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All right, so now that we've talked about Crohn's and

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a little bit about how we perform enterography and

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the anatomy, we get to talk about the pathology in

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Crohn's and what we typically see for these patients.

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And so here's a pretty classic, typical case

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of active Crohn's disease and what we see.

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And so when we approach these cases, I'm going

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to give you a quick overview about how I talk

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about it, and then we'll talk in depth about

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all the individual findings and how to discuss

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those findings, what's important to talk about,

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and how much detail that I go into and how much

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detailed experts recommend people put into reports.

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And so here's a case of Crohn's with

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active terminal ileal and sickle disease.

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And what you see on each sequence is different.

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So for our T2 sequence, what we

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see is that the wall is thickened.

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We see there's some edema that we want to

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verify with our fat-saturated sequences.

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To make sure it's not fat, but it does look

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brighter than the adjacent muscle.

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So we're thinking that's edema.

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And, um, we also see that there's some

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abnormal signal in the fat surrounding.

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This is a fat-saturated sequence.

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The fat saturation isn't great peripherally,

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but it's pretty good in the abdomen.

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So I think this, this works and

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you can see that is true edema.

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The true FISP isn't as critical in this case

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because we have good quality T2 images, but

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we see the same findings on our true FISP

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or steady-state precession sequence.

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On the pre-contrast, we can kind of get a sense

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that that wall is thickened, but then as we move

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to the post-contrast, it really jumps out at us

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that this, this is an abnormal segment of bowel.

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So we see a lot of inflammation extending

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up into the ascending colon down

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through the TI into that distal ileum.

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And so that hyperenhancement on the

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early phase tells us that this looks to

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be pretty active inflammatory changes.

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When we look at our axial sequences,

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we see the same, same findings.

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Again, we want to compare that to the

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adjacent bowel and see that it is clearly

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hyperenhancing inflammatory change.

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And so that's the general findings that we see that

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make up the classic hallmarks of identifying Crohn's

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disease, but once we've identified the disease, we do

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want to talk in pretty good detail about the different

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findings because each finding that we see can have

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different implications for whether it's a chronic

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disease and can have implications for how severe the

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disease is and what kind of management is needed.

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So for the next piece, we're going to talk individually

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about the different types of findings, how to talk

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about them, and what to be looking for when we do that.