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Wk 3, Case 5 - Review

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15-year-old boy,

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Salter-Harris injury.

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Question mark.

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Football injury, two days ago.

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Pain and swelling in the left knee.

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Evaluate for meniscus or ACL tear.

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Okay, let's stay consistent.

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Let's go right to the axial.

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We're drilling through the axial because

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that's what came up first.

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Let's make it a little bigger.

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The patella is drifting a little bit.

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Don't like that, especially in a child.

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So, the child's going to have some

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patellofemoral maltracking

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because the child isn't even standing up.

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The knee isn't flexed,

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so they probably have some element of maltracking.

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It's quite a bit of swelling anteromedially.

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Let's check out the TTTG distance,

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just for giggles.

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So, there's the trough of the trochlea.

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Vikram had a good question earlier

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about measurements.

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This is the one measurement I would do in a child.

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So, let's go right over to the tibial tubercle.

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And our measurement is

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1.4 getting up there.

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So, the TTTG distance is at the upper limits of normal.

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How about our ACL?

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Nice and straight and black.

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Although, there's this funny little ridge

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in the femoral notch,

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which may weigh heavily when the

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patient is 40years of age.

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That can produce a problem for the ACL.

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There's some subtle notch stenosis anteriorly.

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The ACL doesn't like that,

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so that could be a cause of an

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ACL injury later on in life.

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There's the PCL.

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Seems a little bit dusky right there.

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We'll check it out in another projection.

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Here's our MCL.

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Looking good.

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Here's our LCL.

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Looking good.

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Let's go down to our tibial tubercle again.

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A lot of swelling right there.

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That should not be there.

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Some irregularity of the anterior surface.

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So they said,

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you know, know evaluate for Salter-Harris injury,

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and they told us what to look for.

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Let's pull down our water weighted images.

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Let's stay with the same approach.

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Let's start with a sagittal posterolateral corner.

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Fine.

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Little bit swollen back there,

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but fine, otherwise.

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Posteromedial corner,

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there's our direct head of the semimembranosus.

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Remember, it was up here in the last case,

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so that one's not pulled off.

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There's our meniscocapsular reflection

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There is no posterior displacement

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of the medial meniscus.

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There's no failure of the brake stop mechanism.

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There's no ramp lesion.

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There's no anterior cruciate ligament rupture.

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Yes,

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it's a little bit,

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a little bit ill defined there.

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We better check it out on the T2

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and make sure it's okay.

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In fact, let's do it right now

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just to be complete.

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And it's intact.

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So, that was a little volume averaging

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phenomenon there.

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And it's a bit swollen, but it's intact.

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There's no abnormal translation,

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so that isn't the problem.

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The PCL is intact.

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It's a little gray because the knee is an extension,

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so the PCL is a little bit wavy.

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We're getting a little artifactual

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magic angle phenomenon.

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Let's return to our water weighted image.

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Our growth plate,

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15 years old,

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we would expect it to be open,

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and it is in the femur.

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Our growth plate in the tibia,

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we would expect it to be open, and it is.

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But look at the difference

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in the two growth plates.

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This one is pretty dark,

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except for this bright area right here.

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There.

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That is the spongiosis zone of the metaphysis.

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Even this right here,

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this is where blood vessels come in.

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Now,

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if you go from the epiphysis to the metaphysis,

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you've got a group of layers.

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You got the germinal layer,

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the proliferative layer, the hypertrophic layer,

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and finally,

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the zone of provisional calcification,

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or ossification,

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which is what you're seeing here.

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So, these are blood vessel loops that create

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this ill defined, waxy,

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wax on, wax off appearance

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of high signal adjacent to the ossified growth plate,

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the ossified zone of the growth plate

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due to blood vessels.

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That is different than this.

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Intrasubstance within the physis,

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it's too bright.

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And then, you also have a little vertical

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signal in the back.

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Right here.

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Right there.

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Let's put up the T1.

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We'll come back to this coronal image in a moment.

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And there's a tremendous amount of swelling here,

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especially as the pes anserine

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wraps around and inserts on the tibia.

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And that is very curious.

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What do you think that might be?

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That is periosteal elevation.

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We have a huge problem here in this child.

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And then, we have that high signal.

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Again, very disparate-looking.

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Spongiosis zone, ill-defined,

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too well-defined,

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too high a signal in the growth plate of the tibia itself.

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Elevation of the periosteum from blood.

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There is subperiosteal hemorrhage.

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One of your next jobs is to make sure

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no periosteum is stuck in there.

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Let's do that.

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Put up the T1

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and let's scroll them together

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at about the same size.

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So, look how much different the growth plate

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of the tibia looks than the femur.

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Too fat,

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too gray,

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too wide.

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Getting wider anteriorly,

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dissecting into the tibial tubercle portion

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of the apophyseal attachment.

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So, it's going all the way from here

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to here,

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to here.

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And there's also involvement of

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the posterior metaphysis.

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So, this is going to be a Salter-Harris injury

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that is higher grade.

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Please mute yourself if you can.

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So, it's not a Salter-Harris one,

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right?

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When you got a metaphyseal fragment,

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it's going to be a Salter-Harris two.

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If it involves the epiphysis,

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it's a Salter-Harris three.

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If it goes all the way through,

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it's a four.

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If it crushes the growth plate,

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it's a five.

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You have to be very careful because these are

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x-ray Salter-Harris designations.

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On MRI,

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you're going to see contusions in everybody.

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So everybody that has a Salter-Harris one,

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they're going to have a contusion.

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That is not the equivalent of a fracture.

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This linear,

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well-defined, vertical area of separation,

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that is a fracture.

