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Case Review: 22 Year Old Male with Knee Pain. Had Prior ACL Repair

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This is a 22-year-old man with knee pain.

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He's had prior ACL surgery.

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They can either perform this surgery

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using some hamstring,

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they can use patella,

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or they can use cadaveric tendon.

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And this patient,

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you can tell right away from the axial projection,

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there's a defect in the anterior patella.

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So they have used bone, tendon bone,

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the other piece of bone coming from the tibia or

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an autograft to make this new ACl for this

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patient. Now, there are some advantages of this,

0:34

namely the longevity of the graft as opposed to,

0:38

say, cadaver, which may last a little less time,

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but the recovery period is a lot easier when

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you use cadaveric, non native acl.

0:46

As I scroll up and down,

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this patient's got a small effusion and quite a

0:51

bit of scarring anteriorly or low signal

0:53

intensity in the axial projection.

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And you can see the graft as this linear structure

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along the lateral aspect of the femoral notch.

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And the femoral notch is fairly generous in its

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breadth or diameter from side to side or width.

1:09

let's look at the sagittal projection.

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And one of the things I do in somebody that's had

1:17

an autographed is I look at the character

1:21

of the patellar mechanism,

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because when you harvest tendon from the patella

1:26

and then take a strip of tendon down to the tibia,

1:29

the patella will often overgrow,

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it'll get too big,

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and sometimes that leads to patellar tendinopathy.

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That is one of the complications of using

1:39

a bone tendon, bone autographed,

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or a piece of patellar tendon.

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And this patient actually has it.

1:44

They weren't born with a pointy inferior patella.

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Now, that may not be problematic today,

1:50

but that can be problematic in the future.

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And that's one of the reasons why I personally

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would go for a cadaveric graft at my age,

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and many people would.

2:00

The quadriceps tendon usually

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isn't affected in patients that are harvested

2:07

because the harvesting usually occurs right

2:09

about here and then the strip comes down.

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The other thing I like to look at is the character

2:15

of the patellar tendon. And admittedly,

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it's a little hazy. It's a little thickened,

2:20

but there's no focal tear on

2:22

the water weighted image.

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There's a little bright signal

2:25

right under that spur. Again,

2:27

the patient isn't symptomatic there yet.

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There's also a little bit of signal intensity

2:32

at the patellar tendon insertion.

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And I would comment on that

2:35

in the body of the report,

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and there's extensive scarring at the site

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where the clinician has entered the knee.

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Now,

2:46

another caveat when you're looking at patellar

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harvesting is to look at the length of the

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patellar tendon, because it can contract.

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And this is one of the causes of patella baja.

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In other words,

3:00

a patella that's pulled down from scarring

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of the infra patellar tendon.

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And this one is a little bit short.

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I like to see the length of the patella and the

3:11

length of the patellar tendon about one to one.

3:13

And just eyeballing it,

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I think you would all agree that the patella

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is substantially longer than the tendon.

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So you would say that there is a tendency

3:24

for patella baja in this patient.

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But let's move on to our graft.

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Now, there are a few ways to look at the graft.

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the conventional way would just simply be to do

3:35

your standard orthogonal views and hope you catch

3:38

the graft correctly, which, by the way, we did.

3:41

Another way to do it would be to look at the graft

3:44

and then angle with the graft distally, and then,

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if necessary,

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make a second angle along the femoral tunnel so

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that you can see the intra tunnel portion or

3:56

the intraskeletal portion of the graft.

3:58

You only need to really do that in complex cases

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where there's definite graft laxity.

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This patient had pain but not locking

4:08

and not any known instability.

4:10

Or you may have to do it as part of an

4:12

add on or bring back examination.

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Another technique that you might use,

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especially in difficult cases where there's

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extensive swelling, scarring,

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and the graft is hard to really discern,

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is you might take an axial oblique that's

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perpendicular to the graft, like that,

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so that you can see the graft in cross section and

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assess its overall diameter from top to bottom.

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Now, let's take away this line,

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and let's look at where our

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graph should be placed.

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Our graft should be placed as close as humanly

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possible to this line right here,

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which is the posterior femoral line.

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So we bring that line down,

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and then at the intersection between

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that line and Blumenstadt's line,

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which is going to be right here,

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that's where as close to the cortex as possible,

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the femoral tunnel entrance should be drilled,

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which takes a fair amount of courage and talent,

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because you are right subcortical,

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you're right next to the cortex.

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So a lot of nervous nellies may take that hole

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and put it too far forward or too far down.

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And that is a lot worse than moving

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the tibial tunnel around.

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This is the more important of the tunnel

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entrances. Now, the tibial tunnel,

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I give a fair amount of latitude to that tunnel.

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I like to see the tunnel a little further back.

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If the roof is very vertical,

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I like to see it forward a bit.

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If the roof were very horizontal.

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What do I mean by a horizontal roof?

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It goes this way, our roof is going this way.

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A vertical roof would go this way.

