Interactive Transcript
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Dr. Stern, we're here with a 29-year-old
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3 00:00:02,940 --> 00:00:05,490 professional athlete, throwing athlete.
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And, uh, this patient experienced,
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I believe, a deceleration injury.
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I think his hand got caught on somebody
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else's helmet in the middle of a throw.
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And, um, when he walked off the field, his thumb
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could just kind of droop down to the side, although
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he wasn't in much discomfort at the time.
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We've got, uh, coronal, what I call
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UCL views with the proper angulation.
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Fat-suppressed, uh, T1 anatomic image, and
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then the axial, uh, water-fat-suppressed image.
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And he's got a very complex, high-grade
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UCL. Just to give the audience the anatomy,
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here's the adductor aponeurosis, and you can see how
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it's dipping underneath the ulnar collateral ligament.
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Now, an inexperienced reader might think that
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that was the adductor aponeurosis because of
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its slope and kind of blend these two together,
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but this is actually the UCL that normally
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would exist under here and attached there,
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and now it's sitting on top of the aponeurosis.
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You can see it on the, uh, coronal T1.
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Looks like kind of a yo-yo on
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a string sign, if you will.
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And then in the short axis view, there's this extra
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nubbin of tissue that normally wouldn't be there.
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That's the UCL that is sticking out
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dorsally because these things retract.
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They fold backwards on themselves, uh, this way.
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And then they kind of lob themselves on top of the
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adductor aponeurosis, which I've depicted here.
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And then this is the UCL sitting well on top of
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the adductor aponeurosis, which is underneath it.
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So, a couple of issues on this case.
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Obvious, high-grade, stenotypic, trapped UCL.
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What matters in the acute stage?
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And what determines what kind of
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procedure you're going to do here?
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Because I think this patient
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has a pretty shredded UCL.
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Yeah, this is, uh, even an orthopedic
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surgeon could make the diagnosis here.
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The ligament is completely blown out, uh,
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and as you said, it flips over backwards.
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And it's actually, the reason it looks like it's on
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top of the adductor aponeurosis, it's actually flipped
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over, approximately 180 degrees, and it's
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actually just proximal to the adductor aponeurosis.
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At any rate, the question is
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whether or not, exactly as you've drawn,
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the question is whether or not
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this can be repaired.
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And in these cases, from a clinical
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standpoint, you really want to get
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to the surgery as quickly as you can.
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Sometimes the ligament is so
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damaged, hemorrhagic, and edematous
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that you cannot reattach it.
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In that case, you would have to substitute
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it with some type of tendon graft.
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Again, usually the palmaris longus
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is our go-to graft situation.
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If you can reattach it, fine.
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You can put it down with a suture anchor.
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There are a number of different techniques.
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This was another patient of mine,
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and in this case, the patient
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elected to finish out the season and we did a
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reconstruction with a palmaris
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graft, and he returned the following
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season to have a successful career.
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He did well.
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Now, for the audience,
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this was his non-throwing hand.
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Had it been his throwing hand, there's no
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way that he would have been able to continue.
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He wouldn't have been able to hold the ball.
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Yeah, that's correct, absolutely correct.
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83 00:03:42,184 --> 00:03:44,514 But it was his non-throwing hand, and with a lot
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of, uh, taping and splinting, uh, he was able to,
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uh, handle the ball and finish out the season.
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Question for you.
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If you, if you avulse off a piece of the
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base, how does that change the surgery?
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Well, in gen, uh, no, it
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absolutely will change the surgery.
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If you think you can fix, uh, a fracture
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fragment, which is, uh, certainly a variable
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size, as you mentioned earlier, it can be very
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tiny to, uh, you know, uh, fairly substantial.
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We would always try to fix bone to
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bone rather than tendon to bone.
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So if we can preserve that fracture fragment and
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put it down, uh, by a variety of techniques, screws,
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pins, uh, pull-out buttons, et cetera, more than the
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radiologist would need to know, we would do that.
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Uh, sometimes, uh, however, the fragment is
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small enough that it ends up being excised and
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then you do a ligament to bone reattachment to
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the volar ulnar base of the proximal phalanx.
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In terms of timing, you mentioned, you
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mentioned this when we were off camera.
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You said, you know, it's important to fix these
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in the acute stage if you can, uh, functionally
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based on, you know, the patient's job, etc.
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But one of the reasons you want to fix them early is
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because if you let them go, that changes the procedure.
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Could you explain that a little bit?
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Over time, the, uh, the tissue, the
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ligamentous tissue, the collagen just deteriorates
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and becomes very edematous, uh, and almost, I
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wouldn't say friable, but it's edematous
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and you just cannot get a reliable repair.
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So, uh, ideally, in an injury like this, if you
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could repair this, and I've done this, day one
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or day two, that would be the ideal situation.
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But over time, the collagen kind of coalesces, becomes,
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for lack of a better word, mushy, and it precludes any
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type of acute reattachment, necessitating a free graft.
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And this patient had, as you said, a free graft.
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He's also got two of the other
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findings that you alluded to.
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In prior vignettes, you can see that
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he is extended during this exam.
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It's probably an almost full
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extension, as much as he can get.
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And if you look at the cortex of the metacarpal,
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and then you look at the cortex of the
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proximal phalanx, they're offset a little bit.
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So he does have a little bit of radial deviation.
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And he also has that sag sign that we talked about
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before, where the proximal phalanx is moving forward
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due to loss of the secondary stabilizers.
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Um, any other comments before we get off this case?
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No, I, I think you've described it quite well.
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Great.
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Dr. P and Dr. Stern, out.
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