Interactive Transcript
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Here is a case that modern-day pediatric
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radiologists even miss quite frequently.
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So why do I say modern-day?
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Because typically in the old days, a child
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would come in with foot pain, they would get X
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rays, X-rays would be called negative, and the
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next step would be getting a bone scan, right?
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And when you get a bone scan for this
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condition, it lights up like crazy.
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Nowadays, we don't do very
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many bone scans anymore.
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So oftentimes, we call this radiograph
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negative, the kid goes home, continues to have
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bone pain or foot pain, and later will come
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in, uh, get repeat X-rays or, or get an MRI.
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So an MRI will reveal the abnormality,
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will reveal the abnormality, but you
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can, you can capture what's going
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on actually in the plain films.
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So here are two views of a toddler's
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lower extremity, particularly the foot.
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Here is an AP view and here is an oblique view.
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What's the abnormality?
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I'm going to point it out to you
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and hopefully I can convince you.
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If you look at your tarsal
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bones, here is your cuboid,
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here is one of your cuneiforms,
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here is your talus, sorry, your
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calcaneus, and here is your talus.
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Look at the density of those bones, right?
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The density of this bone pretty much matches
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the density of this bone, of this bone.
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And this part of the cuboid bone, if you
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look very, very carefully, you notice
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that this part right here is a little
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brighter, a little bit more sclerotic.
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Look on the oblique view, again, look at the
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density here, look at the density on this part
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of the bone, but look at the density over here.
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It's a little bit too bright, okay?
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So what has happened?
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And I'll give you the typical history
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on this kid and any kid who has
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this particular type of process.
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This child had a fracture of
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the tibia several months ago.
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That fracture was treated with a
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cast, and the patient was immobilized.
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The fracture healed and the kid was
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allowed to go play again, run around.
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Well, during that time when
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the kid was immobilized,
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he was not using his lower extremity, so
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that meant the bones became very osteopenic.
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They became weak because the
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calcifications were being leached.
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As soon as he started playing again,
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this bone right here, which is your
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calcaneus, banged up against the talus.
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And because he was predisposed
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by that osteopenia, it caused an
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impaction fracture of the cuboid.
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So this is what an impaction
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fracture of a cuboid looks like.
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Now, you may say, well, it looks
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like it's subacute or not acute.
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I agree with you.
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It's not completely acute because you
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already see a little bit of sclerosis.
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That's the healing process.
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Well, then you go, why don't
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I see a periosteal reaction?
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Because that's how bony fractures should heal.
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You remember that tarsal and carpal bones
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don't undergo ossification by periosteal
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formation because they don't have a periosteum.
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They undergo ossification endochondrally.
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So in other words, calcification
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happens from the inside out.
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It's the cartilage itself that becomes ossified.
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The periosteum doesn't cause
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any cortical formation.
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So that's why when you have
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healing of bones that don't have a
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periosteum, all you see is sclerosis.
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You don't see, quote,
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unquote, periosteal reaction.
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So this is a great example of a cuboid impaction
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fracture, and the history is so important.
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Oftentimes, like I said, these kids
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have an injury that happened a few weeks
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ago that caused them to be immobilized.
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Something very similar can actually also
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happen in the carpal bones of the hands if
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they've had a fracture in the radius or ulna.
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They won't use it for a while, and
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then, and then they will use it
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after the cast has been taken off.
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Maybe they're doing handstands, maybe
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they're a gymnast, and you'll develop
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these impaction-type fractures.
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In the carpal bones also.
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