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So I might put my tibial tunnel back a little bit

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with the vertical roofs so it doesn't get bumped.

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Now,

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another thing I also like to do in looking at my

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graft is look at this distance right here.

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I like to have about a centimeter between the

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graft and the femur when the patient is lying

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on their back. Now, think about this.

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If a patient is lying on their back and this piece

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of bone is already touching the

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trampoline of the graft,

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imagine what happens to the graft

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when they're standing up. Bang.

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It gets slammed every time they jump or run.

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So that is a little pearl,

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a little nugget that is not often discussed that

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you can take away from this brief talk.

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Now,

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I'm sure a lot of you are honing in

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on this structure right here,

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which we'll discuss in a minute,

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but let's just go back to the

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tibial tunnel for a minute.

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I usually take the center of the PCL

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and the center of the tunnel,

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and I like it to be about 2 cm.

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Beta has already stated there's a little bit of

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variability and flexibility here depending

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upon the femoral roof. Now,

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let's go to this structure

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that is a round mass anterior to the Acl.

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It's ill defined on the T1 because it's buried in

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this tissue, which is synovium, and this tissue,

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which is proteinaceous fluid, and this tissue,

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which is synovium. And this is synovium.

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So synovium against the background of proteous

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fluid and synovium makes it

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a little hard to discern,

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a lot easier to discern on the sagittal water

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weighted image about the T2 weighted image.

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Let's have a look.

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Let's go to the T2 weighted image.

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And now it's really easy.

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Now you can see the mixed heterogeneous character

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of this mass, which is known as a cyclops lesion.

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So hopefully you'll keep an eye out for the

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cyclops lesion because it looks

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a little bit like an eye,

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and sometimes it even has a pupil in the middle of

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it. This one a bit more amorphous and ill defined,

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but rather large.

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This is a definite cause of pain.

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It restricts range of motion.

7:59

But despite all that,

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let's look at the position of the tibia with the

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patient on their back. Is the tibia translated?

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So is there passive instability?

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Right. We're not examining this patient.

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So we're not examining this patient

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for dynamic instability,

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but we can assess for passive instability with

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indirect signs like anterior displacement of the

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tibia relative to the back of the femur.

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I like these to line up about 5 mm

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within each other vertically.

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So if I take a line at the back

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of the femoral cortex,

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I want to be within 5 back of the tibia

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on either side, and I certainly am.

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So there is no passive anterior

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tibial translation.

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And I take the sagittal line or the lateral line,

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the line that I just drew,

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and I draw it where the tibia is furthest back.

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So you can see they're almost congruent.

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So this patient does not have passive

9:01

anterior tibial translation.

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It's highly unlikely that they'll have dynamic

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anterior tibial translation as well.

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But there's going to be some micro instability and

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friction going on in the notch because that's

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how you get the cyclops lesion.

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Let's look at a few other findings that

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may catch your eye and define them.

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One finding that may catch your eye is this,

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this vertical signal in the

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posteromedial meniscus.

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How long will that vertical signal persist there?

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Forever.

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How do you know that's not a sign of a new recent

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pivot shift injury? Well, here's one easy way.

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The history. There's no new trauma.

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Why not use what you've been given?

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So it wouldn't make sense to have a pivot shift

9:56

injury without a history of trauma.

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Second,

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if this was a new vertical longitudinal

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tear from another new pivot shift,

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the ACl wouldn't look so good,

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and you'd have some signal above and

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below the area of abnormality.

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No,

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this is a tear with granulation

10:16

tissue that's healing. Now,

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you might say what do you do about it?

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Usually nothing.

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Sometimes if it's very long,

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it's got a lot of length.

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A physician will stitch it,

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but that has become less common now,

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as we've learned that red, red zone, outer third,

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vertical longitudinal tears that are not gapped

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will heal on their own, scar on their own.

10:42

And you certainly can't go in here and take

10:44

out the outer third of the meniscus.

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That's a disaster.

10:48

Let's look at the posterolateral meniscus.

10:50

Do we have something similar?

10:51

Not really,

10:52

but we do have a little bit of swelling at the

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meniscocapsular junction from the original

10:57

pivot shift injury. It's kind of bland.

11:00

It's not very swollen.

11:02

There's no bone injury around it.

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So there has not been a new pivot shift phenomena.

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That being said,

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there are biomechanics here consisting of micro

11:13

instability that are a this graft

11:16

that are producing cinnavitis,

11:18

that are resulting in a cyclops

11:20

lesion and an effusion.

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So this patient has impingement syndrome

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of their ACL graft with a large,

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obvious cyclops lesion best depicted on the T2

11:31

weighted image. Let's do another one, shall we?

Report

Description

Faculty

Stephen J Pomeranz, MD

Chief Medical Officer, ProScan Imaging. Founder, MRI Online

ProScan Imaging

Tags

Trauma

Syndromes

Musculoskeletal (MSK)

MRI

Knee

Acquired/Developmental

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