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Case: Focus on the Subacromial Arch

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Okay, we're talking about the six key components of the

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rotator cuff complex: the supraspinatus,

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the infraspinatus, the teres minor, the subscapularis,

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the capsule, and the coracohumeral ligament.

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We've shown you, in other vignettes, injuries of all of

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them and how to assess them in different projections.

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But I want to focus just for a moment on the

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surrounding anatomy.

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Specifically, I want to focus on the subacromial arch.

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This case of a massive rotator cuff tear,

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there is no supraspinatus.

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There is massive retraction with the fibers seen medially,

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was instigated, at least in part,

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by this funny-looking acromion.

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The acromion has been a subject of

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much debate and much discussion.

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There's even a categorization of the acromion called

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the Bigliani subtypes of the acromion.

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I hardly ever use that.

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What I do use is the proper descriptors.

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I'd like to see if the acromion is downsloping

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in the coronal projection.

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There is a sloping angle,

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but that's a little bit beyond the discussion today.

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How often do I use it? Never.

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I rarely measure things,

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but I do look at them.

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So downsloping in the coronal projection is a little

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different than downsloping in the sagittal projection,

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but I pay attention to both.

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Then there is the shape of the acromion.

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Very often,

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people that have anatomic impingement have an acromion

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that has a little shelf on it, that looks a little

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bit like the end of a telephone receiver.

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You'll also hear this called a keel-like acromion

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I often refer to it as a telephone receiver acromion.

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Now, when you're younger,

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the acromion plays a scant role in impingement,

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unless you have a humeral head that won't stay down,

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in other words,

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it starts floating up when you pick your arm up.

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In other words, there's microinstability.

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The humeral head doesn't rotate

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in the center of the cup.

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And if you pick your arm up and the humeral head

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goes up, what does it bump into? Your acromion.

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As you get a little bit older, the acromion remodels,

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and it becomes like this,

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keel-like.

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And now you've got a self-perpetuating prophecy.

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The humeral head is up. The acromion is in the way.

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The acromion further damages the rotator cuff.

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The humeral head comes up higher, and eventually,

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and unfortunately, they meet bone to bone.

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There is another structure that participates in this

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process, and that is the coracoacromial ligament.

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The coracoacromial ligament comes off the acromion

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and heads towards the coracoid.

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Now, this patient's coracoacromial ligament

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is not that well seen.

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It's right here

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and it's not that thick. It's a little bit thick,

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but most of the time,

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if you went to decide whether the ligament or the bone

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is a bigger contributor to anatomic encroachment,

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it's more often the ligament than the bone.

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It's about 60-40, 70-30, but either one can do it,

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and sometimes it is both. In this case,

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it's more bony than it is ligamentous.

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Let's take a quick look at the sagittal projection for a moment

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and see what our acromion looks like

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in the sagittal projection.

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When we say that the acromion is downsloping,

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here's what we mean.

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Here's our humeral head,

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not a very good drawing of it.

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And if our acromion is tilted down from posterocraniad

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to anterocaudad, it's positively downsloping.

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If it's straight in line with the humeral head,

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something like this, then it's neutral.

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And if it's angled up, then it's negative slope.

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We don't pay too much attention to that.

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We are more interested in positive sloping,

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but especially

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whether the acromion has a hook

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or a spur associated with it.

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And it often looks quite different in the sagittal projection.

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It often looks like it has a little snout,

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and that snout is where the coracoacromial ligament attaches.

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So you have a fat acromion that has a snout or a spur.

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And if it's downsloping sagittally,

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which this one really is not.

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And in the coronal projection,

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you have a kell-like or telephone receiver acromion,

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which in this case it is.

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And if you have a thick coracoacromial ligament,

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which in this case it's a little bit thickened,

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but not very much.

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All of those components are part of the analysis of

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impingement syndrome and contributors

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to rotator cuff pathology.

Report

Description

Faculty

Stephen J Pomeranz, MD

Chief Medical Officer, ProScan Imaging. Founder, MRI Online

ProScan Imaging

Tags

Trauma

Shoulder

Musculoskeletal (MSK)

MRI

Bone & Soft Tissues

Acquired/Developmental

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