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Training Collections
Library Memberships
On-demand course library with video lectures, expert case reviews, and more
Fellowship Certificate™ Programs
Practice-focused training programs designed to help you gain experience in a specific subspecialty area.
Ultimate Learning Pass
Unlock access to our full Course Library and all self-paced Fellowships.
Noon Conference (Free)
Get access to free live lectures, every week, from top radiologists.
Case of the Week (Free)
Get a free weekly case delivered right to your inbox.
Dr. Resnick's MSK Conference
Learn directly from the MSK Master himself.
Lower Extremities MRI Conference
Musculoskeletal Imaging
Emergency Imaging
PET Imaging
Pediatric Imaging
For Training Programs
Supplement your training program with case-based learning for residents, registrars, fellows, and more.
For Private Practices
Upskill in high growth, advanced imaging areas.
Emergency Call Prep
Prepare trainees to be on call for the emergency department with this specialized training series.
2 topics, 7 min.
8 topics, 32 min.
16 topics, 1 hr. 11 min.
Cerebellar Hematoma with Tonsillar Hernia
3 m.Intraventricular Hemorrhage
5 m.Parenchymal and Subdural Hematoma
5 m.Retroclival Subdural Hematoma
3 m.Acute on Chronic Subdural Hematoma
3 m.Midline Shift
4 m.Recurrent Subdural Hematoma
4 m.Subarachnoid Hemorrhage
5 m.Follow-up Imaging of Brain Trauma
6 m.Venous Epidural Hematoma
3 m.Venous Sinus vs. Arterial Epidural Hematomas
7 m.Evolution of Epidural and Subdural Hematomas
4 m.Diffuse Axonal Injury
9 m.Lucid Interval in Epidural Hematomas
3 m.Brainstem Hemorrhage
8 m.Pediatric Skull Fractures
7 m.6 topics, 28 min.
4 topics, 18 min.
14 topics, 1 hr. 6 min.
Secondary Traumatic Injuries
2 m.Traumatic Intracranial Dissection
5 m.Vertebral Artery Dissection, Pseudoaneurysm
6 m.Arteriographic Evaluation of Dissection
4 m.Traumatic Dissection of the MCA
4 m.Overview of Brain Herniation Types
5 m.Mechanisms of Brain Herniations
9 m.Herniation resulting in Infarction
9 m.Acute Hemorrhage on MRI
6 m.Subacute Hematoma on MRI
7 m.Chronic Hematoma on MRI
8 m.Hyperacute Hematoma on MRI
2 m.CT of Blood
3 m.Brain Trauma Summary
3 m.0:00
I would be remiss if I didn't mention the marker for
0:04
hyperacute hematoma, and that is oxyhemoglobin.
0:08
Oxyhemoglobin is diamagnetic.
0:11
By that, we mean that it has no characteristics of Proton
0:15
Relaxation Enhancement, nor proton-electron dipole interaction.
0:20
Oxyhemoglobin effectively looks like water,
0:23
and therefore, is dark on a T1-weighted scan
0:25
and bright on a T2-weighted scan.
0:28
It is incredibly uncommon to see oxyhemoglobin on MRI.
0:33
Because of the rapid conversion of
0:35
oxyhemoglobin to deoxyhemoglobin,
0:38
we usually see acute hemorrhages as dark on T1
0:42
and dark on T2 because of the proton relaxation enhancement
0:46
of deoxyhemoglobin.
0:48
Remember, however,
0:49
that that proton relaxation enhancement characteristic is
0:53
field strength-dependent,
0:55
and therefore, at 1.5T and 3T,
0:59
we see the conversion of oxyhemoglobin to deoxyhemoglobin
1:03
relatively easily,
1:04
and therefore, hemorrhages are rarely
1:07
seen as dark on T1 and bright on T2.
1:10
However, if you are using a low field strength magnet,
1:15
which is less sensitive to the conversion
1:17
of oxyhemoglobin to deoxyhemoglobin,
1:20
you may get fooled by seeing something that looks like edema
1:25
but is actually a hyperacute hematoma
1:28
dominated by oxyhemoglobin.
1:31
So, this is a potential pitfall at low field strength.
1:34
You see something,
1:35
it looks just like edema, not a hematoma,
1:39
because you are less sensitive to the presence of
1:42
deoxyhemoglobin than at high field strength.
Interactive Transcript
0:00
I would be remiss if I didn't mention the marker for
0:04
hyperacute hematoma, and that is oxyhemoglobin.
0:08
Oxyhemoglobin is diamagnetic.
0:11
By that, we mean that it has no characteristics of Proton
0:15
Relaxation Enhancement, nor proton-electron dipole interaction.
0:20
Oxyhemoglobin effectively looks like water,
0:23
and therefore, is dark on a T1-weighted scan
0:25
and bright on a T2-weighted scan.
0:28
It is incredibly uncommon to see oxyhemoglobin on MRI.
0:33
Because of the rapid conversion of
0:35
oxyhemoglobin to deoxyhemoglobin,
0:38
we usually see acute hemorrhages as dark on T1
0:42
and dark on T2 because of the proton relaxation enhancement
0:46
of deoxyhemoglobin.
0:48
Remember, however,
0:49
that that proton relaxation enhancement characteristic is
0:53
field strength-dependent,
0:55
and therefore, at 1.5T and 3T,
0:59
we see the conversion of oxyhemoglobin to deoxyhemoglobin
1:03
relatively easily,
1:04
and therefore, hemorrhages are rarely
1:07
seen as dark on T1 and bright on T2.
1:10
However, if you are using a low field strength magnet,
1:15
which is less sensitive to the conversion
1:17
of oxyhemoglobin to deoxyhemoglobin,
1:20
you may get fooled by seeing something that looks like edema
1:25
but is actually a hyperacute hematoma
1:28
dominated by oxyhemoglobin.
1:31
So, this is a potential pitfall at low field strength.
1:34
You see something,
1:35
it looks just like edema, not a hematoma,
1:39
because you are less sensitive to the presence of
1:42
deoxyhemoglobin than at high field strength.
Report
Description
Faculty
David M Yousem, MD, MBA
Professor of Radiology, Vice Chairman and Associate Dean
Johns Hopkins University
Tags
Vascular
Trauma
Physics and Basic Science
Neuroradiology
MRI
Hematologic
Emergency
Brain
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