Interactive Transcript
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One of the entities that one may be called upon to evaluate
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the patient for in the spine is a spinal cord infarct.
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These are incredibly uncommon and cause
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a lot of anxiety because, frankly,
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our pulse sequences to evaluate the spinal
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cord for infarction as opposed to myelitis
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have not yet reached mainstream as far as their quality.
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This is a diffusion tensor imaging of the spinal cord
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with the ADC map and T2-weighted scan.
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Most institutions and private practices probably do not have
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good pulse sequences for the evaluation for spinal cord
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infarct with diffusion weighted imaging
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or diffusion tensor imaging.
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Here you can see on the diffusion tensor imaging, B 1000
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map that there is bright signal intensity
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in the spinal cord at the
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C7-T1 level, which corresponds,
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although not a very good quality image, with
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dark signal intensity on the ADC map.
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Now,
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this would be the imaging findings that we would worry
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about for spinal cord infarct. A word about this case,
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you notice that there is decreased caliber to the
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spinal cord through this area of infarction,
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which is verified on the sagittal STIR image.
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The sagittal STIR image does not show bright signal intensity
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in this spinal cord. So initially, my evaluation said,
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well, this is an old injury because we have myelomalacia, indicative
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of an older injury because the caliber of the spinal cord is
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decreased. However, given the diffusion weighted scan,
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I had to worry about a superimposed infarction on
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a previously injured spinal cord.
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In point of fact.
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This patient had acute symptoms.
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So this is a little bit of a complicated case,
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but suffice it to say that diffusion weighted imaging is
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going to be our best means for identifying a spinal cord
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infarct in those very rare instances in which you're called
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upon to evaluate that in the emergency department.
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This was an individual who had acute onset of paraparesis
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and the CT scan of the cervical spine was unremarkable.
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So this is what we would call the SCIWORA,
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the cervical cord injury without radiographic abnormality.
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On the MRI scan, however,
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we see that from C4 through the upper C7 level,
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we have abnormal signal intensity in the spinal cord and
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you have surveyed ill-defined area of contrast enhancement.
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So in this individual with an expanded spinal
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cord and the abnormal signal and
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faint enhancement,
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you most likely be concerned about a myelitis or,
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in the appropriate age group, a demyelinating disorder.
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In this situation,
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I would be descriptive about the abnormality and recommend that
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there be correlation with CSF sampling in order to determine
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whether or not there is an infectious markers in the CSF,
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elevated white blood count,
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as well as looking for demyelinating markers in the
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cerebrospinal fluid. Well, this is a little bit of a trick case.
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This patient's symptoms started abruptly,
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and in the situation where you have abrupt onset of the
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patient's symptoms, you have to consider a spinal cord infarct.
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This is, in fact,
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the same case as the previous one that I just demonstrated,
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showing abnormal restricted diffusion.
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This was the acute episode where the diffusion weighted
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scan was not performed to identify a cord infarct.
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But on the follow up examination, we did the DWI and
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the cord was still bright in signal intensity on DWI,
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although it did show the myelomalacic change. So, vast
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majority of cases, we think of infectious or noninfectious
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myelitis. We think of demyelinating disorder.
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In this case, a longitudinally,
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extensive transverse myelitis or longitudinally extensive lesion,
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possibly from demyelination or inflammation.
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And in that rare instance, a spinal cord infarct.
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