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Wk 3, Case 4 - Review

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Okay,

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the next case is a 59-year-old man

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with severe pain, discoloration, swelling,

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and weakness of the left knee after

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sustaining an injury at work.

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The primary findings,

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some of you suggested

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was a rupture of the ACL,

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a rupture of the patellar ligament.

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Secondary findings were complex tears

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of the medial and lateral meniscus.

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Tertiary finding,

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complete tear of the lateral collateral ligament.

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So, let's go with that for now.

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That's not necessarily the answer,

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but that's what many of you did say.

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So, let's begin with

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the axial projection,

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which is where I usually start,

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and there is very extensive swelling.

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Here's the helmet shape of the patella.

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There are some spurs here, medially and laterally.

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I don't use the term osteoarthritis unless

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I have spurring.

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And I do have spurring.

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While we're coursing through the imaging,

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on the medial side,

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we can see the posterior cruciate ligament.

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On the lateral side,

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we can see some swelling in the neighborhood

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of the linear anterior cruciate ligament.

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I'll also use this projection to look for any

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encroachment on the neurovascular bundle

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to look for phlebitis,

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or what I call pseudo-phlebitis,

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where there is a Baker's cyst that's

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pressing on the popliteal vein.

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Okay, now that we've looked at the axial,

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we know we have a very swollen anterior knee.

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Let's turn to the sagittal projection,

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and we'll take them two up.

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So on the left,

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we have a water-weighted image

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with excellent fat suppression,

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a very nice TE close to around 40.

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And let's tackle the patellar tendon first.

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Once again,

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we see osteoarthritis of the patella

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with extensive spurring.

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I use this projection to look at the trochlea.

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The trochlea is abnormal.

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There's an erosion.

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There's a subchondral spur right there,

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a very small one.

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And the obvious finding is rupture of the patellar tendon.

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So, we would describe where it is.

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It's mid substance.

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We would describe the gap.

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You see it extremely well on the

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sagittal T2 weighted imaging.

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And that's going to be a major

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finding for the case.

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Now,

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for those of you that thought there was an ACL,

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anterior cruciate ligament tear,

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a couple of teaching points,

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most patients, not all, but most,

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like 90%,

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are going to have some form

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of pivot shift bone pattern.

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And what does that consist of?

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An anterolateral femoral fracture,

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a posterolateral tibial chip fracture,

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posteromedial tibial chip fracture.

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This patient has none of those,

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so that should be a concern for you.

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Second, the tibia should translate anteriorly.

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In other words,

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the tibia should translate this way,

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not this way.

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So the tibia should translate to the front.

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That is not happening.

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In fact, the tibia is translating posteriorly.

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It's sitting a little bit back.

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If we draw a perpendicular line along the back

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of the femoral condyle,

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in multiple locations,

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as we scroll across,

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the tibia is sagging the other way.

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So, we have what's known as the tibial sag sign.

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So, for those of you that said

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there was a PCL tear,

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you'd be right.

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There is a PCL tear.

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Now, unlike the ACL,

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the PCL does not usually transect.

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It usually tears interstitially.

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This way maintains its shape

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but does not maintain its signal.

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Sometimes it'll take a piece of bone with it

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and avulse from the base of the PCL.

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That's about 10% of PCL tears.

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Another way to corroborate your thought regarding

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the PCL tear is to look at the coronal.

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You find the PCL, which is medial.

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That's medial.

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That's lateral.

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The PCL is filled with blood.

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There it is.

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There.

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There.

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And it makes what I call an archery target sign.

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Now, admittedly,

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the ACL is extensively swollen

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and a little hard to see,

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but you go back to the sagittal T2 weighted image,

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and even though the ACL

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is very thick and irregular,

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there still is a linear structure.

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It is surrounded by scar tissue.

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So if you said chronic ACL tear,

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scarred ACL,

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I wouldn't argue with that.

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But there's no acute ACL tear,

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and there's no signs of ACL deficiency.

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As we scroll along,

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you can also see there is tibiofibular osteoarthritis

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in keeping with our theme of osteoarthritis.

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Let's go back and discuss our osteoarthritis.

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We do have OA,

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and I will Kellgren Lawrence grade

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my osteoarthritis, one through four.

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I would urge you to google that,

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since it's a plain film classification system

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that we extrapolate to MRI.

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We also have a penetrating erosion,

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weight bearing erosion in the

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medial femoral condyle.

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You are correct in that we have meniscal tears.

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They are predominantly chronic,

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degenerative meniscus tears.

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One medially,

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you can see right here.

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The meniscus is small,

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consistent with DJD and OA.

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The meniscal root is truncated on both sides.

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Consistent, in this case,

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with DJD and OA.

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And then when we look at the lateral meniscus,

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it's a little beat up.

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All of this stuff is chronic.

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The menisci are a little bit extruded.

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They're a little bit malformed.

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They're a little bit small.

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They have some inner third cleavage type signal.

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The medial meniscus is really macerated here.

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So, this is all chronic meniscus pathology.

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There's bone on bone on the medial side.

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There's eburnation and sclerosis.

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So the overriding theme here is OA,

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and with Kellgren Lawrence graded.

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The patient has an acute PCL tear.

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The patient has notch impingement syndrome

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with a heavily scarred knee notch.

