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Musculoskeletal Imaging
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Prepare trainees to be on call for the emergency department with this specialized training series.
Training Collections
Library Memberships
Sale 25% OffOn-demand course library with video lectures, expert case reviews, and more
Fellowship Certificate™ Programs
Sale 25% OffPractice-focused training programs designed to help you gain experience in a specific subspecialty area.
Ultimate Learning Pass
Sale 30% OffUnlock access to our full Course Library and all self-paced Fellowships.
Continuing Medical Education (State CME)
Complete all of your state CME requirements in one convenient place.
Noon Conference (Free)
Get access to free live lectures, every week, from top radiologists.
Case of the Week (Free)
Get a free weekly case delivered right to your inbox.
Case Crunch: Rapid Case Review (Free)
Register for free live board reviews.
Dr. Resnick's MSK Conference
Learn directly from the MSK Master himself.
Lower Extremities MRI Conference
Musculoskeletal Imaging
Emergency Imaging
PET Imaging
Pediatric Imaging
For Training Programs
Supplement your training program with case-based learning for residents, registrars, fellows, and more.
For Private Practices
Upskill in high growth, advanced imaging areas.
Compliance
NewTrack, fulfill, and report on all your radiologists' credentialing and licensing requirements.
Emergency Call Prep
Prepare trainees to be on call for the emergency department with this specialized training series.
1 topic, 2 min.
24 topics, 2 hr. 9 min.
Clinical Scenario 1: New Neurologic Deficit Introduction
3 m.Case: Left MCA Stroke on Non-Contrast CT
5 m.Case: Left MCA Stroke on CTA
13 m.Case: Left MCA Stroke on MRI
9 m.Non-Contrast Findings in CT and Stroke
7 m.ASPECTS Score
4 m.Perfusion Evaluation
7 m.Timing of Therapy for Stroke
6 m.Case: Occluded Right MCA
11 m.Case: Acute Left MCA Infarct with Penumbra
12 m.Case: RAPID Analysis
4 m.Case: Right M1 Occlusion on MRI
9 m.Case: Old and New Strokes: Cardioembolic Phenomenon
7 m.Case: Basilar Artery Clot on CTA, CT, CTP
8 m.Case: Childhood Stroke on MRI, MRA, MRP
7 m.Case: Moyamoya Syndrome
4 m.Case: Childhood Stroke, Moyamoya on CT
4 m.Case: Superior Sagittal Sinus Thrombosison CT, CTV
4 m.Case: Imaging of Sinus Thrombosis
6 m.Case: Cortical Vein Thrombosis on CT, MRI, MRV
4 m.Case: Cortical Vein Thrombosis on CTV
3 m.Case: New Neurologic Deficit from Multiple Sclerosis
2 m.Case: Glioblastoma
3 m.New Neurologic Deficit Lesson Reinforcement Quiz
29 topics, 1 hr. 40 min.
Clinical Scenario 2: Head Trauma Introduction
3 m.Case: Head Trauma wtih Multicompartmental Hemorrhage
6 m.Case: SDH with Active Bleeding
4 m.Traumatic Brain Injury
7 m.Cortical Contusions
7 m.Extra-Axial Collections
3 m.Case: Subdural Hematoma on CT
2 m.Case: Epidural Hematoma on CT
3 m.Case: Epidural Hematoma from Transverse Sinus Injury on CT
3 m.Case: Epidural Hematoma from Transverse Sinus Injury, Prognosis on CT
2 m.Acute Epidural Hematomas
2 m.Epidural Hematomas, Continued
2 m.Case: Isodense Subdural Hematoma
4 m.Acute Subdural Hematomas & Diffuse Axonal Injury
10 m.Density of Falx/Tentorium
6 m.Depressed Skull Fractures
4 m.Case: Occipital Bone Open/Depressed Fracture on CT
3 m.Role of MRI in Head Trauma
3 m.Case: Non-Accidental Trauma
6 m.Non-Accidental Trauma CT (Part 1)
3 m.Non-Accidental Trauma CT (Part 2)
2 m.Posterior Fossa Lesions from Trauma
3 m.Case: DAI on MRI
7 m.Case: DAI on CT
3 m.Diffuse Axonal Injury
3 m.Case: DAI with Blood Products on CT
3 m.Traumatic Injuries: Herniation
6 m.Case: Herniations on CT
4 m.Head Trauma Lesson Reinforcement Quiz
19 topics, 1 hr. 24 min.
