Interactive Transcript
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So this patient is a seven-year-old
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patient with cirrhosis, and we were asked
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to evaluate for hepatocellular carcinoma.
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And I wanted to show this case for a
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specific finding that will sort of build
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upon something we discussed in another case.
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So starting off with the T2-weighted
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images, you know, we look at the liver and
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there's a lot of stuff going on over here.
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Looks like there is lots of T2 hyperintense
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abnormality, sort of in the central portion,
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clustered in the central portion of the liver.
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Some of it is extending out to the periphery,
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but the majority of it is in the center.
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And if we look at the portal vein, we
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notice that this sort of appearance
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on both sides of the portal vein.
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And I think if you were to look at this
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in the beginning, you know, someone may
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consider this being dilated bile ducts.
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But as mentioned in one of the other cases,
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there's also this entity of peribiliary cysts,
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which are these dilatations of glands within
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the periductal tissue of intrahepatic bile ducts
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that don't communicate with the biliary tree
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and for whatever reasons, particularly
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patients who are cirrhotic, they can get
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obstructed and these glands can dilate.
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They dilate and they look like clusters
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and clusters of cysts that are on both
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sides of the portal vein and they have no
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communication to the biliary tree itself.
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And one of the reasons I wanted to show
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this case is that, you know, certainly this,
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this has that sort of appearance, but just
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to kind of showcase the fact that these are
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not communicating with the biliary tree,
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this particular patient was done, you know,
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with and without intravenous contrast.
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And for contrast, we use an agent
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which has partial hepatobiliary
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excretion, which is Eovist.
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And so what we see on the post-contrast images
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done at around 20 minutes after giving the
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contrast agent is that there is actual excretion
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of the contrast agent in the bile ducts, right,
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hepatic ducts, and some of the left hepatic
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ducts over here, but there is no excretion
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of contrast within these clusters of cysts.
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And so that again proves the point
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that these are cystic structures
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that represent dilatation of
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ducts associated with the biliary tree.
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But they don't communicate with it.
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If they did communicate with it, they would
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also contain some of this contrast agent.
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But the fact that they remain hypointense
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and don't contain any of this contrast agent,
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again, highlights that important concept
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that these are peribiliary cysts that just
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sort of are adjacent to, but separate
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really from the bile ducts themselves.
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And once again, this finding in and of
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itself is of no clinical importance.
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As the patient's liver disease gets worse, we
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can sometimes see the finding get worse,
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but as such, nothing really to worry
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about when you see them, even though
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they have quite a dramatic appearance
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sometimes on these T2-weighted images.
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