Interactive Transcript
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This is a 56-year-old male that has
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osteoarthritis, instability, innumerable
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complaints, and also a question of deficiency
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or tear of the posterior tibial tendon.
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So let's talk about this tendon for a minute,
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which arises from the tibia and fibula and
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inserts on the navicular, on the medial
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cuneiform, and then has small areas of
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insertion on virtually every bone of the
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midfoot and even on the base of the metatarsals.
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This is a foot structure that is
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involved in the eversion process.
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Inversion is usually formed by the fibularis
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brevis and longus and is its antagonist.
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So now let's move on to the case itself.
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Let's scroll the case and
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the findings are massive.
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It's one of these overwhelming cases.
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Where do you start?
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What's your search pattern?
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And I would suggest that with an arthritic
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case like this, with effusions everywhere,
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that you start out simple with just a skeleton.
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25 00:01:05,200 --> 00:01:07,060 So you go up and down on the skeleton.
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You can either go axial or sagittal.
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I'm using axial simply because I know that
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the posterior tibial tendon is at risk and
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it's easier to see axial than sagittal.
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Most of the time, I begin sagittal.
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And I always begin with a
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fat weighted and a water weighted.
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Unfortunately, I don't have
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any fat weighted axials.
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So I'm going with my T2 and my
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heavily fat-suppressed PD SPIR.
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So let's scroll and look
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at the bones for arthritis.
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Yes, there's osteoarthritis.
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Yes, there is spurring.
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But not horrible.
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And there are some changes in the
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bone that consist of erosions.
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What am I going to do next?
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I'm going to look at the ligamentous
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anatomy because I have a history
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of instability and osteoarthritis.
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And I cannot really find an
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anterior talofibular ligament.
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It's gone and that's why there's instability.
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If there's no anterior talofibular ligament
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and it looks that bad, the odds of having
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an intact calcaneofibular ligament, low.
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In fact, it stops right there.
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It's too thick.
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It's torn.
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How about the posterior talofibular ligament?
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That's part of my search pattern.
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You already know from prior discussion that
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that never tears unless you have an ankle
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dislocation, which there's no history of.
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And here are some fibers of the
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posterior talofibular ligament.
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Let's go higher into the high ankle,
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where we see a deficient anterior tib-fib
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ligament, maybe a little widening of the
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syndesmosis, and some irregularity and ill
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definition of the posterior tib-fib ligament.
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But it certainly doesn't look acute.
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So I'm covering my ligaments.
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I don't really care that much
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at this point about the deltoid.
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Although the deltoid is swollen,
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we hardly ever repair it.
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We'll come back to it.
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The retinacula.
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I would do a quick check.
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Extensor.
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Lateral.
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Medial.
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And also see what's going on with the posterior
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medial and posterior lateral retinacula.
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All of which are slightly wavy.
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thickened, and somewhat redundant. Now, coming back to my skeleton, even
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though I have lots of spurs, I am paying
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very careful attention to certain ones,
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like the spurs in the retrofibular groove,
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because I want to know if I'm at risk for
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having a peroneus longus and brevis tear, and I do have one.
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I have a peroneus brevis split tear evolving
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right here in this spurred irregular groove.
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What's another groovy thing
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that I'm interested in?
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I'm interested in the posteromedial and lateral
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processes and their groove, which is narrowed.
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It's dysplastic and the spurs that come off them.
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In other words, the hallucis isn't
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sitting very nicely in the groove.
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Is the hallucis torn?
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Well, no.
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There's the hallucis tendon and muscle.
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It's not torn.
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Is it normal?
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No, it's a little gray on the PD SPIR.
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It's getting a little grayer.
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It's getting a little grayer and a little
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fibrillated and a little irregular.
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It's sitting behind these two small
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excrescences, but no, it's not torn.
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Okay?
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Let's move on to the rest of
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the structures on that side.
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We've already talked briefly
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about the brevis and longus.
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We didn't track them all the way because
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that's not what this case is about.
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But we would have in the proper situation.
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And it would have gone achilles, one,
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120 00:04:32,972 --> 00:04:37,690 peroneus brevis and longus, two, three, Tom, Dick, and Harry.
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Tom is nowhere to be found.
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And then, Tom, Harry, Dick,
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and the peroneus tertius.
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So, one, three, four.
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Where there should be three, there's only two.
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We've got a problem.
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And no, that is not the posterior tibial tendon.
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That is a vessel.
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So let's go up high so we
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can find the three tendons.
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And there they are.
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There is Tom.
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Tom is huge.
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There's Dick.
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And here is Harry.
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Now let's follow Tom.
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Tom is a big fat thing right here.
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There's Tom.
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It's getting thinner.
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There's Tom.
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There's Tom sitting behind this irregular
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tibial groove, which should not have a spur.
