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Value of Neurovascular Imaging for Seat Belt Injury

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I do want to keep you up to date with some of the

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literature that has come out of Johns Hopkins about

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traumatic injuries to the blood vessels in the neck.

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This is an article that was written by one

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of my research fellows, Farzan Sherbach.

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And she looked at a multi-institute study that

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included the University of Pennsylvania

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and University of Texas,

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San Antonio and looked at the value of emergent

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neurovascular imaging when the clinical

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indication is seatbelt injury.

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So in those individuals who have motor vehicle

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collision and have to-and-fro back-and-forth injury,

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quite often you'll see discoloration across

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the neck from the seatbelt,

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the lap belt that's coming across and protecting the

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individual in addition to obviously the airbag.

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So we looked at five institutions and we looked at

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the incidence of vascular injury when the only

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etiology, the only indication was seatbelt injury.

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In other words, there were no fractures,

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there was no stroke,

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there wasn't any other findings on the imaging other

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than a clinical history of seatbelt injury.

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And when we looked at 535 adults and

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32 children in this time frame,

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what we found was only 0.5% of the patients who had

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CT scan CTAs done for seatbelt injury actually

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had a vascular injury and of those,

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none of them had any neurological sequelae of it.

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So in the isolated situation where the patient

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doesn't have other imaging findings of fractures

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or hematomas or dissections or whatever,

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in that situation where you only have the

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lap belt demarcation discoloration,

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the value of doing CTA is pretty low.

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This is a case of a patient who did have a motor

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vehicle collision and presented

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with Horner syndrome.

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This is an example of what is a pseudoaneurysm seen

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on MRI scan with variable signal intensity.

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And this is typical of pseudoaneurysms

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being evaluated with an MRI scan.

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You can have all different types of signal intensity

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characteristics because of the different stages

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of hematoma. You can have deoxyhemoglobin,

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which is dark on T2 and dark on T1.

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You can have intracellular methemoglobin,

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which is bright on T1 and dark on T2.

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You can have extracellular methemoglobin,

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which is bright on T1 and bright on T2.

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And very rarely you can have calcification in the

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wall of an old pseudoaneurysm which obviously

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is going to be dark on T1 and dark on T2.

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In this case,

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we were looking at this and saying, well,

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could this be a mass or an enhancing

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tumor schwannoma?

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See that the parapharyngeal fat is displaced

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anteriorly and when you look at

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the conventional arteriogram,

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what you're seeing is that swirling contrast

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within the lumen of the pseudoaneurysm,

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which accounts for the mixed density

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and or intensity on MRI scan.

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So remember that traumatic aneurysm is Biffl stage

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three. And if you also have luminal narrowing,

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which is seen proximally here,

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if it's less than 25%, Biffl stage one,

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greater than 25% Biffl stage two.

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So if you look at these patients who have

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the injury to the intima media,

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that is what is going to lead

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to the pseudoaneurysm.

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And that can lead to subsequent either

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partial thrombosis or narrowing,

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again using the Biffl staging.

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So if it's completely occluded,

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it would be grade four.

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Even with a pseudoaneurysm,

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the risk of stroke with a pseudo

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aneurysm is about ten to 20%.

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Remember that I had said previously that most people

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report five to 30% for strokes associated with

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dissections. 25% of pseudoaneurysms will enlarge,

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25% will resolve on their own

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and 50% will simply persist.

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These are usually found below the skull base.

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And remember that dissections are more

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common in the vertebral arteries,

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but pseudoaneurysms are more common

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in the carotid arteries.

Report

Description

Faculty

David M Yousem, MD, MBA

Professor of Radiology, Vice Chairman and Associate Dean

Johns Hopkins University

Tags

Vascular Imaging

Vascular

Trauma

Neuroradiology

Neuro

MRI

Head and Neck

CT

Angiography

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