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So you must learn to recognize macro fractures,

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which are included in the Salter-Harris grading system

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from microtrabecular injuries,

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which are not included

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in the Salter-Harris grading system.

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So, one of the tip offs that you're dealing with a

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true fracture and not the metaphyseal spongiosa,

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or maybe one growth plate is closed and the other

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one's open, and they look asymmetric,

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is the periosteum.

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Once you have seen periosteal bleeding,

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you know you have a fracture.

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Look at this massive periosteal hemorrhage.

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Right here.

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And also, look right there.

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Look right there.

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That is a piece of periosteum that is dipping

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right into the fracture space.

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Hard to see on the T1 weighted image.

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Let's go back to the coronal image for a minute.

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Now, there are a couple of zones here that are

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probably board-worthy,

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one is called the Latarjet zone,

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and one is called the Ring of La Croix,

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and another one is called the zone of Ranvier.

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If there are any residents on,

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I would google those.

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You'll probably get asked those in your core exam

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or maybe in your second exam.

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They are areas of growth plate anatomy.

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I think that's a little much for us today,

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but all you need to know is there is

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a periosteal sleeve right here,

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and that periosteal sleeve, disrupted.

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Blood underneath, blood on top,

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and there's the piece of periosteum,

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right there,

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that got stuck inside the fracture.

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The menisci were normal.

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The cruciate,

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even though there is this little signal,

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it was volume averaging.

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On the next cut, it's intact.

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So the anterior cruciate ligament was intact.

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There were no other major injuries in this case.

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It was a Salter-Harris two fracture.

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There was periosteal entrapment.

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There was also some injury to the deep fibers of

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the patellar tendon. You can see that right here.

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There is an ununited tibial apophysis.

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So there was pre existing oskid

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schlotters syndrome,

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and the patient had an injury

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to the pests and serenus.

Report

Patient History
15-year-old boy, status post football injury 2 days ago, with pain and swelling in the left knee. Evaluate for meniscus and/or ACL tear.

Findings
Menisci:

Medial Meniscus: Intact.

Lateral Meniscus: Intact.

Ligaments:

Anterior Cruciate Ligament: Intact.

Posterior Cruciate Ligament: Intact.

Medial Collateral Ligament: Intact.

Lateral Collateral Ligament: Intact.

Posterolateral Corner Structures: Intact.

Posteromedial Corner Structures: Intact.

Extensor Mechanism:

Patellar Tendon: Distal deep fiber insertional partial-thickness tear. Extensive periligamentous edema and hemorrhage. Incomplete avulsion of the tibial tuberosity, with elevation/diastasis of the anterior proximal tibial physis. Subperiosteal hematoma at the tibial tuberosity extending into the anterior proximal tibial metaphysis. 0.6 x 0.6 cm corticated ossicle adjacent to the deep fibers of the distal insertional patellar tendon, in the vicinity of the infrapatellar bursa. Reactive infrapatellar bursal thickening without effusion.

Distal Quadriceps Tendon: Intact.

Medial Patellofemoral Ligament: Intact.

Medial and Lateral Patellar Retinacula: Intact.

Hoffa’s Fat Pad: Unremarkable.

Articulations:

Patellofemoral Compartment: Incidental patellar lateralization and tilt. Borderline increased TT-TG distance measuring 1.8 cm. Trochlear dysplasia with hypoplastic medial facet (Dejour C). No intermediate or high-grade patellar trochlear chondromalacia or traumatic osteochondral defect.

Medial Compartment: Unremarkable.

Lateral Compartment: Unremarkable.

General:

Bones: Proximal tibial metaphyseal fracture extending into the physis. No extension into the proximal tibial epiphysis. Mild widening of the anterior proximal tibial physis. Anterior tibial tuberosity subperiosteal stripping and subperiosteal hematoma. Extensive periligamentous and periosteal edema with hemorrhage noted adjacent to the tibial tubercle. No other acute fracture or physeal injury

Effusion: Small reactive suprapatellar effusion/hemarthrosis.

Baker’s Cyst: Multiloculated gastrocnemius semimembranosus bursal cyst measuring 2.1 x 1.8 x 1.1 cm. No dehiscence or rupture. No extension into the popliteus fossa or compression of the neurovascular.

Loose Bodies: None.

Soft tissue and Neurovascular: Diffuse post-traumatic hematoma and edema surrounding the tibial tuberosity/apophysis as described above. Small grade 2 sprain with small intramuscular/myotendinous hematoma at the origin of the tibialis anterior, measuring approximately 9 x 6 mm.

Conclusion

1. Nondisplaced Salter-Harris 2 fracture of posterior proximal tibia.
2. Anterior proximal tibial physeal widening and incomplete tibial apophyseal avulsion (form friste Salter-Harris 1 injury) with adjacent subperiosteal hematoma adjacent to the tibial apophysis and proximal anterior tibial metaphysis.
3. Partial thickness tear distal insertional deep fiber patellar tendon.
4. Small grade 2 strain (small intramuscular hematoma) proximal tibialis anterior origin/myotendinous junction.
5. No ACL injury or meniscal tear.

Case Discussion

Faculty

Omer Awan, MD, MPH, CIIP

Associate Professor of Radiology

University of Maryland School of Medicine

Stephen J Pomeranz, MD

Chief Medical Officer, ProScan Imaging. Founder, MRI Online

ProScan Imaging

Jenny T Bencardino, MD

Vice-Chair, Academic Affairs Department of Radiology

Montefiore Radiology

Edward Smitaman, MD

Clinical Associate Professor

University of California San Diego

Tags

Musculoskeletal (MSK)

MRI

Knee

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