Clinical Scenario 3: Worst Headache of Life Introduction
2 m.Case: Ruptured PCA Aneurysm Leading to IPH on CT, Arteriogram
5 m.Case 26: Basilar Artery Aneurysm on CT, CTA
7 m.Localization of Aneurysm with SAH
3 m.Imaging of Aneurysms
9 m.Case: Mycotic Aneurysm on CT, CTA
4 m.Case 28: Non-Infectious Mycotic Aneurysm on CT
4 m.Arteriovenous Malformation
5 m.Case: Hypertensive Bleed, IPH with IVH on CT (Case 1)
4 m.Case: Hypertensive Bleed, IPH with IVH on CT (Case 2)
3 m.Signal Intensity of IPH on MRI by Age
12 m.Reversible Cerebral Vasoconstriction Syndrome (RCVS)
4 m.Non-Aneurysmal Perimesencephalic SAH
4 m.Cerebral Amyloid Angiopathy
4 m.Case: Idiopathic Intracranial Hypertension on CTA, CTV
5 m.Idiopathic Intracranial Hypertension (IIH)
6 m.Case: Intracranial Hypotension on MRI
6 m.Case: Intracranial Hypotension - Spinal Imaging on MRI
5 m.Worst Headache of Life Lesson Reinforcement Quiz
16 topics, 41 min.
Clinical Scenario 4: Found Down Introduction
2 m.Case: Anoxic Brain Injury
3 m.Metabolic Brain Disease
5 m.Case: Hyperammonemia on MRI
3 m.Case: Thiamine Deficiency on MRI
5 m.Thiamine Deficiency
3 m.Posterior reversible encephalopathy syndrome (PRES)
5 m.Case: PRES: MRI
3 m.PRES Variants
2 m.Cytotoxic Lesions of the Corpus Callosum (CLOCC)
2 m.Case: CLOCC from Seizure Medication on MRI
2 m.Case: Toxic Leukoencephalopathy on MRI
3 m.Case: Toxic Leukoencephalopathy from Medication on MRI
2 m.Toxic Leukoencephalopathy
3 m.Case: Hypoxic Ischemic Encephalopathy
6 m.Found Down Lesson Reinforcement Quiz
9 topics, 26 min.
Clinical Scenario 5: Fever and Seizure Introduction
2 m.Case: Herpes Encephalitis on MRI
6 m.Case: Herpes Encephalitis in a Lung Cancer Patient on MRI
3 m.Case: Listeria Rhombencephalitis on MRI
4 m.Status Epelipticus, CJD, and Encephalitis
4 m.Case: Abscess on MRI (Case 1)
4 m.Case: Abscess on MRI (Case 2)
3 m.Case 37 - Subacute BE with ventriculitis and sceptic emboli
4 m.Fever & Seizures Lesson Reinforcement Quiz
4 topics, 14 min.
0:01
Well, if superior sagittal sinus or transverse
0:04
sinus thrombosis is not subtle enough to identify,
0:09
a cortical venous thrombosis is even more subtle.
0:14
And what you're looking for are veins that
0:17
are leading towards the superior sagittal
0:20
sinus or transverse sinus that are too dense.
0:23
This happens to be a Grade A,
0:27
very obvious case, but here we have a patient
0:31
who had a new neurologic deficit.
0:33
In this case, it was left-sided hemiparesis.
0:38
And on the CT scan, we saw this low-density area
0:42
in the perirolandic motor area on the right side.
0:49
But in addition, we noticed that there
0:52
were some bright blood vessels that were
0:55
leading towards the superior sagittal sinus.
0:58
So these bright blood vessels coming from the
1:01
periphery would be our cortical vein thrombosis.
1:06
This would probably be better demonstrated
1:09
on the thinnest section imaging.
1:11
And if I scroll all the way up and window appropriately,
1:16
you can see those hyperdense veins coming together
1:22
and then joining the superior sagittal sinus.
1:25
So the sinus itself was not thrombosed,
1:27
but the peripheral veins were thrombosed,
1:30
leading to this patient's area of abnormal density.
