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Wk 3, Case 1 - Review

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Our next patient is a 17-year-old boy.

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17-year-old boy complaining of the knee

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giving out with medial, lateral,

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and posterior knee pain after playing basketball.

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Query Meniscal tear.

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So, let's get that one cooking here.

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All right, so let's do what we did before.

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We're going for the axial first,

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because that's what comes up first.

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Let's go through our quick search pattern,

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just to get a general feel for what's going on.

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The patella, weird.

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Really strange, right?

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Like American politics.

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Very strange.

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Look at this weird bump.

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Look at how you don't really have a medial facet.

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You don't have a lateral facet.

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You've got a fissure right there.

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You've got intrasubstance tearing

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of the patellar apex.

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You've got very gentle,

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superficial crab meat fissuring of

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the superficial patella.

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And yes, the surgeon will see that

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you've got patellar dysplasia.

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You have trochlear dysplasia.

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Well, you certainly have a pretty deep trochlear groove.

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When the groove gets a little shallow,

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we call that a Dejour A.

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When the groove is very flat, like,

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straight across, we call that trochlear dysplasia,

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Dejour B.

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When the groove has a bump in the middle of it,

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we call that a Dejour C.

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If you can't remember that,

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you can google Dejour or take some notes.

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How about our anterior cruciate?

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Now this time, looking good.

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Nice and straight and linear, and black.

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How about our posterior cruciate ligament?

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Looking also excellent.

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How about our medial collateral ligament?

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Looking excellent.

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Remember in our last case,

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we had some swelling right there?

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Not this time.

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Looking quite good so far.

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How about our lateral collateral ligament?

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Also looking good.

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How about our effusion?

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Much smaller than the last case.

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So we're not thinking violent,

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high grade injury.

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I would call the effusion 1+.

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Trace effusion would be just slightly

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more than 1 by 1 cm fluid.

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If the fluid is in the front and in the

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back but not distending the capsule,

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I'll call that 1+.

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So now, let's go right to our

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water-weighted images.

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We already know we have patellofemoral dysplasia.

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We know we have varying degrees of chondromalasia.

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One deep fissure, grade 3.

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One intrasubstance area of fissuring,

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and then some superficial fraying,

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grade 1,

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superficial grade 3, chondromalatia patella.

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So, the patella is not gliding very nicely

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through the trochlear groove.

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Let's get our water-weighted images up now.

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We'll go right to the mid-coronal plane

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and check out our collaterals.

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And we'll go right to the midline

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and check out our cruciates.

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Anterior cruciate intact.

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Posterior cruciate intact.

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Humphrey in the front.

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Wrisberg in the back.

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Posterior capsule and oblique popliteal ligament.

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Let's go over to the posteromedial meniscus.

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Looks fine.

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Meniscocapsular reflection, fine.

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No swelling.

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There is the posterior oblique ligament

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of the knee right there,

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which has a fiber that goes towards

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the posterior superior meniscus,

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anterior horn, body, posterior horn, all fine.

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Cartilage, all fine.

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Should be.

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It's a kid.

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Let's go to the other side.

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Quickly.

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Quickly, we see popliteus tendon.

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Pop fib ligament, not stretched.

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Posterior capsule and arcuit, nice and straight.

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How about our attachments?

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There's one.

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Meniscopopliteal fascicle looks good.

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Bottom one, looks good.

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Remember the last case?

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They were ruptured.

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This time, we don't have a pivot shift.

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We don't have translation, by the way,

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while you're on the lateral side,

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you can look at the tib fib ligament.

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Now, when I dictate,

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I'll occasionally use templates,

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but not very often.

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I either free dictate or I'll go by compartment.

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Anterior, posterior, medial, lateral, tib fib,

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intraarticular,

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other, and also central.

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I left that one out.

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So, central.

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So, those are usually my dictation styles.

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When do I use a template?

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When there's only one or two things wrong and

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everything else is normal. Otherwise,

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I stay away from templates because the clinicians

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recognize that you're just parroting and they're

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afraid you're not really looking at the case.

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So, here is the coronal.

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We got the MCL with its three layers.

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The superficial crus layer right there.

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I'll put an arrow on it so you can see it a little better,

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layer one.

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The tibial collateral ligament, layer two.

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And then, it's hard to see layer three

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because there isn't much fluid in there.

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It's the meniscocapsular attachments and the capsule.

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All looking good.

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Collaterals look good.

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Center of the knee looks good.

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Spines look good.

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Central cartilage looks good.

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Medial compartment, cartilage normal.

