Interactive Transcript
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Okay,
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the next case is a 59-year-old man
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with severe pain, discoloration, swelling,
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and weakness of the left knee after
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sustaining an injury at work.
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The primary findings,
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some of you suggested
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was a rupture of the ACL,
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a rupture of the patellar ligament.
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Secondary findings were complex tears
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of the medial and lateral meniscus.
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Tertiary finding,
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complete tear of the lateral collateral ligament.
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So, let's go with that for now.
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That's not necessarily the answer,
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but that's what many of you did say.
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So, let's begin with
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the axial projection,
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which is where I usually start,
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and there is very extensive swelling.
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Here's the helmet shape of the patella.
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There are some spurs here, medially and laterally.
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I don't use the term osteoarthritis unless
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I have spurring.
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And I do have spurring.
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While we're coursing through the imaging,
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on the medial side,
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we can see the posterior cruciate ligament.
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On the lateral side,
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we can see some swelling in the neighborhood
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of the linear anterior cruciate ligament.
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I'll also use this projection to look for any
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encroachment on the neurovascular bundle
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to look for phlebitis,
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or what I call pseudo-phlebitis,
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where there is a Baker's cyst that's
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pressing on the popliteal vein.
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Okay, now that we've looked at the axial,
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we know we have a very swollen anterior knee.
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Let's turn to the sagittal projection,
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and we'll take them two up.
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So on the left,
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we have a water-weighted image
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with excellent fat suppression,
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a very nice TE close to around 40.
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And let's tackle the patellar tendon first.
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Once again,
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we see osteoarthritis of the patella
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with extensive spurring.
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I use this projection to look at the trochlea.
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The trochlea is abnormal.
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There's an erosion.
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There's a subchondral spur right there,
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a very small one.
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And the obvious finding is rupture of the patellar tendon.
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So, we would describe where it is.
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It's mid substance.
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We would describe the gap.
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You see it extremely well on the
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sagittal T2 weighted imaging.
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And that's going to be a major
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finding for the case.
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Now,
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for those of you that thought there was an ACL,
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anterior cruciate ligament tear,
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a couple of teaching points,
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most patients, not all, but most,
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like 90%,
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are going to have some form
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of pivot shift bone pattern.
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And what does that consist of?
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An anterolateral femoral fracture,
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a posterolateral tibial chip fracture,
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posteromedial tibial chip fracture.
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This patient has none of those,
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so that should be a concern for you.
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Second, the tibia should translate anteriorly.
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In other words,
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the tibia should translate this way,
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not this way.
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So the tibia should translate to the front.
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That is not happening.
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In fact, the tibia is translating posteriorly.
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It's sitting a little bit back.
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If we draw a perpendicular line along the back
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of the femoral condyle,
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in multiple locations,
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as we scroll across,
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the tibia is sagging the other way.
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So, we have what's known as the tibial sag sign.
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So, for those of you that said
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there was a PCL tear,
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you'd be right.
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There is a PCL tear.
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Now, unlike the ACL,
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the PCL does not usually transect.
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It usually tears interstitially.
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This way maintains its shape
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but does not maintain its signal.
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Sometimes it'll take a piece of bone with it
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and avulse from the base of the PCL.
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That's about 10% of PCL tears.
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Another way to corroborate your thought regarding
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the PCL tear is to look at the coronal.
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You find the PCL, which is medial.
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That's medial.
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That's lateral.
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The PCL is filled with blood.
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There it is.
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There.
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There.
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And it makes what I call an archery target sign.
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Now, admittedly,
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the ACL is extensively swollen
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and a little hard to see,
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but you go back to the sagittal T2 weighted image,
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and even though the ACL
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is very thick and irregular,
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there still is a linear structure.
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It is surrounded by scar tissue.
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So if you said chronic ACL tear,
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scarred ACL,
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I wouldn't argue with that.
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But there's no acute ACL tear,
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and there's no signs of ACL deficiency.
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As we scroll along,
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you can also see there is tibiofibular osteoarthritis
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in keeping with our theme of osteoarthritis.
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Let's go back and discuss our osteoarthritis.
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We do have OA,
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and I will Kellgren Lawrence grade
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my osteoarthritis, one through four.
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I would urge you to google that,
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since it's a plain film classification system
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that we extrapolate to MRI.
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We also have a penetrating erosion,
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weight bearing erosion in the
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medial femoral condyle.
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You are correct in that we have meniscal tears.
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They are predominantly chronic,
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degenerative meniscus tears.
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One medially,
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you can see right here.
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The meniscus is small,
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consistent with DJD and OA.
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The meniscal root is truncated on both sides.
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Consistent, in this case,
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with DJD and OA.
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And then when we look at the lateral meniscus,
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it's a little beat up.
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All of this stuff is chronic.
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The menisci are a little bit extruded.
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They're a little bit malformed.
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They're a little bit small.
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They have some inner third cleavage type signal.
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The medial meniscus is really macerated here.
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So, this is all chronic meniscus pathology.
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There's bone on bone on the medial side.
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There's eburnation and sclerosis.
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So the overriding theme here is OA,
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and with Kellgren Lawrence graded.
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The patient has an acute PCL tear.
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The patient has notch impingement syndrome
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with a heavily scarred knee notch.
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The main finding,
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the first finding in the conclusion should read
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patellar tendon rupture
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with 3-4 cm of diastasis.
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Anything else that is worth commenting on here?
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Give it some thought
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as we kind of scroll around.
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There's some generalized muscular atrophy.
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You can see especially on the lateral side.
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Look how much fatty metamorphosis and replacement
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we have in the lateral compartment.
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And the patient, as we said,
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has a PCL deficient knee.
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Now, this is a gradient echo image.
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In this particular case, it doesn't add much.
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And I think we'll move on from this case.
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I don't have anything else to add.
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There is an effusion,
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and there's extensive prepatellar swelling.
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Okay.
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