Interactive Transcript
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Okay, well, I'm out here for you.
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It is not a pride-swallowing siege,
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and I want to share with you a diagram
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perhaps you've seen across the globe:
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the staging diagram for hip labral pathology.
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Not unlike, but different than the
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grading system used for menisci of the
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knee, for the labrum of the meniscus of the knee have
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some common traits.
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One of those common traits
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is intra-meniscal signal.
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The intra-meniscal signal in the knee, I'll draw
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a knee meniscus, frequently looks like this.
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Looks like two little bunny ears coming down.
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Just kind of fades away and stops
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in the center of the meniscus.
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That doesn't occur with
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any frequency in the hip.
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It's more of an amorphous signal intensity
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in the center of the meniscus that I've
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colored in in this type A meniscus,
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which is more triangular, and I've colored it
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in in the type B meniscus, which is more
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round or meniscoid, the less common shape.
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Now, what can you attribute this signal to?
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Well, it is an accumulation of water that occurs
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in the meniscus through diffusion along collagen
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bundles, and it is present in almost every adult
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and is an asymptomatic finding, unless it is
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bright on T2 associated with meniscal expansion.
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So I frequently will either ignore it,
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or comment on it and then
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dismiss it as an incidental finding.
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Let's go to stage two.
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We have two variations of the meniscus:
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one triangular, one round.
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Remember, you can also have a flat meniscus,
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which in my experience is uncommon.
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And an absent meniscus, which is very rare.
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In my experience, less than 1%.
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Literature says 3%.
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So here we've got a partial tear, and it looks
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like you've come in with your scissors and made
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a cut and then your hand got a little trembly
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and you made another cut right next to it.
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It is not a smooth, round area of shallow
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signal alteration, but rather somewhat serrated.
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Now, what do you do with this?
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Is it a tear?
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Yes, it's a tear.
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Especially if it's in the superior
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or anterior superior quadrant.
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But a lot of elderly people have these,
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whether it's in the type A or type B meniscus.
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So what I do with it is,
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I use my common sense.
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I use my neocortex to figure out
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what else is going on.
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Is there an effusion?
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Where are the symptoms?
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Are there hip and groin symptoms?
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Do I have a cyst?
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Do I have chondromalacia?
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Do I have swelling in the region?
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Is the meniscus displaced?
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I use a lot of secondary signs.
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And what type of patient is it?
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If it's a 78-year-old man, I am certainly
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not going to go out and preach that
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this patient has a traumatic tear.
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I'm going to preach that the patient
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has a chronic progressive labral tear
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associated with some of the other
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degenerative changes that I might see.
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If it's a 30-year-old that's playing
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tennis, that has a bump cyst complex,
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an asphericity of the hip, and other signs of
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CAMP-type impingement. I'm going to be more
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aggressive with this partial thickness tear,
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looking again for the other indirect signs
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that the tear is indeed contributing,
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perhaps with an erosion or abrasion of the
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hyaline cartilage, to the patient's symptoms.
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So not only am I going to use the secondary
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signs, I'm going to put it in the context
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of the patient's age and what they do.
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I'm going to synthesize.
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On the other hand, if it's a 15-year-old and
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they are running cross-country, this is highly
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likely to be symptomatic even though they may
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have only mild changes of dysplasia or mild
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changes of CAMP-type impingement with asphericity.
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So very active individuals, people
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that are jumping, running, hurdling,
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are more likely to be symptomatic
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with this kind of lesion.
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So I am much less aggressive with
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the 50-year-old, 60-year-old,
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and 70-year-old in describing the tear.
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That's not to say I don't call it a tear.
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That's to say that I'm not going to draw
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as much attention to it so that the tear
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becomes repaired or surgerized in that
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55 or 60-year-old with DJD, unless there are
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other findings that specifically point to
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this labrum as the cause of the symptoms.
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When we get to the bottom, it's easy.
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You've got a through-and-through tear,
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the labrum is displaced, you'll often have secondary
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signs like the capsule being stripped away off
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the periosteum, it'll be separated this way.
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You'll have areas of hyaline ulceration,
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hyaline penetration, there'll be a large
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effusion, there'll frequently be a history
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of trauma, the patient will have groin
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pain, and many other supportive signs
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that tell you that this is the one that is
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causing the patient's clinical syndrome.
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Let's move on, shall we?
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