Interactive Transcript
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So here we have a patient.
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History, uh, is elevated lipase.
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She's a lady in her fifties.
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And so we're getting an MRI without
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intravenous contrast and an MRCP sequence
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added onto it in order to evaluate
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the etiology of the elevated lipase.
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And presumably they're looking for
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choledocholithiasis in this instance.
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And so as we scroll on in our T2
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weighted sequence, we can see that
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the intrapathic biliary tree is
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larger than what we would expect
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in this patient.
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So probably something's happening.
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So we'll have to evaluate it further.
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The common bile duct also looks quite large.
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And as we scroll all the way down, we can see
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that the common bile duct is on the bigger side.
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It does taper as we go downwards.
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You can see the caliber of this sort of circle
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getting a little bit smaller, smaller, smaller,
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as it goes all the way down to the ampulla.
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So let me sort of zoom in on this, and
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you know, if you've gone through the other
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cases, you're reasonably familiar with
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what choledocholithiasis looks like, which
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is a T2 hypointense filling defect, right?
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And when we say hypointense, we're
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really looking at something that looks
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as dark as this sort of signal, or this
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sort of signal, or this sort of signal.
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We don't see anything like that
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inside the common bile duct in
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this instance, all the way through.
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Now you can certainly verify that before
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we draw that conclusion on other sequences
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here, we have a coronal T2-aided sequence.
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Now there's not a lot of motion on this study.
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This patient tolerated the MRI quite well.
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You can see here, um, the common hepatic and
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common bile duct is dilated, tapers nicely
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as we get to the ampulla, but certainly no T2
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hypointense filling defects are seen within it.
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Again, findings, uh, you can look at
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the MRCP sequence, another fairly good
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sequence, just a little bit of motion,
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but as you scroll through it, really no
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choledocholithiasis that we can identify.
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Now, remember, we may potentially miss tiny,
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tiny stones, so it is possible that the
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patient has a tiny stone or maybe passed a
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recent stone that we're not seeing, but the
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reason that I wanted to show you this case is
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for this finding within the common bile duct.
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If you look at it, say, on this slice
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over here, you do notice that
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the inside of the common bile
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duct is not particularly clean, right?
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This is what bile looks like.
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It's fairly T2 hyperintense.
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Look at it more posteriorly.
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There is a signal alteration there that looks a
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little bit more hypointense than clean bile, right?
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It's not as dark as what a stone would look
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like, but it's a little bit darker than bile.
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And if you kind of look upwards here,
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again, there's a little bit of a layering
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effect where there's some semblance of
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a straight line, even, that separates bile
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that's relatively clean, superior to it,
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and stuff that's relatively hypointense
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inferior to it.
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We also have these oblique T2-weighted
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images where we have angled, we're angling
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with respect to the bile duct as well.
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And I just wanted to show this to
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you because some of that layering
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becomes more apparent right over here.
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If we were to window this, we can see
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that over here, it looks relatively clean
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and over here, more dependently, there's
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a layering effect right over here as
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well, where it's relatively hypointense.
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So this finding is highly suggestive of the
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presence of sludge inside the common bile duct.
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And in fact, if we look at the
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adjacent gallbladder, we can see some
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sludge layering within it as well.
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So some of the sludge may have sort of
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transferred over to the common bile duct.
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And, you know, we can often see this
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in the gallbladder, as I mentioned,
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but I just wanted to show you an
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example of what that could look like
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within the common bile duct.
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And much like stones, sludge can also cause
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relative stasis of the biliary tree where
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the bile is just not emptying very nicely.
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You can see it over here as well.
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And so this patient had an ERCP and indeed
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they removed some sludge and a few tiny,
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tiny debris from the bile ducts themselves.
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