Interactive Transcript
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I do want to keep you up to date with some of the
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literature that has come out of Johns Hopkins about
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traumatic injuries to the blood vessels in the neck.
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This is an article that was written by one
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of my research fellows, Farzan Sherbach.
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And she looked at a multi-institute study that
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included the University of Pennsylvania
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and University of Texas,
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San Antonio and looked at the value of emergent
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neurovascular imaging when the clinical
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indication is seatbelt injury.
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So in those individuals who have motor vehicle
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collision and have to-and-fro back-and-forth injury,
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quite often you'll see discoloration across
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the neck from the seatbelt,
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the lap belt that's coming across and protecting the
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individual in addition to obviously the airbag.
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So we looked at five institutions and we looked at
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the incidence of vascular injury when the only
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etiology, the only indication was seatbelt injury.
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In other words, there were no fractures,
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there was no stroke,
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there wasn't any other findings on the imaging other
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than a clinical history of seatbelt injury.
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And when we looked at 535 adults and
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32 children in this time frame,
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what we found was only 0.5% of the patients who had
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CT scan CTAs done for seatbelt injury actually
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had a vascular injury and of those,
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none of them had any neurological sequelae of it.
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So in the isolated situation where the patient
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doesn't have other imaging findings of fractures
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or hematomas or dissections or whatever,
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in that situation where you only have the
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lap belt demarcation discoloration,
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the value of doing CTA is pretty low.
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This is a case of a patient who did have a motor
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vehicle collision and presented
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with Horner syndrome.
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This is an example of what is a pseudoaneurysm seen
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on MRI scan with variable signal intensity.
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And this is typical of pseudoaneurysms
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being evaluated with an MRI scan.
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You can have all different types of signal intensity
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characteristics because of the different stages
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of hematoma. You can have deoxyhemoglobin,
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which is dark on T2 and dark on T1.
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You can have intracellular methemoglobin,
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which is bright on T1 and dark on T2.
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You can have extracellular methemoglobin,
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which is bright on T1 and bright on T2.
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And very rarely you can have calcification in the
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wall of an old pseudoaneurysm which obviously
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is going to be dark on T1 and dark on T2.
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In this case,
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we were looking at this and saying, well,
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could this be a mass or an enhancing
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tumor schwannoma?
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See that the parapharyngeal fat is displaced
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anteriorly and when you look at
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the conventional arteriogram,
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what you're seeing is that swirling contrast
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within the lumen of the pseudoaneurysm,
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which accounts for the mixed density
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and or intensity on MRI scan.
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So remember that traumatic aneurysm is Biffl stage
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three. And if you also have luminal narrowing,
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which is seen proximally here,
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if it's less than 25%, Biffl stage one,
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greater than 25% Biffl stage two.
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So if you look at these patients who have
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the injury to the intima media,
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that is what is going to lead
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to the pseudoaneurysm.
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And that can lead to subsequent either
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partial thrombosis or narrowing,
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again using the Biffl staging.
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So if it's completely occluded,
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it would be grade four.
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Even with a pseudoaneurysm,
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the risk of stroke with a pseudo
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aneurysm is about ten to 20%.
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Remember that I had said previously that most people
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report five to 30% for strokes associated with
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dissections. 25% of pseudoaneurysms will enlarge,
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25% will resolve on their own
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and 50% will simply persist.
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These are usually found below the skull base.
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And remember that dissections are more
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common in the vertebral arteries,
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but pseudoaneurysms are more common
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in the carotid arteries.
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