Interactive Transcript
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Let's take a look at this 12-year-old young man,
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boy, with anterior knee pain
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and open growth plates.
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We have on the left, the T1 fat-weighted image.
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In the middle is a T2 star turbo field echo,
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using a very short TR and TE,
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but with very generous fat suppression.
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And then another water weighted image,
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also gradient echo.
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This one, not as fat-suppressed
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or not fat suppressed.
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So, you can compare these two and sort of get
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a feel for the difference between each.
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If you are a stickler,
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I'll certainly be happy to give you
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a proton density fat suppression.
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Now, I'll blow this one up.
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This is a little more water-weighted than the
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gradient echo that you saw on the far right.
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In fact, it's a lot more water-weighted.
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That standard non fat-suppressed
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gradient echo image,
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clearly not as sensitive in picking up areas of
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marrow edema and soft tissue edema
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as this fat-suppressed PD spur
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and this center image,
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which is a gradient echo with excellent fat suppression.
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Now, there's another caveat here.
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This study was done with a TE of 14
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on the PD spur,
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so they did not actually even maximize
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the water sensitivity of this sequence.
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Because, as many of you know,
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I don't like to do PD fat suppression imaging
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with short TEs.
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I like to use intermediate TEs of 30, 35, or 40
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to bring forth the water signal intensity even further.
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That set aside,
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we've got plenty of water signal intensity present.
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Now, let's bring back our gradient echo.
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Now, when we think about anterior knee pain
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in the young juvenile,
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by far, the most common cause is mild patellar dysplasia
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or mild micro instability
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with patellofemoral maltracking.
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So, what do you see in cases like that?
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Well, you might see the dysplasia
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or the misshapen patella
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or misshapen trochlea,
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which will be a story for another day.
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And we have different discrete shapes that we describe,
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patella magna, pebble patella,
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the berg shapes, or Weinberg shapes,
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one through four, and so on.
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But right now,
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I'd like to just share with you this
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this indurated appearance of the retro patellar fat,
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which is a sign of shearing,
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that there is excessive friction between
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the patella and the tissues around it,
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and it can cause some irritation
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and inflammation of the fat pad
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and can even hypertrophy the fat pad.
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In fact, the fat pad can get so big
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that it prevents a child or young adult from flexing
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and extending the leg.
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And that is called fat pad impingement syndrome.
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And the fat pad may actually have to be
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resected and that is a real entity.
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You're also probably very familiar as imagers
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and orthopedic clinicians with the entity
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of Osgood-Schlatter's syndrome,
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which is an apophysitis.
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It's not just failure of union
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of the tibial apophysis,
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because lots of people have that,
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and they have no swelling, no inflammation,
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they're not symptomatic.
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So, what do I call that?
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Ununited tibial apophysis.
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Pretty simple.
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But when it's inflamed,
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when there's cystic change,
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when there's swelling of the tendon,
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then I'll call it inflammation of the tibial
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apophysis, or tibial apophysitis,
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consistent with clinical Osgood-Schlatter's syndrome.
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The same thing can be said here
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in the inferior aspect of the patella,
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where there is an apophysis that has
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remained unfused in this child,
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that is swollen,
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that involves the patellar tendon.
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So this is known as Sinding-Larsen,
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or Sinding-Larsen-Johansson syndrome.
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With the patellar swelling,
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you might also invoke the clinical
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syndrome of Jumper's knee,
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because these two bony structures are wiggling.
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In other words,
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there's some micro instability between them.
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You are seeing friction induced stress reaction
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in the main patellar body and in this inferior,
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ununited tubercle,
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and it is clearly having an effect on the hoffus space
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and even the suprapatellar fat space.
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Now, another question
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that you're probably asking yourself,
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and you should, is, okay,
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patellar tendon is sick, it's swollen,
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it's inflamed, it's a child,
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but is it torn?
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Should I use the T word?
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And perhaps,
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that's when you turn to the other projections.
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So, let's do that.
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Let's turn to the axial,
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and let's scroll down and see what a healthy
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tendon looks like.
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Nice and black, curved,
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convex forward, and we see the tendon fibrils.
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Those are not tears.
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Those are areas of interdigitation of soft tissue
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with the tendon.
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Just fine.
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They'll become a little more conspicuous
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as the tendon gets swollen.
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So, you're separating out the tendon subunits.
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They're delicate, they're linear,
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they're repetitive.
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None is really brighter than the other.
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Yeah, maybe that one's a little bit brighter.
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I'll give you that, but not enough.
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Let's keep going.
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Now, we're getting into the piece of bone.
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Let's keep going.
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We're in bone.
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Let's keep going.
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e're in bone,
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and now we're seeing the prepatellar plate.
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This certainly is below my threshold
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for reading a tear.
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If you wanted to call it a microtraumatic injury,
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that's fine.
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But essentially,
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this patient does not have a patellar tendon
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macro tear.
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Now, we're not done yet.
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We can look at this tendon in the coronal projection,
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and we should,
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so that we decide whether we're going
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to invoke the T word or not.
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We're not.
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Let's look at the coronal projection.
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Do we see anything that is dominant as a gap,
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that is etched, that is well defined,
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that is fluid-like signal, and the answer is,
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no, not really.
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That is not in the tendon.
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That's the soft tissue swelling.
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When we get back in the tendon,
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things look pretty darn good.
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Yes, there is some swelling right here,
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but that's bone.
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That is a piece of bone.
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That's swelling in the bone fragment.
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But there is no gapping or separation
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of this tendon.
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These fibrillated areas of striation that are
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repetitive, not very bright, very similar,
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they line up all in a row,
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they're redundant,
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that is typical of the tendon fiber subunits
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of an intact patellar tendon.
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Sinding Larsen Johansson Syndrome,
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jumper's knee, intact tendon,
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patellofemoral maltracking
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with retropatellar induration.
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Next.
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