Interactive Transcript
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We're talking PCL in a 54-year-old man
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who's had an unknown mechanism of injury
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but now has knee swelling.
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Let's scroll the axial T2 where
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most people might start,
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and you should be struck by this fluid
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fluid level or blood fluid level,
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which suggests a serious injury
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and most likely a fracture.
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Now, on T2 imaging the signal intensity,
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while very bright for heavily fluid laden
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areas such as blood and fluid or water,
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doesn't do as good a job when you look
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inside ligaments and tendons,
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because acute blood is black.
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Tendons are black, ligaments are black.
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So, in the acute setting,
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unless you've got a lot of fluid and generalized
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swelling, if there's a lot of blood present,
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you could get fooled into thinking
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your structures are intact.
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That's also true chronically on a T2 On a T2
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weighted image, the tendon is going to be black.
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Fibrous tissue is black. Hemocytrin is black.
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You could potentially miss a chronic posterior
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cruciate and even anterior cruciate ligament tear.
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So, buyer beware.
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Combine your T2 with your heavily water weighted,
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fat suppressed pd spur or stir imaging.
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So,
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let's look at our pcl on the sagittal water
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weighted image. And it just looks too thin.
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So, is that atrophy,
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or are we losing a lot of the bulk of the PCL as
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it blends in with the surrounding swollen tissues?
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And the answer comes into the coronal projection.
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You've got to do what intel does.
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Intel inside. You got to look inside the PCL.
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Let's scroll it.
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And inside the PCL is a well defined area of
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hyperintensity right in the center of the archery
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target. You're not volume averaging the fluid,
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the sheath, the swelling,
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and the blood around the pcl,
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you are actually seeing damaged tissue inside
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the PCL. So that's how you do it.
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That's how you affirm the diagnosis of a PCL tear.
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This patient also has a huge Humphrey ligament
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and does not have a ligament of Wrisberg,
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which is a variation and has a very large
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footprint of insertion of the Humphrey and the
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PCL on the femur. But there's so much more.
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We're going to talk about the corners,
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because a corner injury with a PCL injury is a
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much different animal than an isolated PCL injury.
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Now most of you are noticing that the ACL is
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attached to some bone that is no
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longer attached to the tibia.
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So we have an ACL deficient knee
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and a PCL deficient knee,
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and we haven't even gotten to the corners yet,
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but we will.
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Let's have a look at what happens to the knee when
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you lose the PCL and either one of the corners.
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Let's start out with the medial
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corner in the back.
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The medial corner consists of the semimembranosus,
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the posterior oblique ligament of the knee,
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the pol, the OPL, the oblique popliteal ligament,
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the meniscus,
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and the meniscal capsuler attachments.
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If I say it enough times,
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you're going to remember it when you lose those
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structures and you externally rotate the knee.
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Now the tibia will jut forward on
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the medial side excessively.
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You can see it from a side view as well.
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So this is Amri,
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PCL plus posteromedial corner.
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What about PLRI?
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Posterolateral rotatory instability,
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also sometimes called posterolateral recurrent
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instability. Rotatory type when it's missed,
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also in external rotation. But this time,
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instead of the medial tibia jutting forward,
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the posterior instead of the anteromedial
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tibia jutting forward,
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the posterolateral tibia goes backwards.
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So look at it from the back and then
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look at it from the front.
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In external rotation,
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the tibia is dropping back on the lateral side.
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To make matters worse,
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you often lose this ligament right here,
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the fibular collateral ligament, as part of PlRi.
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So you also have val varus instability.
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So the joint opens in a varus pattern,
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so the tibia goes back and the joint opens with
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a combined fibular collateral ligament tear.
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Posterolateral corner injury and a PCL.
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This is a very serious type of injury
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that often requires reconstruction.
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