Interactive Transcript
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Okay, this next case is the case of a 14
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year old male who presented to the emergency department with chest pain.
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Eventually, he was worked up.
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They found that he had an elevated troponin of around 70. Very high.
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And then the concern was that he had acute myocarditis.
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Cardiac MRI was ordered to confirm the diagnosis.
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This is not uncommon reason to perform cardiac MRI.
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We get quite a few referrals for this.
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And the main things that we're looking
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for are, we want to see T1 abnormalities and T2 abnormalities.
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The T1 abnormalities are going to be the late gadolinium enhancement,
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and the T2 abnormalities are going to be the edema.
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And so, we're going to start here with the cine four chamber view.
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And what you can see is that the patient has nice preserve function.
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I'll tell you that acute myocarditis
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almost every case I can think of that I've ever seen, they have preserve function.
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So generally, in the acute phase,
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you're going to have pretty normal myocardial function
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if it is an acute viral myocarditis
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in a child. There is such an entity as chronic myocarditis.
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So kids that get myocarditis, and then for whatever reason, the infection doesn't
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clear. Those kids can get reduced cardiac function.
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But generally, that first presentation, they're going to have normal function.
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What you can see here, though,
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and this is before we've given any gadolinium, is that there is this bright
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signal here in the lateral wall of the left ventricle.
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So remember that these SSFP images are
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a combination of both T2 and T1 weighting.
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So if you see any bright signal in the myocardium on the pre-contrast
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imaging, usually that means it's going to be edema.
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So that T2 signal abnormality kind
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of almost like shines through the background on these SSFP images.
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What is part of the standard evaluation
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of patients who have myocarditis is the black blood imaging.
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The black blood T2 weighted images are used to look for edema.
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In this case, this patient has really extensive and bright edema in the lateral
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wall of the left ventricle here in the subepicardial space.
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And this is a classic for myocarditis.
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Myocarditis love to involve the sub epicardium of the lateral wall,
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particularly at the base, mid cavity and apex.
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And it's often this sort of linear pattern
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where it's not so much a blob, it's not discrete nodules.
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It's really this linear pattern along
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the length of the subepicardial surface of the lateral wall.
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Now, if we move ahead to the two chamber
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cine view, you're going to see that, again, the function is preserved.
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And then when we look at the short axis
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cine images, again, the function is preserved. Normal wall thickness.
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But you do see that bright signal in the lateral wall shining through
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again. When we move to the short axis late
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enhancement images, you're going to see that there's quite
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a bit of enhancement in this patient, in a pattern very consistent with myocarditis.
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And that pattern is subepicardial lateral wall and inferior wall.
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Classic for myocarditis, particularly at the base and the mid cavity.
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This patient's actually got basal sparing, which is sometimes a little bit unusual,
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but the remainder of the case is normal and typical, where you have this lateral
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wall involvement stretching around to the inferior wall
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really extensive.
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Even a little bit of mid wall involvement of the septum.
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And that's really classic for myocarditis.
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And certainly, in the clinical setting of having an elevated troponin and chest
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pain, this is basically makes the diagnosis.
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I just want to show you that late enhancement on the long axis images.
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It's right here.
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All this subepicardial late enhancement, it corresponds to the abnormalities
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that we saw on the T2 weighted black blood imaging.
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So we have a T2 abnormality.
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We have a T1 abnormality.
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Those are the key components of making the diagnosis of myocarditis.
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So, in summary, this patient has
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late enhancement in the lateral wall and a non-ischemic subepicardial distribution,
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with edema in the same areas compatible with acute myocarditis.
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