1:36
When we look at the accompanying MRI scan,
1:41
which actually was performed after the CT,
1:46
what you see is the edema of the affected
1:52
tissue along the motor strip region.
1:56
This is the motor strip coming
1:57
along here in this patient.
2:02
And there is that amount of edema.
2:05
Venous infarctions have a high
2:07
rate of potential hemorrhage.
2:11
So on the susceptibility-weighted and gradient
2:14
echo scan, you may see blood products here.
2:17
What we're also seeing is the
2:19
clot in that superficial vein.
2:22
So this is a dilated vein filled with black clot,
2:28
acute clot, as well as parenchymal hemorrhage
2:33
in the area of venous injury.
2:35
Curiously enough, when we make a move to
2:39
the diffusion-weighted imaging, this is
2:43
the ADC map, you don't see all that much.
2:47
And that's one of the things about venous infarctions,
2:49
they can have a very confusing appearance on the DWI.
2:54
Largely because not only do they cause cytotoxic
2:58
edema, but because of the venous obstruction,
3:01
they also cause vasogenic edema in the outflow.
3:06
The MR venograms are going to be hard to
3:09
interpret because we're looking at not the sinus,
3:11
which is the easiest part, but those vessels
3:14
leading to the sinuses that will be thrombosed.
3:18
In this case, assuming everything is symmetrical,
3:22
we see the normal draining veins on the left side
3:25
and the missing draining vein on the right side.
Interactive Transcript
0:01
Well, if superior sagittal sinus or transverse
0:04
sinus thrombosis is not subtle enough to identify,
0:09
a cortical venous thrombosis is even more subtle.
0:14
And what you're looking for are veins that
0:17
are leading towards the superior sagittal
0:20
sinus or transverse sinus that are too dense.
0:23
This happens to be a Grade A,
0:27
very obvious case, but here we have a patient
0:31
who had a new neurologic deficit.
0:33
In this case, it was left-sided hemiparesis.
0:38
And on the CT scan, we saw this low-density area
0:42
in the perirolandic motor area on the right side.
0:49
But in addition, we noticed that there
0:52
were some bright blood vessels that were
0:55
leading towards the superior sagittal sinus.
0:58
So these bright blood vessels coming from the
1:01
periphery would be our cortical vein thrombosis.
1:06
This would probably be better demonstrated
1:09
on the thinnest section imaging.
1:11
And if I scroll all the way up and window appropriately,
1:16
you can see those hyperdense veins coming together
1:22
and then joining the superior sagittal sinus.
1:25
So the sinus itself was not thrombosed,
1:27
but the peripheral veins were thrombosed,
1:30
leading to this patient's area of abnormal density.
1:36
When we look at the accompanying MRI scan,
1:41
which actually was performed after the CT,
1:46
what you see is the edema of the affected
1:52
tissue along the motor strip region.
1:56
This is the motor strip coming
1:57
along here in this patient.
2:02
And there is that amount of edema.
2:05
Venous infarctions have a high
2:07
rate of potential hemorrhage.
2:11
So on the susceptibility-weighted and gradient
2:14
echo scan, you may see blood products here.
2:17
What we're also seeing is the
2:19
clot in that superficial vein.
2:22
So this is a dilated vein filled with black clot,
2:28
acute clot, as well as parenchymal hemorrhage
2:33
in the area of venous injury.
2:35
Curiously enough, when we make a move to
2:39
the diffusion-weighted imaging, this is
2:43
the ADC map, you don't see all that much.
2:47
And that's one of the things about venous infarctions,
2:49
they can have a very confusing appearance on the DWI.
2:54
Largely because not only do they cause cytotoxic
2:58
edema, but because of the venous obstruction,
3:01
they also cause vasogenic edema in the outflow.
3:06
The MR venograms are going to be hard to
3:09
interpret because we're looking at not the sinus,
3:11
which is the easiest part, but those vessels
3:14
leading to the sinuses that will be thrombosed.
3:18
In this case, assuming everything is symmetrical,
3:22
we see the normal draining veins on the left side
3:25
and the missing draining vein on the right side.
Report
Faculty
David M Yousem, MD, MBA
Professor of Radiology, Vice Chairman and Associate Dean
Johns Hopkins University
Tags
Vascular
Neuroradiology
MRV
MRI
Emergency
CT
Brain
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