Interactive Transcript
0:01
This is a 49-year-old man.
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He's a weightlifter.
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Congratulations at age 49.
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He's very muscular, and you should notice that right away.
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No fatty infiltration in large
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muscles, but he's not 20 years old.
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And what's the difference?
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Well, the difference is obvious, but
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20-year-olds have acute dislocations.
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The incidence of acute collision type unidirectional
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dislocation diminishes with age, even in weightlifters.
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Even in people that are active.
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People that are active that are 40, 50, 60 years old tend
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to have remote bouts of macro unidirectional instability,
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or not, but bouts of micro-instability of varying degrees.
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So the odds of them having, at age 50 or
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60, multi-directional micro-instability
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compared to the 20-year-old are much higher.
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And that's important because there are two basic
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spectrums, or two basic continuums of instability.
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At one end of the spectrum is the major
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traumatic dislocation first-time event.
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And at the other end, there's either no
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antecedent trauma or remote antecedent trauma.
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And that's what we have here.
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Now how do you know whether you've had remote
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antecedent trauma when you were 24 years old?
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Well certainly, history might help.
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Or you can simply look at the architecture.
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Let's look at the architecture on this T1-weighted image.
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T1-weighted image.
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Nice and round.
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When we get to the back of the shoulder
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though, look at the top of the humeral head.
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It looks a little bit serrated.
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A little bit flattened.
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And that's, this suggests to us that, actually
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this is anterior, this suggests to us that the
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patient has had supra-inferior instability.
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In other words, that the humeral head has
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at some point been bounded up and down.
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Yes, this is anterior because there is the coracoid.
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So you can see I made a quick
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mistake there, even I make mistakes.
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We also notice the glenoid remodeling.
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Anteroinferiorly, the glenoid is serrated, it's irregular,
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there are some spurs, there's a small spur off the humerus.
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Another sign in a weightlifter who's active,
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who's a strong guy, of micro-instability.
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So now let's look at the sequela of micro
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instability, which will include what I call
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partial labral tears, less profound labral tears.
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And what do they consist of?
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Glad lesions, guard lesions, fissures, cysts, slap lesions.
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Let's have a look.
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Let's take all three water-weighted images.
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I'm gonna put up the sagittal water-weighted image on
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the far right, the coronal water-weighted image in the
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middle, and the axial water-weighted image on the left.
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Let's start out with the axial, because
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that's what you're most comfortable with.
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And we go all the way to the back, and
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we see a little bit of signal here.
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Is that fluid or is that a tear?
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No, it's outside the labrum.
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It's fluid.
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All right.
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Let's keep going, shall we?
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Let's go forward on our coronal.
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We see recesses between individual structures, like the
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biceps and the rotator cuff and caracohumeral ligament.
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So we've got this laminar pattern, but no slap lesions.
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As we come down, things look a little bit busy.
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I think it's time to go to the axial projection.
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Let's scroll our way down.
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There's our biceps up high.
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We go down, we see labrum.
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And any fissures and separations of the labrum
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from the glenoid up high should go away.
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Do they?
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No, they don't.
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In fact, one in the substance of
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the labrum becomes more prominent.
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There's swelling of the hyaline cartilage at its base.
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Not surprising, because we already
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know there's glenohumeral arthropathy.
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And our lesion has busted out anteriorly.
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As a small anterior paralabral cyst.
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Is that all?
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No, that's not all.
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We've got a small little erosion in the glenoid.
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take a look at the posterior labrum.
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Is it normal?
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No, it's not normal.
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It's not detached, but it's got evidence of
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an old, healed, scarred, partial rim tear.
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I'm gonna make it bigger, so you can see it.
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Right there.
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How do you know it's not hyaline cartilage?
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Because hyaline cartilage doesn't go all the way out.
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Let's take a look at the capsule.
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Capsule's right there.
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It comes around.
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It's got a little bit of synovial thickening in it.
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It's redundant.
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So there's a little bit of capsular plasticity.
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There's an old chronic labral tear.
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There's a little bit of arthrosis in the inferior quadrant.
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We clearly have a tear in the labrum.
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Perhaps not through and through.
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Part of it's healed.
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Part of it's cystic.
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Part of it's busting out anteriorly.
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It's creating.
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Inflammation in multiple directions of the shoulder.
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The rotator cuff is intact and this is a weightlifter
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with multi-directional micro-instability as
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opposed to the single collision event athlete.
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Let's have a look at another one, shall we?
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