Interactive Transcript
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As I mentioned,
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the secondary complications of traumatic brain injury are
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more likely to cause permanent damage
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than those of the primary injury.
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So far, we've looked at some of the vascular complications,
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secondary complications of traumatic brain injury,
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demonstrating dissections, occluded vessels,
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as well as pseudo aneurysms,
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with the potential for embolic infarcts,
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secondary to the pseudo aneurysm.
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Now, I'd like to talk to you a little bit about herniations.
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Again,
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this is usually not what is occurring at the initial time of
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the insult with the traumatic event is usually something
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that we see over the course of time with
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delay after the initial event.
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The different types of hernia depend upon where the
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patient's lesion is and these lesions that I refer to may
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be interparenchymal. That is a hematoma or contusion.
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Or they may be extra-axial.
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That is a subdural hematoma or an epidural hematoma.
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Both of them can cause enough mass effect that leads to
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herniation. Now, when we refer to the right left shift,
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that can occur with an extra-axial collection.
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In this case, an isodense subdural hematoma,
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we usually refer to the degree of displacement
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from the midline of the septum pellucidum.
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However,
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simple displacement from the midline does not necessarily
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lead to or qualify for subfalcine herniation.
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By subfalcine herniation,
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we mean that the patient has brain tissue which
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is crossing over from, in this case,
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the right side to the left side under the falx cerebri.
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And this is the free edge of the falx cerebri.
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So why?
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While normally we can have a degree of midline shift without
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brain tissue herniating over when we see the brain tissue
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actually crossing from right to left, in this instance,
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that's where we refer to the term subfalcine herniation.
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So although midline shift is concurrent
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with subfalcine herniation,
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it does not complete the qualifications
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for subfalcine herniation.
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Subfalcine herniation is usually due to mass
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effect in the superior portion of the brain,
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not the inferior portion of the brain,
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and in the supratentorial portion of the brain,
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certainly not the infratentorial posterior fossa
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of the brain. In a similar fashion,
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we have a degree of uncal herniation.
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By uncal herniation,
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we mean that the medial temporal lobe
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extends into the cistern and will.
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Will lead to effacement of the cisterns
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around the brain stem.
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So that would be uncal herniation when that uncus or when
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the amygdala or when the medial temporal lobe
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then crosses downward the tentorial edge.
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And you see brain tissue below the tentorial edge,
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for example, in a coronal image,
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then we would say that the patient had transtentorial uncal
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herniation or transtentorial temporal lobe herniation
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or transtentorial herniation. So again,
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this is a degree midline shift to subfalcine
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herniation uncal deviation,
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uncal herniation to transtentorial herniation as it goes
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downward. These are, again, supratentorial herniations.
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When we refer to cerebellar herniations,
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we're obviously talking about the posterior fossa.
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And again, this may be both an intraparenchymal.
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Hemorrhage or it may be extra-axial as in an
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epidural hematoma or a subdural hematoma.
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So again,
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posterior fossa for upward and downward
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cerebellar herniation.
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A lesion in the central aspect of the cerebellum may lead
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to this cerebellar tissue herniating upwards through the
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tentorial incisura the opening of the tentorium superiorly
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in that place it will compress the midbrain and efface
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the perimesencephalic cisterns. At the same time,
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1 may have downward cerebellar herniation what
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we usually refer to as tonsillar herniation.
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And that's when the tonsils are seen at the foramen magnum
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herniating downward through the foramen magnum.
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This then causes compression on the cervical medullary
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junction and the medulla and can lead
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to cardiorespiratory arrest.
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