Interactive Transcript
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This was a 56-year-old who was thrown from
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the motorcycle after a motor vehicle collision.
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The CT scan is quite dramatic
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in that the patient has
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a large amount of blood that has collected.
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If we start from superiorly,
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we see hemorrhage along the intrahemispheric fissure
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and a small subdural collection in a parafossean
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location. We also see that there is a large collection
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of irregular blood products
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which is seen overlying the
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right frontal and parietal
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lobes and extends to the temporal region.
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The collection we could measure,
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but it's clearly greater than 10mm
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in width and therefore as a surgical candidate
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actually measures 17mm.
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When we look to measure the degree of midline shift
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at the level of the septum plus C idem,
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we see that there is 10.8 mm of midline shift
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greater than the 5 mm that would normally be a
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surgical criterion,
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and these 2 would
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suggest that the patient requires
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no surgical intervention.
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You notice also that there is
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tissue which appears to be extending.
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The gray matter appears to be extending from
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right to left
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across the edge of the Falx
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over here, just too much gray matter here,
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and this represents the Subfalcine herniation
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that is due to the large collection.
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When we look further inferiorly,
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we see that there is tissue
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which looks unlike normal cerebellar vermis
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that is crossing the edge of the tentorium.
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The tentorial edge
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is seen
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on the left side here
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and on the right side incompletely. Seen
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because there appears to be brain tissue
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which is extending across from the right to the left.
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You note that the
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cistern on the right side
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has been displaced and the midbrain
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this is the cerebral aqueduct.
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The midbrain appears to be shifted from right to left.
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This is the area of the uncus or the amygdala
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and what?
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Whereas the normal amygdala is ending over here on the
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left side. On the right side, we see tissue
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which is effacing the cistern of the
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periaqueductal region,
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and so we have evidence of uncal herniation,
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probably evidence of
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transtentorial herniation of temporal lobe structures.
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This patient
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developed 3rd nerve palsy,
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the so-called blown pupil.
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So a blown pupil is largely secondary to compression
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of the oculomotor nerve,
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the 3rd nerve
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secondary to herniation of tissue
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into the perimesencephalic cistern
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where the 3rd nerve will
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be coursing, and this often happens with uncal herniation
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because of the numerous
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neurologic complaints, the
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size of the subdural hematoma
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and the degree of midline shift
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the patient underwent,
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nor surgical evacuation of the subdural hematoma.
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This is the post-operative evaluation.
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You see that in point of fact
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the patient had a craniotomy.
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By craniotomy, we mean that
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portion of the calvarium
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was removed and not replaced,
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so craniotomy. We replaced the bone.
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Craniectomy we've removed the bone
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and this allows the brain to expand outward,
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rather than causing midline shift and right-left shift
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you notice that the patient still.
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Has an element of midline shift,
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so as we measure
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here and to the septum plus idum
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it now measures approximately 5mm,
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whereas previously it was approximately 11mm.
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So they have relieved
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the midline shift.
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We no longer see
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very much in a way of subfalcine and
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herniation of tissue
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a little bit right over here
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of the falx tent,
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and as we look down at the
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uncus,
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there's dramatic improvement here. Here is the uncus,
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the mediotemporal
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lobe on the left side.
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Here is now that tentorial edge
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with no apparent impression
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of the uncus, maybe a little bit over here,
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but that's not that much
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different from the contralateral side,
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so the right side a little bit
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and we have a more normal appearance to the
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cerebellar tissue,
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and along the tentorial edge
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patient has a collection of blood along the posterior
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portion of the occipital lobe
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as well as on the posterior falx,
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but at this juncture along with that interhemispheric
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at this juncture, we'd say looks pretty good
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post-operative evaluation.
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This is 24 hours later.
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24 hours later
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we are seeing low-density areas within the anterior
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medial right frontal lobe.
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Those with super good eyes might
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also note a low-density area in the
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posterior portion of the mediotemporal
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on the right side. Compared with the left side
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patient still has the craniotomy
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and the midline shift is still resolving
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not much in a way of uncal herniation,
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so let's go back
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to the previous. Scan
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one day
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earlier. That was the immediate post-operative scan.
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Now in retrospect
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we might wonder
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about the low-density in the
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medial frontal lobe on the right side
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as well as this area of low density
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more posteriorly,
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what has happened?
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Well, we get more clarity if we look at the MRI scan
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and this MRI scan
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was performed
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48 hours after surgery.
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This is the diffusion-weighted scan.
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48 hours after surgery.
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Remember for diffusion-weight imaging. It's our best
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sequence to look for infarcts.
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What do we see?
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We see high signal intensity in the medial
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temporal lobe
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extending to involve the occipital cortex
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on the right side
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as we proceed further superiorly,
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we are seeing
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areas on the posterior cingulum and parietal lobe
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as well as the medial frontal lobe of infarction.
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There's also an area over here' that will deal with
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that is really related to susceptibility artifact
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what happened?
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This is an example of one of the complications of
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head trauma that leads to a secondary injury.
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This patient's subfalcine herniation
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led to compromise of the anterior cerebral artery,
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leading to infarction
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of the medial aspect of the right frontal lobe.
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This patient's
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uncal herniation and transtentorial herniation
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led to compromise of the posterior cerebral artery
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as well as potentially the anterior choroidal artery
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leading to infarction of the medio temporal lobe
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as well as the occipital lobe
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despite almost
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immediate evacuation of this collection.
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So infarctions of
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occipital lobe, temporal lobe, and frontal lobe
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demonstrate on the
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diffusion-weighted scan and ADC maps
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affecting medial frontal lobe,
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parietal lobe,
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portions of occipital lobe, and even the temporal lobe
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from the vascular complications of herniation.
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