Interactive Transcript
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Here is an unfortunate teenager
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with long-standing chronic disease.
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And that chronic disease is Juvenile
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Idiopathic Arthritis, or JIA.
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Uh, and this is a more advanced case.
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On the left image, I have a fat
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suppressed fluid-sensitive sequence.
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The middle image is a T1 fat
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suppressed post-contrast image.
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And on the right is our Dual
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Echo Steady State, or DES.
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Thin, high-resolution images that are great for
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looking at cartilage and cartilaginous surfaces.
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So let's start with the fluid
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sensitive sequence, STR.
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Here we have what looks like a little
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erosion or a geode in the right femoral head.
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We can already tell that the surface of
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that femoral head is very, very irregular.
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There is an effusion, joint effusion.
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And there's almost bone on bone, at
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least what appears to be bone on bone,
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on the coronal images on this side.
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Looking at the other side, we see similar
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areas of erosion, slightly bigger over here.
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You can actually see a little communication
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of that erosion to the joint space.
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Okay.
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Again, a large amount of effusion,
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irregularity, and maybe even a little bit
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of collapse of that right femoral head.
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Looking at the post-contrast images,
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fat suppressed, so everything that's
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bright here is not going to be
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fluid, it's going to be enhancement.
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So what do we have?
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In the region of the effusion, the bright stuff
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is no longer fluid, but it's the synovium
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that's lining the joint that's lighting up.
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So we have synovial enhancement on
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the right side and on the left side.
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And if you look, those areas that we saw cystic
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areas, on the right side, for example, here,
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there's a little bit of misregistration, but
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this corresponds to this right over here.
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That is enhancement, because some of the
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synovium from the joint can invaginate into
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these geodes, or these little cystic structures.
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This is enhancement.
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A secondary finding of osteoarthritis.
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So the juvenile idiopathic arthritis in
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this person has gotten so bad that you're
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now seeing secondary osteoarthritis.
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Because the final common pathway
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of all joint destruction, joint
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inflammation is osteoarthritis.
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And now we're seeing sequelae of
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osteoarthritis even though this is a JIA kid.
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So geode with synovial
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enhancement and invagination.
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You can see something similar
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on this side over here.
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It's not as impressive, but
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you can see it right over here.
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And looking at other parts again, there's
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a little bit of enhancement in the
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subchondral bone, showing that
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there's some synovial enhancement and, and
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invagination in, even in the subchondral area.
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Again, a large amount of effusion that
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is enhancing involving the synovia.
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Looking at our Dual Echo Steady State,
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which is great for looking at cartilage,
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like I mentioned, look at this very
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irregular surface of that femoral head.
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All this is damage to the cartilage.
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Look how little cartilage is left.
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Now, remember this is actually
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two thicknesses of cartilage.
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You have cartilage on the acetabular surface,
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and you have cartilage in the femoral head.
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It should be much thicker than
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this, much smoother, much rounder.
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It has an abnormal appearance both on this side and
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you can really appreciate it on the left side
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where you can see that irregularity coming or
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pooching in with some flattening even of that
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femoral head. The shape of the femoral head is
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also abnormal. It's not as well rounded anymore.
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There's flattening.
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There's a little bit of collapse.
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There is Acetabular Protrusion, meaning that
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a big portion of that femoral head is
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traveling in this direction. Let me just
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show you what I'm talking about with a pen.
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This femoral head has gone in this direction
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into the acetabulum more than it should.
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So it's protruding into the acetabulum.
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So this is a great example of chronic juvenile
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idiopathic arthritis that still shows active
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inflammation, but also shows chronic changes
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such that osteoarthritis has also developed.
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