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So now we got three groovy areas to check out.
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The retrofibular groove, the groove between the
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posteromedial, and lateral talar processes,
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and then a groove that does not belong along
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the medial free edge of the tibia, which is
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associated with posterior tibial tendon tears.
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150 00:05:45,905 --> 00:05:47,055 Let's keep going, shall we?
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Wait a minute.
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Where did Tom go?
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There's Tom.
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Tom's getting smaller.
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Tom's getting smaller.
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Tom is gone.
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There's a big hole.
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So Tom's retracted way, way up.
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Now let's see if we can find Tom down.
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So, no Tom.
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There's Dick.
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There's Harry.
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There's the neurovascular bundle.
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Let's keep going, shall we?
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Our superficial ligament looks terrible.
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It's thickened.
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It's irregular.
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It's got a tear in it.
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But we need to find Tom.
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Run the hunt
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for red October for Tom.
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No Tom, just fluid.
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We're gonna find Tom, right at the very end.
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Here comes Tom, right as it
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inserts on the navicular.
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And you can see one of the slips
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going off towards the cuneiforms.
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There are innumerable slips.
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Because there's so much fluid in the ankle
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joint, dissecting down into the patent,
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we can see Henry's master knot, and the knot
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consists of the crossing of the flexor digitorum and hallucis.
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They come together, and sometimes this
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big sheath that's distended full of
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fluid is confused with a ganglion.
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It's simply fluid coming
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down from the ankle joint,
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offloading into this space.
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We also have fluid around the peroneus
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longus, which is going plantar,
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and the peroneus brevis, which is intact.
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We also have a large, irregular,
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degenerated tear with intrasubstance
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delamination of the tibialis anterior.
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So, lots of things are going on in this
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ankle as we do our complete axial survey.
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Let's put up the sagittal, shall we?
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Because we need to see where the torn end is.
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Now, that is the stump right there of
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what's left of Tom, the tibialis anterior.
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There's Harry in the front.
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You could easily come up with a
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pitfall where you confuse the flexor
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digitorum with the tibialis posterior.
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That would be a mistake.
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Where is the tibialis posterior?
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Up here.
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So there is a tremendous amount of
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separation between that location and
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this location, which you have to measure.
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You should also measure above,
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either the anterior or posterior funiculus or interfunicular
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groove of the tibia
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so the clinician knows exactly where to go fishing
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for the proximal end of the tibialis posterior.
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Now let's keep scrolling a sagittal and we
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see our large pseudomass and Henry's master
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knot tracking down from the joint space.
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We're also doing our inspection again of bones.
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There are innumerable erosions.
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We're not going to spend a tremendous amount
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of time talking about the arthritis other than
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the massive anterior spur that is contributing
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to anterior tibiotalar impingement syndrome.
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But the coronal fat suppression water-weighted
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image has limited value in this particular case.
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It will show you the very swollen irregular
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deltoid and all the osteoedema of the medial
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malleolus and the lateral malleolus from
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arthrosis, reactive edema, and micro instability.
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But I'm more interested in where is, where is Tom?
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And that's a tough one to solve.
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There's Tom right there.
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That's the stump of Tom.
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There's nothing going down beyond that.
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So, if we want to find the proximal end,
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we'd measure from some key landmark to
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this location to assist the surgeon.
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Now while we're at it, we also found some bodies,
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some synovial metaplastic bodies, that were
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hanging around in the flexor hallucis sheath.
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So we're doing a capsular check.
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We're looking for other bodies.
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There's another body.
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And we could keep going.
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So we're going to search the entire
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articular surface for erosions,
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and bodies, and hyperplasia, and metaplasia.
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That's all part of the search pattern.
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We're going to look for soft tissue masses,
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innumerable ones, all consisting of capsular
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distension, bursal distension, and stuff
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that is located within these structures.
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We're also going to do an overall bone survey
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again in the other projection, just to make sure
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there isn't a specific pattern of bone injury.
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And then finally, in a case like this,
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although not relevant, we would be
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checking for the neurovascular bundle,
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especially in the posterior tibial region,
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to make sure that the patient doesn't have
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a mass there that's encroaching on the
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neurovascular bundle structures, and there isn't.
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Searching for classic tarsal tunnel syndrome.
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And we also alluded to our search for anterior
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and posterior impingement, which this patient has.
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And finally on our way out, we'll check the
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plantar fascia, which is probably the best
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thing that this patient has going for them.
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An example of massive
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posterior tibial tendon tear.
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Small peroneus brevis tear, small tibialis
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anterior tendon tear, anterior impingement
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syndrome, tibiotalar posterior impingement
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syndrome, tibiotalar capsulitis,
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synovial metaplasia, osteoarthritis, and
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the list, and hits, just keep on coming.
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