Interactive Transcript
0:00
Let's start out with our perfunctory views,
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the ones I love, namely the sagittal T1
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and the sagittal, fat-suppressed, water
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weighted image, which is kind of like
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your search and destroy. I hate to call it
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this, but it's kind of your dummy image.
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Because all you have to do is find the white
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spots, the areas of water, edema, or swelling,
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and we have found a big one right here in the back.
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Now this patient has mid-tailor
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and posterior ankle pain.
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So let's focus on the posterior
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ankle pain differential diagnosis.
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What are some possibilities?
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Could have an achilles problem.
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Could have a calcaneal problem.
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Could have posteromedial impingement.
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Could have posterolateral impingement.
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Could have a problem with
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the flexor hallucis longus.
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Could have a problem with the calcaneus
0:50
itself and the way it's shaped.
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Namely a Haglund deformity.
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Could have a posterior osteochondral defect.
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Or could have a manifestation
0:58
of Tarsal Tunnel Syndrome.
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That's a pretty broad differential diagnosis.
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But we've got everything going on in the
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subtalar space in the posterior ankle joint.
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We've got dogs and cats living together.
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Ten days of darkness, the plague, mass hysteria.
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It's all here on this sagittal view.
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I mean, look at what we've got.
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We have capsular swelling.
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We have this funny-looking protruding bone.
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We have a spur coming up from the calcaneus.
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We even have a body floating in
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the back, surrounded by capsule.
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The remainder of the soft
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tissues are massively swollen.
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And to make matters worse, the posterior
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facet of the subtalar space is sclerotic,
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dark, but also edematous, bright,
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and the joint itself is swollen and bright.
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And then we get into the sinus tarsi canal.
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Things are also still a bit swollen,
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but now we're starting to move away
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from our prime area of pathology.
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Now, we want to make sure that the, the region
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of the tibia and the talus is not involved.
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And while there's a little bit of remodeling
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of the posterior tibia, it looks pretty good.
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And most of the inflammation
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is a little bit lower.
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So now it's time to look at our axial projection.
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Let's do that.
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So, on the left.
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And the red boxing trunks is
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the T1 fat-weighted image.
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And the blue boxing trunks, on the
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right, is the T2-weighted image.
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Now you remember, from prior vignettes that
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you probably have trolled through, that the T2
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weighted image is an okay water-weighted image.
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It's more of a modifier.
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But it does do a very good
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job of showing you Intel.
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In other words, what's inside the
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chip, or what's inside your computer.
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And what's inside this
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collection is some gray stuff.
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That's synovium.
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Well, let's keep going, shall we?
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If we want to see bone, what do we prefer?
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The T1-weighted image, of course.
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And look at the back of that talus.
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It is not normal.
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That's the normal signal intensity of the talus.
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The talus is getting rubbed, and buffed,
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and scuffed, and compressed, and irritated.
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It's unhappy.
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It's got an erosion.
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We have a body floating in the back.
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We've got, here's another body, posteriorly,
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floating even further in the back.
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So a very complex pattern of bone anatomy that
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perhaps is more easily seen and I will float the
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sagittal by you in a minute to try and figure
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out exactly what structures we're looking at.
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But we also have another problem up front that
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we'll save for, for a separate discussion, which
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is a big fibular spur, an extensive swelling in
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the anterolateral fibular gutter, but I'd like
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to concentrate right here on the back for now.
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So one of the critical structures to analyze
3:53
in a patient with this, you know, serious
3:55
problem is the flexor hallucis longus.
3:59
So here is the flexor hallucis right here, and
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it's located between the lateral talus process.
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Here, and the medial talus process, here.
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The lateral talus process is the insertion
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site for the posterior talofibular ligament.
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So, if some of these structures start to encroach
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more medially, they can have a rather dramatic
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effect on the flexor hallucis longus, and we see
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this in one of the subtypes of this condition,
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which is called posterior impingement syndrome.
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So, what are those subtypes?
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What I call ballet dancer impingement,
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and chronic repetitive trauma impingement.
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Now, chronic repetitive trauma impingement
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is probably a little more easily manageable,
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uh, if the ballet dancer has a repetitive
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injury than if she's had an acute traumatic
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event where she's fractured her steator
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process and developed, you know, massive
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amounts of edema and injured the FHL.