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Lateral compartment, cartilage normal.

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Shape and conformity, normal on the lateral side.

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A little bit abnormal on the medial side.

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What's abnormal about the shape and conformity?

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Look at it for 5 seconds, see if you can spot it.

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Wasn't in the report.

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Right there.

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See how there's this funny little dippity do

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in the femoral condylar cartilage?

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And the cartilage is a little swollen

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and has a slight signal alteration.

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Let's call up the sagittal T1.

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No bone abnormalities.

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Menisci, we've covered.

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Growth plate, open on the femur,

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just about closed on the tibia.

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And look at the prefemoral fat pad.

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Indurated.

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Look at all stuff right here.

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How about the infrapatellar fat pad?

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Indurated.

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That means this child is having friction on

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patellar glide, and look at the sclerosis of the patella.

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So, this child has patellar dysplasia,

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patellofemoral maltracking

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fat pad impingement and fibrosis or cicatrisation

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chondromalasia patella.

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And that is the case.

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Are there any questions about this one?

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There is an effusion, obviously,

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and that effusion is all generated

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by patellofemoral maltracking,

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the most common cause of knee pain in an

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atraumatic knee in somebody under age 20,

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Patellofemoral maltracking.

Report

Patient History
17 year old boy complaining of the knee “giving out” and medial, lateral and posterior knee pain after playing basketball. Query meniscal tear.

Findings
Menisci:

Medial Meniscus: Intact.

Lateral Meniscus: Intact.

Ligaments:

Anterior Cruciate Ligament: Intact.

Posterior Cruciate Ligament: Intact.

Medial Collateral Ligament: Intact.

Lateral Collateral Ligament: Intact.

Posterolateral Corner Structures: Intact.

Posteromedial Corner Structures: Intact.

Extensor Mechanism:

Patellar Tendon: Intact.

Distal Quadriceps Tendon: Intact.

Medial Patellofemoral Ligament: Intact.

Medial and Lateral Patellar Retinacula: Intact.

Hoffa’s Fat Pad: Focal superolateral Hoffa’s fat pad edema consistent with patellar tendon-lateral femoral condyle friction syndrome related to patellofemoral maltracking.

Articulations:

Patellofemoral Compartment: Dysplastic patella with short medial facet and an elongated concave odd facet that is partially covered with cartilage. Low-grade trochlear dysplasia with slightly insufficient trochlear groove (Dejour type A). Mild patella Alta with an Insall-Salvati ratio of 1.5. Normal TT-TG distance measuring 1.1cm. A full-thickness chondral defect at the patellar ridge and lateral patellar facet. No underlying chondral plate penetration or subchondral edema.

Medial Compartment: Old healed osteochondritis dissecans involving the posterolateral aspect of the medial femoral condyle. Overlying cartilage slightly thickened but intact.

Lateral Compartment: Normal.

General:

Bones: Mild stress-related osteoedema or contusion involving the mid patella. Incidental mildly dysplastic wide intercondylar notch.

Effusion: Small suprapatellar effusion.

Baker’s Cyst: None.

Loose Bodies: 5 mm chondral body within the lateral aspect of the suprapatellar recess effusion. A possible 3 mm chondral body within the knee joint effusion between the medial facet and the medial trochlea inferiorly. Both chondral fragments are likely to arise from the lateral patella chondral defects

Soft tissue and Neurovascular: Normal.

Conclusion
1.Full-thickness chondral defects/fractures at the patellar ridge and lateral patellar facet without underlying subchondral plate penetration. 5 mm and 3 mm chondral fragments within the superolateral joint space effusion and adjacent to the medial facet patellofemoral articulation.

2.Dysplasia: Chronic patellofemoral maltracking on a background of low-grade trochlear dysplasia (Dejour A) and patellar dysplasia with evidence for patellar tendon-lateral femoral condyle friction syndrome.

3.Old healed osteochondritis dissecans medial femoral condyle as described above.

4.No internal derangement or meniscus tear.

Case Discussion

Faculty

Omer Awan, MD, MPH, CIIP

Associate Professor of Radiology

University of Maryland School of Medicine

Stephen J Pomeranz, MD

Chief Medical Officer, ProScan Imaging. Founder, MRI Online

ProScan Imaging

Jenny T Bencardino, MD

Vice-Chair, Academic Affairs Department of Radiology

Montefiore Radiology

Edward Smitaman, MD

Clinical Associate Professor

University of California San Diego

Tags

Musculoskeletal (MSK)

MRI

Knee

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