Interactive Transcript
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This is a 42-year-old woman who's got, uh, wrist pain.
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You're shown two images, a coronal T1 fat suppression
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image with IV contrast and a short axis or axial
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T1 fat suppression image with contrast as well.
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Let's look at the relationship of the
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radius and ulna to compare their variants.
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When the ulna is...Start over.
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9 00:00:33,520 --> 00:00:33,880 Ready.
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This is a 42-year-old woman with wrist pain.
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You're shown a coronal fat-suppressed
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T1 contrast-enhanced image, and the same sequence
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used in the short axis or axial projection.
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Now, as I scroll the coronal projection, there
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are innumerable findings, but one that probably
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strikes you right away is the relationship
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of the distal ulna to the distal radius.
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Now, if we were to perform an X-ray with the
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strict variance position, we would do so with
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the shoulder abducted 90 degrees, the elbow
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flexed 90 degrees, the forearm neutral, and
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the hand aligned with the forearm axis.
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You wouldn't have any flexion or extension of the hand.
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It'd be neutral and it'd kind of be
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at the side, almost with the thumb up.
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And this is the typical X-ray position
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that we acquire for assessing variance.
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And there should be an equidistant position
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of the ulna relative to the radius.
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In other words, the ulna should
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be over here, not back here.
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So we would describe this as negative ulnar variance.
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And this distance would be,
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say, 5, 6, 7, 8 millimeters.
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Now, normally when measured in the proper hand
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surgery, radiographic position, that variance or
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disparity between the distal margin of the radius
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and ulna should be about a millimeter or less.
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With hand clenching, the ulna juts forward.
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Now, it's kind of strange in men, the variability
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of this distance is less than it is in women.
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As one ages, the variability of these two distances
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Now, if the ulna were to project distally
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to the free edge of the radius, we would
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describe that as positive ulnar variance.
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Now in MRI, because we're not in the exact same
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position that we use for measuring on X-ray, we
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don't say positive variance or negative variance.
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We say positive ulnar variance posture, or in
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this case, negative ulnar variance posture.
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And we look for the indirect sequelae,
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or signs, of negative ulnar variance.
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Which are, by the way, Kienböck's disease,
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which is known as lunate necrosis.
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The patient doesn't have it.
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Let's scroll.
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There is no necrosis or collapse.
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Another potential complication would
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be radioulnar impingement or abutment.
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We start to get some undulation and
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irregularity under inflammation in this area.
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And you can see that there is some remodeling
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occurring along the ulnar aspect of the radius.
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And then the third one, which is not very well
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advertised, but is present in this case, is because
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the ulna is disparately proximal to the radius, it puts
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a traction on the peripheral and dorsal attachments
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of the TFCC, especially the attachments to the
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styloid and to the extensor carpi ulnaris subsheath.
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These areas become inflamed due to this traction.
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Because again, when the ulna is short, there
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is a taut pressure placed on these attachments.
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And that is what you're seeing here.
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You're seeing diffuse inflammatory enhancement
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of the ulnar side of the capsule, of the
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dorsal radioulnar mechanism, and of the
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extensor carpi ulnaris subsheath, right there.
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Now let's look in the axial projection for a minute.
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This would be at the level of the TFCC.
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This is the area of the volar radioulnar ligament.
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This is the area of the dorsal radioulnar ligament.
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We don't see them properly, but what we
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do see is a congruent relationship.
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Not too much dorsal floating, and not too much
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palmar floating of the ulnar relative to the radius.
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Why is that important?
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Because in people with negative ulnar
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variance, you will get radial ulnar
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incongruity in the anteroposterior direction.
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So you might have to perform neutral,
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pronated, and supinated short axis views
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to make sure that that is not happening.
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And you can do that on CT.
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So those are the three major sequelae
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of negative ulnar variance posture.
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Radial ulnar abutment or impingement,
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we have some of that here.
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Although not much inflammation, but some remodeling.
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Kienböck's disease or lunate
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necrosis, which we don't have.
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And sprain, chronic strain of the ulnar capsule
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and its attachments, which we absolutely do have
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and are seen in the form of diffuse swelling.
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Along the ulnar capsule, surrounding
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the ulnomeniscus homolog, and involving
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the extensor carpi ulnaris subsheath.
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Subsequently, these patients often will evolve
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into extensor carpi ulnaris disease with micro
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instability and small tears, intrasubstance is
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how they start, and eventually the ECU will split.
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Now, one other caveat.
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Anomalies play a big role in some
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cases of negative ulnar variance.
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You're all familiar with the Madelung
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or reverse Madelung deformity, which can
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produce positive or negative ulnar variance.
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You can also get changes in variance from
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fractures up by the elbow or by the wrist,
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premature closure of the growth plates.
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But another important variable that is associated with
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changes in variance, especially negative variance,
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is an enlarged, irregular, or dysplastic styloid.
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Perhaps that's the fourth thing on the
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checklist that I should have added to
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those big three that I initially gave you.
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And when there is negative ulnar variance, the styloid
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tends to be rather large and dysplastic looking.
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And it may abut against the triquetrum,
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especially in ulnar deviation, and cause notching
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or pseudocyst formation of the triquetrum.
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That is not the case here.
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This is a normal notch.
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It's well corticated, there's no edema of
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the triquetrum, all the swelling is in the
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proximal soft tissues, and it's related to
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stretching and a taut appearance or a taut
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mechanism of these attachments in patients who
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are pronating and supinating and hand gripping.
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So this concludes our discussion
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of negative ulnar variance.
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I've given you four main potential
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sequelae of negative variance to look for.
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Kienböck's disease, radial ulnar impingement,
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inflammation or strain/sprain of
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the peripheral and dorsal attachments,
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and dysplasia of the ulnar styloid.
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