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So I would divide these up into basically
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acute toe pointing injuries, And chronic
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toe pointing injuries, the chronic
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ones, are a little bit easier to manage.
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Because the ones that are acute,
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they like to go back to a very acute
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activity, a very serious activity.
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And once this deformity ensues, it's very,
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very hard to return them to their original
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anatomic structure and configuration.
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So let's continue on with this.
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Terrible phenomenon in this
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patient of posterior impingement.
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Got a lot of bone disease, got
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a lot of bones in the back.
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Let's talk about what things can contribute
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to this type of posterior impingement, which
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is the chronic repetitive trauma arthrosis.
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So let's talk about the structure that is
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most often associated with this condition,
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and it is called the os trigonum.
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The, the Os Trigonum is a structure in
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the back, right here, that mineralizes
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at about 7 to 13 years of age, and
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then it fuses at about 1 year of age.
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So it fuses pretty early, but it
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ossifies a little bit later on.
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And when these become really large, and
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they have fused, and they point out,
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they're known as steator processes.
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10 percent of the time, it persists
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as a round, separate structure.
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called the Os Trigonum ossicle.
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That Os Trigonum ossicle, especially if
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it's big, can produce, and is responsible
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for many cases, of posterior impingement
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syndrome in repetitive toe pointing athletes.
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So we've got the separate ossicle, this is not.
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We've got the attached ossicle
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with the big process, this is.
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We've also got a big process, a big steator
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process, that sticks out that breaks right
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here, that's called a shepherd's fracture.
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That's another cause.
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We've also got big posterior tuberosities
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and spurs coming up from below.
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Oh, we have that too.
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So we have this big process.
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It's not fractured, but it's thinking about it.
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We have this coming up from below.
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And finally, if you have a really
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downslope tibia that comes down with a
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hook, it too can contribute to a fracture.
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Posterior Impingement Syndrome.
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We don't have that.
7:27
And finally, the last thing
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that contributes to it, Bodies.
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Do we have that?
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You bet we do.
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There's a body right there.
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So out of all the things I've mentioned,
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we've got at least three of them.
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We've got a body, we've got this weird upward
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spur coming up from the calcaneus, crunching
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the structures in the back, and finally
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we have a large, lateral, tailor process.
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And you can see that funny
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looking process right here.
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And it's starting to break.
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There's a little bit of irregularity right there.
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So it's starting to break off and eventually
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will mature into a Shepard's fracture.
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Now, let's talk about, you know, the
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differential diagnostic considerations.
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And I want to focus on one in particular.
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One that, that often goes, uh,
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unrecognized and is not commonly discussed.
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And is commonly misnamed.
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So a lot of your colleagues will look at the
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Medial malleolus after an ankle sprain, and
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it'll be swollen, and they'll call it a tear.
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Now when you invert your foot, basically what
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you're doing is, you're doing this, right?
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When you have an ankle
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sprain, you have an inversion.
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So you're inverting it this way.
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So what happens is this bone swings up
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against your deltoid, and it crushes it.
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And it gets swollen and contused.
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Now, you don't want to read that as a tear.
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We also don't repair those.
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But here's a problem.
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If it stays irritated and swollen and becomes
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synovitic, that swelling then starts to dissect
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posteriorly towards the medial process right here.
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So this area then becomes swollen
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and dissects towards that area.
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Here's your deltoid right here.
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This is your deltoid.
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So it's gonna swing back and it's
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gonna start to affect the structures
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near the medial tailor process.
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And what's near it?
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The FHL.
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That's called Medial Posterior
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Impingement Syndrome.
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But we're dealing with Lateral
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Posterior Impingement Syndrome.
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So either one can come over and start to crunch
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and crush and entrap the FHL, producing a
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major problem for any toe pointing athlete.
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But this one is Posterolateral
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Impingement Syndrome.
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In keeping with our differential
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diagnostic list, The Achilles is fine.
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There was no coalition.
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The patient had nothing going
10:00
on in the tarsal tunnel space.
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And there was not a posterior
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osteocondyl defect of the Taylor dome.
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So we've eliminated a lot of the other
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differential diagnostic considerations.
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And we make the diagnosis of
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posterolateral impingement syndrome.
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