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Wk 1, Case 5 - Review

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Okay, let's stay with our same theme.

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So, usually with a hyperextension injury,

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we often see fractures of the anterior

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condyles and the anterior tibia.

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Now, for those of you that are new to MRI,

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T2 spin echo imaging,

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long TR, long T imaging,

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not very good for looking

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at bone unless you fat suppress.

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We do have a beautiful view of our intact linear,

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ACL.

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There's our more globular-looking PCL.

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On the medial side, we've got the MCL,

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predominantly the tibial collateral component.

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Around back,

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that becomes the oblique popliteal ligament.

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We've got the popliteus tendon.

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We've got the fibular collateral ligament.

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We've got the biceps femoris tendon.

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Then we also have the neurovascular bundle,

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including the tibial nerve.

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Taking off from it,

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the common perineal nerve.

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We should really have about a two thirds,

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one third relationship between the lateral

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facet and the medial facet.

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We do.

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It should be smooth and non undulated.

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It is.

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We should have a trochlear groove.

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We do.

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And we should inspect

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the medial patellofemoral ligament

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and see its continuity with its crossing

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of the adductor tubercle region.

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We do.

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So, so far, we've come up with very little.

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Let's look at the sagittal and stay

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with our continued search pattern.

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On the lateral side,

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we have the popliteus origin and tendon.

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This time,

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we do have an intact popliteofibular ligament.

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We have a nice, clean arcuate.

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Let's go over to the medial side.

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The meniscocapsular reflection is normal.

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The meniscus is normal.

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We have a posterior triangle

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and an anterior triangle.

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On the lateral side,

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the triangles don't hang together

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as in discoid lateral meniscus.

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They separate very nicely.

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We've got open femoral growth plates

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starting to close.

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The tibial growth plate is closing.

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That's a little premature for a 15-year-old,

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so he's not going to grow a lot more.

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And then, we've got a bone injury.

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So, let's talk about bone injuries.

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The lowest grade of bone injury is a contusion.

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Now,

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don't confuse a contusion with that giant

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impact fracture I showed you earlier.

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In a contusion, especially a low grade contusion,

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you can see the architecture and the weight

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bearing trabecular through the contusion.

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When you have an impact fracture,

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all you see are these little speckles of fat.

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The fat basically implodes.

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So, this is different.

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This is a much lower grade injury

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than we saw before.

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And then, you have to start to look for lines.

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And we do have a discrete line right here.

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So we have a fracture.

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It communicates with the cortex.

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It breaks the cortex.

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You can see it going right through the cortex,

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but that is unlikely on a planar image to be seen.

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So, fractures that are dominant in the medullary cavity,

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the enchondral cavity,

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the spongy bone cavity.

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And I call them enchondral bone fractures.

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I'll often refer to them as microtubecular

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fractures, or enchondral bone fractures,

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unlikely to be seen on conventional radiography.

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Putting the clinician on notice that the plane film

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in their office is likely to be negative.

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But it's still a fracture.

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These do very well.

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They don't take as long as these obvious

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transcortical fractures that we see on x-ray.

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Even though the cortex is involved,

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it's invisible on x-ray.

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They usually get better in about three to four weeks.

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But you have to use the fracture word.

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I don't mind if you say microtubecular fracture.

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I don't mind if you say enchondral, spongy,

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or medullary bone fracture.

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You say where it is,

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you say how big it is,

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and then you use all your conventional

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radiographic tools.

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Gapping, none.

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Depression, none.

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Angulation, none.

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Comminution, none,

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and so on.

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This is an isolated finding

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in this part of the knee.

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There is one other incidental

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finding in this child.

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He's got some swelling of the proximal patellar

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tendon at the tenuous margin.

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And this would be jumper's knee.

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So, your conclusion would read

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hyperextension related,

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intramedullary bone fracture

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or microtrabecular fracture without complication,

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comminution or depression.

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Period.

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Next paragraph.

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Proximal patellar tendon

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inflammation consistent with mild jumpers knee.

Report

Patient History
Left lateral knee pain in a 15-year-old boy who sustained a hyperextension injury while playing football 2 weeks prior.

Findings
Menisci:

Medial Meniscus: Intact.

Lateral Meniscus: Intact.

Ligaments:

Anterior Cruciate Ligament: Intact.

Posterior Cruciate Ligament: Intact.

Medial Collateral Ligament: Minimally swollen.

Lateral Collateral Ligament: Intact.

Posterolateral Corner Structures: Intact.

Posteromedial Corner Structures: Intact.

Extensor Mechanism:

Patellar Tendon: Focal fusiform thickening and increased intrasubstance signal of the proximal patellar tendon deep fibers, consistent with mild proximal patellar tendinosis or “jumper’s knee”. Mild hypertrophy of the tibial tuberosity and subtle thickening of the distal patellar tendon. No tendon tear.

Distal Quadriceps Tendon: Intact.

Medial Patellofemoral Ligament: Intact.

Medial and Lateral Patellar Retinacula: Intact.

Hoffa’s Fat Pad: Unremarkable.

Articulations:

Patellofemoral Compartment: Unremarkable.

Medial Compartment: Unremarkable.

Lateral Compartment: Nondisplaced subcortical fracture of the anterolateral femoral condyle as described below. Otherwise unremarkable.

General:

Bones: Nondisplaced subcortical (subchondral/medullary bone) fracture of the anterolateral femoral condyle with marked surrounding bone marrow edema. Location suggests valgus event. Overlying articular cartilage preserved. No physeal involvement. Femoral and fibular physis remain open. Tibial physis closed.

Effusion: None.

Baker’s Cyst: None.

Loose Bodies: None.

Soft tissue and neurovascular: Unremarkable.

Conclusion

1. Nondisplaced subchondral (medullary bone or spongy bone) fracture of the anterolateral femoral condyle epiphysis, with surrounding osteoedema. No physeal involvement, displaced macrofracture, or cortical step-off.

2. No traumatic medial or lateral meniscus tear. No cruciate or collateral ligament injury.

3. Mild jumper’s knee.

Case Discussion

Faculty

Omer Awan, MD, MPH, CIIP

Associate Professor of Radiology

University of Maryland School of Medicine

Stephen J Pomeranz, MD

Chief Medical Officer, ProScan Imaging. Founder, MRI Online

ProScan Imaging

Jenny T Bencardino, MD

Vice-Chair, Academic Affairs Department of Radiology

Montefiore Radiology

Edward Smitaman, MD

Clinical Associate Professor

University of California San Diego

Tags

Musculoskeletal (MSK)

MRI

Knee

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