Interactive Transcript
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So we do have another case with a similar presentation, and that's this patient here
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who is 69 years old, complaining of shortness of breath.
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And again, if we go through in a very
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organized fashion on the AP view, the portable AP view, again,
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we see the same or very similar degree of haziness throughout the lungs.
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There seems to be a little bit of Perihilar opacifications bilaterally.
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We don't see large pleural effusions.
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We don't see focal consolidations.
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There's no pneumothorax at all.
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We do see overlying monitoring wires.
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There is a spinal stimulator here. On the bone-enhanced windows,
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you know, it's really more of the same,
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but you get a sense of how busy the interstitial might be.
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And it really does seem to be a much more
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centralized appearance, which can be seen in pulmonary edema.
0:50
And so, you know, I think we were prepared to call this pulmonary edema,
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but also just kind of saying with the caveat,
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you know, if there are signs and symptoms that suggest infection,
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then, you know, please consider that because, again, the overlap can be very real.
1:04
And so this patient did have a CT, and I'll share that with you now.
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And just on the scout view here,
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a very similar appearance of the prominent interstitium, the involvement of the hilar
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region, the spinal stimuli that we saw before in the overlying monitoring wires.
1:21
We'll take a look through.
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Again, we did give contrast here. From the top,
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flying down, just taking a look at the vessels.
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Major vessels are looking good so far.
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The carotid's thyroid gland is normal.
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Three vessel arch.
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Just taking a look at the aorta down
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into the left ventricle, and then back out. Again, looking at the entirety
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of the aorta, there's no dilatation. There is no evidence of dissection.
1:53
And we can see that the celiac and the SMA are patents in their branches and their takeoffs.
2:00
Underneath the diaphragm looks fine.
2:02
As we come back up through the diaphragm,
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I do notice that the esophagus is pretty thickened.
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This is the esophagus here, almost as large as the aorta itself.
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As we move through, and again, we get a peak in the lumen.
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But the esophagus is thickened, almost in its entirety so far.
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And again right here, still pretty thickened. Still pretty thickened.
2:33
So it's diffuse circumferential thickening of the esophagus.
2:37
So an esophagitis certainly could look like this.
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It's not focal. It doesn't look like an apple core lesions.
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So something very focal, like a cancer,
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probably not as much, but something more diffuse-like.
2:50
Inflammation or an infection can certainly look like this.
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Patients with long-standing, really horrible GERD and reflux, could
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certainly have an esophagus that looks like this.
3:00
Take a look at the mediastinum to see if there's any sort of adenopathy.
3:04
And we don't. One thing about the esophagus,
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just to go back, there's this nonspecific calcification that is right here.
3:13
And it's not really clear if we're looking at calcification or we've given contrast.
3:18
It's the same attenuation, it's just a small little bleed. On this one phase,
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it's hard to know.
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We didn't give a non-contrast run, so we're going to differentiate that out.
3:28
So this is something to kind of, you know, check with.
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If this patient is anemic, if this patient has got guaiac positive
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blood or coughing up blood, I would put this in a different category,
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versus this is something that's just calcified.
3:42
Looking at the pulmonary vessels,
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don't see any large evidence of pulmonary embolism.
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The branching vessels, the subsegmental, segmental,
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lobar vessels on the right and the left look normal.
4:01
We see some lymph nodes.
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They don't necessarily look too large in the pretracheal region.
4:06
We got some prominent ones here in the right hilar region. Right along here.
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Let's take a look at the lung windows
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and understand what's happening here, and see if we can put some synthesis
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around this patient's presenting symptoms and what's going on with them.
4:26
So, again, a lot of ground glass opacity is seen bilaterally.
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This is in the more dependent area.
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So sometimes at atelectasis. You might say, 'Yeah, this is atelectasis.'
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But if we move beyond some of those
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dependent areas, we're seeing very similar findings.
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We're going to see it here in, again,
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some of the dependent areas, but also in some of the non-dependent areas.
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So I would not attribute all of this
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stuff, this ground glass opacity, to just atelectasis, all these ground glass.
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So this is like a multifocal ground glass opacity.
5:01
A lot of the ground glass opacities...
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Some of them are beginning to consolidate
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into more noduler forms, and are a little bit more dense than just the ground glass.
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Again, we don't see large effusion.
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We don't see pneumothorax.
5:15
I don't see a dominant mass or even a nodule.
5:20
In a patient with esophagitis, you know, could some of this have been aspiration?
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The distribution is not great.
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It's not just in the dependent areas.
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I don't see anything in the airways,
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but certainly, some sort of inflammatory or infectious process I would consider.
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So when we go back to where we started
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on the x-ray, this is a situation where you have an x-ray that, you know, certainly could have
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fit really nicely into the pulmonary edema pattern.
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But remember that some of that overlap
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between pulmonary edema, and then some of that atypical infection and/or
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inflammation, can be determined or differentiated on CT.
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So give your clinicians a call and try to figure out what's going on.
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If there are signs and symptoms
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that suggest infection or inflammation, the CT may be helpful.
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If it's a straightforward,
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you know, this is a patient who's got a history of edema, or we think this is just
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pulmonary edema, the findings may be adequate on x-ray.
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A lot of the information here is around what's going on with the patient, and then
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being able to actively decide what's the appropriate next clinical imaging step.
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