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The main finding,

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the first finding in the conclusion should read

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patellar tendon rupture

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with 3-4 cm of diastasis.

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Anything else that is worth commenting on here?

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Give it some thought

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as we kind of scroll around.

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There's some generalized muscular atrophy.

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You can see especially on the lateral side.

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Look how much fatty metamorphosis and replacement

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we have in the lateral compartment.

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And the patient, as we said,

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has a PCL deficient knee.

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Now, this is a gradient echo image.

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In this particular case, it doesn't add much.

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And I think we'll move on from this case.

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I don't have anything else to add.

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There is an effusion,

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and there's extensive prepatellar swelling.

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Okay.

Report

Patient History
59-year-old man with severe pain, discoloration, swelling and weakness of the left knee after sustaining an injury at work.

Findings

Menisci:

Medial Meniscus: Chronic complex tear with radial and horizontal components, extending from the posterior root to the anterior body horn junction, measuring approximately 4 cm in length. Associated meniscal body failure with slight partial extrusion.

Lateral Meniscus: Chronic complex tear with radial and horizontal cleavage components involving the posterior horn and body, measuring approximately 3 cm in length. Chronic fraying of the inner edge of the meniscal body. Associated meniscal failure with slight partial extrusion of the body.

Ligaments:

Anterior Cruciate Ligament: Thickened ACL with chronic myxoid degeneration and florid notch synovitis.

Posterior Cruciate Ligament: Expanded mid to proximal PCL with intrasubstance hyperintensity, consistent with chronic tear and hypertrophic myxoid change. Posterior tibial translation, consistent with PCL deficiency.

Medial Collateral Ligament: Intact. Low-grade inflammation and periligamentous edema adjacent to the proximal portion of the tibial collateral ligament, consistent with a low-grade sprain and reactive TCL bursitis.

Lateral Collateral Ligament: Intact.

Posterolateral Corner Structures: Intact.

Posteromedial Corner Structures: Intact.

Extensor Mechanism:

Patellar Tendon: Acute complete full-thickness rupture of the mid patellar tendon, with a defect measuring 2.5-3.0 cm in length. Prolapse/retraction of the proximal and distal tendon fragments observed. Associated proximal retraction of the patella (acquired acute patella alta with an Insall-Salvati ratio of approximately 2.0). Extensive surrounding edema and marked prepatellar bursal thickening with a small effusion measuring 6.1 cm length x 0.5 cm depth.

Distal Quadriceps Tendon: Intact.

Medial Patellofemoral Ligament: Suspect high-grade injury.

Medial and Lateral Patellar Retinacula: Markedly thickened, lax and edematous bilaterally.

Hoffa’s Fat Pad: Edematous/contused.

Articulations:

Patellofemoral Compartment: Diffuse high-grade chronic patellofemoral chondromalacia, particularly involving the central femoral component. Prominent patellofemoral osteophytosis.

Medial Compartment: Diffuse high-grade medial tibiofemoral chondromalacia with moderate chronic subchondral stress reaction involving the medial aspect of the medial compartment. Prominent marginal osteophytosis.

Lateral Compartment: Diffuse intermediate to high-grade chondromalacia involving the posterior and central weight-bearing surfaces of the lateral femoral condyle. No penetrating osteochondral erosion. Mild patellofemoral osteophytosis.

General:

Bones: Subchondral insufficiency related osteoedema involving the medial aspect of the medial compartment as described above. No micro or macro trabecular fracture. No suspicious os

Effusion: Small knee joint effusion.

Baker’s Cyst: None.

Loose Bodies: None.

Soft tissue and neurovascular structures: Unremarkable.

Conclusion

1.Complete full-thickness mid patellar tendon rupture with retraction and prolapse of the proximal and distal tendon fragments. Acute acquired patella alta (Insall-Salvati ratio 2.0), due to proximal retraction of the patella. Marked adjacent soft tissue edema and reactive prepatellar bursitis/seroma.

2.Medial compartment failure with chronic complex medial meniscus tear involving the posterior horn to body, associated high-grade medial compartment chondromalacia and subchondral insufficiency related osteoedema. Kellgren-Lawrence 3 osteoarthropathy.

3.Early lateral compartment failure with chronic complex tear involving the posterior horn and body lateral meniscus with adjacent intermediate to high-grade chondromalacia involving the weight-bearing surface of the lateral femoral condyle. Kellgren-Lawrence 1-2 osteoarthropathy.

4.Low-grade injury or inflammation involving the proximal portion of the medial collateral ligament complex.

5.Incidental posterior cruciate ligament hypertrophic tear with florid notch synovitis and moderate patellofemoral osteoarthropathy. Resultant PCL deficient “tibial sag sign.”

Case Discussion

Faculty

Omer Awan, MD, MPH, CIIP

Associate Professor of Radiology

University of Maryland School of Medicine

Stephen J Pomeranz, MD

Chief Medical Officer, ProScan Imaging. Founder, MRI Online

ProScan Imaging

Jenny T Bencardino, MD

Vice-Chair, Academic Affairs Department of Radiology

Montefiore Radiology

Edward Smitaman, MD

Clinical Associate Professor

University of California San Diego

Tags

Musculoskeletal (MSK)

MRI

Knee

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