiasis.
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Choledocholithiasis refers to the
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presence of stones in the bile ducts.
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Oftentimes, we see it due to the passage of stones
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from the gallbladder that escape and go into
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the extrahepatic biliary tree, but it's
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important to know that they can potentially
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form in the absence of gallstones in the
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setting of biliary stasis for whatever reason.
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Oftentimes, patients may present with
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biliary colic as a result of these stones.
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We also see patients who are relatively
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asymptomatic and just sort of present with
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incidentally noted choledocholithiasis.
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Most times, if patients are suspecting
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choledocholithiasis, they will get an ultrasound,
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but oftentimes, the more distal aspects of the
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common bile duct are tough to see on ultrasound.
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And so an MRI is pretty rare
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often performed if the findings
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are unclear on ultrasound.
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Choledocholithiasis essentially
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looks very similar to this.
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They're dependent filling defects,
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with relatively geographic shapes, such as
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circles or small triangles.
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All of them will be T2 hypointense.
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Most times they're also T1 hypointense.
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In this case, these were T1 hyperintense,
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which you could see with pigmented stones.
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It's also important to understand that
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MRI is very good at detecting these, but
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occasionally, if they are very tiny stones,
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maybe two millimeters or even three millimeters
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or less, we may end up missing those stones.
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So sometimes we'll call it no choledocholithiasis
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and then an ERCP may reveal
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a few tiny stones that were missed.
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But MRI does fairly well for the most part
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in detecting choledocholithiasis, and this is a
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nice case of choledocholithiasis resulting in
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biliary ductal dilatation and presumably the
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elevated liver function tests in this patient.
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