Interactive Transcript
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Okay, this is a 17-year-old young man that
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has had a history of a shoulder dislocation.
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I've got up for you an axial T1-weighted non-contrast
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image and a coronal arthrogram with gadolinium,
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dilute gadolinium, placed into the shoulder joint.
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If we start with the coronal, the finding is rather subtle.
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And normally you'll see a little
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bit of undulation in the glenoid.
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There's normally a little notch here called the
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notch of Oskar and you may see some thinning of
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cartilage here called the bare area of the glenoid.
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But the fact that we have this undulation that's so
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isolated down low in a 17-year-old, that's bothersome.
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So let's go over to the cross-referenced image here.
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And this is at the same level.
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When you look really carefully, and we'll scroll a
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couple slices, this is the main slice right there.
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There is a focal defect at the glenoid base, right
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at the site of hyaline cartilage and where it ends.
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To compound matters, the tissues in
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front of it are tumefactive looking.
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They're mass-like looking.
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They're gray and intermediate signal.
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So perhaps there's some inflammatory tissue,
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or scar tissue, or reparative tissue.
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It really isn't clear yet what's happening.
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Now there is an entity called a
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glom, a glenolabral ovoid mass.
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And that, that is when you have a
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detached labrum that migrates superiorly.
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And frequently it takes capsule with it.
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to produce a, a piece of labrum that, uh, is sitting
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there with some swirling capsular tissue around it and
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some inflammation to create a, a somewhat circular or
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oval mass in the mid to upper quadrant of the shoulder.
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But here we're in the lower quadrant.
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And so I think it's time to pull
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out, uh, our axial arthrogram.
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Now I personally don't do arthrograms
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very frequently in the shoulder.
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Only when I have, uh, an instability situation or
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some other clinical situation that is very concrete
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with a strong history and I have not found the answer.
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And that results maybe in one out of a hundred
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really needing an arthrogram or even less.
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My number one indication for doing the
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arthrogram is the clinician wants it.
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And it's too fatiguing to make that argument not to do it.
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So I just do it.
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Uh, but, but for the most part, I try not to.
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And as I build trust with my clinicians over time, and
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they, they learn that the answers we're giving are correct,
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more and more they, They stopped requesting the arthrogram.
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But this one has one.
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And this is a patient that's had a dislocation.
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And it's interesting.
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What we were seeing as a tumefactive area of
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intermediate signal on T1, let's bring it back
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for a moment, really distends out very nicely.
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So what you were looking at was collapsed
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glenohumeral ligamentous tissue.
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And maybe it distends a little too much.
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You know, maybe there is plastic deformation
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of the capsule that has led to, or is a
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result of this patient's prior dislocation.
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Now, not affecting the labrum, but right at
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its base, involving the hyaline cartilage.
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See, this is hyaline cartilage in gray right here.
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Dressed in gray.
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Dressed in black is the cortex, and
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subcortical bone that's a little sclerotic.
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Let me make that even a little bigger for you.
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So there's your hyaline cartilage, and there is your
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glenoid articulation rim divot, or guard lesion.
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Now, if this lesion had decided to partially enter,
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the anterior labrum, then we would convert it to
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a GLAD lesion, a glenolabral articular disruption.
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Guard lesions can occur in dislocations or in
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minor injuries, whereas GLAD lesions tend to
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occur more exclusively in, in minor lesions.
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And as you know, type Bankart lesions.
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Those occur in flat out dislocations, and
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they are discussed in another vignette.
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The Perthes lesion, which is discussed in another
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vignette, is a tear in which we have a partial rim tear.
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But it does not interrupt the periosteum.
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So the periosteum stays intact and attached.
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And within that tear, we get accumulation of
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some inflammatory material within the labrum.
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Let's see if my colors will work here.
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They do.
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And you may see it puff up, but it
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stops right where the periosteum begins.
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And this Perthes lesion can occur as a result of an
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acute injury or a subacute repetitive microtrauma.
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So this is an example of the guard lesion.
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Let's look at the, sorry, let's look at the
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coronal projection and the sagittal projection
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to see if we have anything even remotely
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resembling a Hill-Sachs lesion, and we do.
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So this corroborates our diagnosis of a former dislocation.
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Now this is a little bit, uh, more apical than I
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would expect in the 12 o'clock position, and maybe
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this isn't related to the dislocation, but this
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certainly is in a classic or typical location.
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Supralateral, about 5 millimeters
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off the apex of the humeral head.
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It is a Hill-Sachs equivalent type lesion, and it
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corroborates and supports our diagnosis of a guard
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lesion with plastic deformation of the capsule as a
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result of a prior dislocation in a 17-year-old boy.
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Let's move on, shall we?
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