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Case Review: 90 Year Old Female Patient, No History of Trauma, Now Has Swelling

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An older adult woman who complains

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of knee swelling,

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but no discrete history of trauma.

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Let's scroll the axial fat-suppressed

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water emphasized image first,

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and that is a swollen extremity.

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There's swelling everywhere.

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There's swelling in the joint.

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There's swelling around the joint.

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And while we're here,

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let's take a quick gander at the ACL.

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It's nice and straight and linear.

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It's intact.

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The PCL,

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it's nice and round and robust,

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and we can follow it from bottom to top.

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It's intact.

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So we probably have to look elsewhere to determine

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what is causing such a severe pattern of swelling.

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When you look inside the effusion,

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you do not see a blood fluid level.

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You do not see synovial hypertrophy.

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Therefore,

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a rheumatologic explanation isn't very likely.

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You don't see clots and you also don't

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see bone erosion or destruction,

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as you might in aseptic arthritis.

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So right now, we have a little bit of an enigma.

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While you're scrolling through the axial,

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you should always take note of the popliteal

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fossil because patients can develop clots from

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swelling like this due to encroachment

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on the popliteal vein,

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and the flow voids are not very prominent.

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So that should be a potential area of concern.

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But that's not why we're here,

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although I guess it could be with a swollen lower

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extremity. But let's keep going, shall we?

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I've got two sagittals up,

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and the goal of this review is to

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take you through the corners,

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specifically the posteromedial corner.

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So I'm going to stop at the posterolateral corner

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for a minute. And, yes, there is a meniscus tear.

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It's chronic looking, it's complex.

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That's going to go in the conclusion

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with the word chronicity.

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And there is class four chondromalacia present

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both on the tibial side and the femoral side

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surrounding this complex, macerated meniscus.

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There is also a popliteus tendon with

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a popliteofibular ligament rupture,

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so that is blunted. And here is the arcuate,

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which should come down and attach to the tip of

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the fibula. It's just hanging down in the back,

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so the arcuate is torn, too.

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So there's a posterolateral corner injury.

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Let's keep scrolling now and

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go over to the medial side.

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Now, on the medial side,

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there's also meniscus pathology.

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There is an oblique under surface,

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chronic appearing tear. Why do I say chronic?

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Because it's not that bright.

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She's older,

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and she also hasn't had a history of trauma.

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What does older have to do with it?

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When you get older, you get more signal,

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you get more incidental degeneration

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and chronic tears in the meniscus.

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But we're here to talk about the corners,

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so I'm going to move my image over a little bit,

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and I'm going to blow this one up.

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And what makes up the posteromedial corner?

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Well, the posterior oblique ligament of the knee,

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or the pol, the OPL,

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the oblique popliteal ligament,

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the semimembranosus and its five expansions,

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or arms,

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the medial meniscus

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and its attachments, which are very swollen.

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You can't really see an interface between the

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meniscus and any other tissue other than boggy,

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hyperintensed tissue. And finally, the capsule,

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and the meniscocapsular attachments,

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which are also obliterated by this swelling.

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So immediately we've identified that the

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meniscocapsular reflection is severely diseased.

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How about the semi membranosis?

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Wow. The semi membrinosis is hanging down.

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It's pointing towards Florida.

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It should actually point towards Texas.

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There are five expansions,

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or arms of the semi membrinosis,

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the main one being the direct deep arm.

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The second important one, which is seen here,

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is the anterior arm,

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also known as the pars reflexa,

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or reflected portion of the semimembrnosis.

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So the main arm,

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the direct arm of the semimembrosis

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has been pulled off.

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So now we have at least three components.

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There's a meniscal injury,

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although it's pretty low grade.

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There's a more serious meniscal capsuler injury.

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The main attachment of the semimembrnosis

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is pulled off. What do we have left?

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We have the OPL, the oblique popliteal ligament,

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that is not seen as a separate structure

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because it merges with the capsule.

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But let's follow the capsule down.

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It should be a clean, straight, black structure,

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and it is to right there.

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And then at that point,

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we see a spidery web of low signal intensities.

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That is the torn, retracted capsule.

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So if the capsule is torn, the OPL,

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or oblique popliteal ligament got to be torn.

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So another component of the posteromedial corner

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is torn. How about the last component, the pol,

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the posterior oblique ligament of the knee?

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It's got three components.

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It's got a superior capsuler component,

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also known as the superior arm.

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It's got a central tibial component which is the

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main component that kind of slopes back like this.

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And then it's got a distal superficial

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arm that goes a little more forward.

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Now let's look at the medial collateral ligament,

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or the tibial collateral ligament,

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which is the middle layer of the MCl,

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which is this black band right here.

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Behind it is going to be,

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and I'll pick another color.

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Behind it is going to be the more broad arcing

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pol, which kind of has this shape.

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I made it orange.

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Maybe I should have picked another color.

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Let's pick green.

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So the pol

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is behind it,

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and it kind of has sort of a

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triangular configuration.

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And you should be able to see wispy oblique fibers

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going this way from super anterior

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to postero inferior,

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and then going this way from superoposterior

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to antero inferior.

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Then these fibers will kind of come down and wrap

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around the back of the knee to merge with the

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posteromedial capsule. Now let's take it away.

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Do you see fibers with that course behind the

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tibial collateral ligament? No, you don't.

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You might see something running up and down

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right here, but after that it is a mess.

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All you see is ill defined hyperintense

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signal intensity.

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Now let's go to the coronal projection

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to seek out our pol,

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our posterior oblique ligament of the knee,

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which is one of the most important structures

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of the postural medial corner,

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along with the semimembrnosis and its expansions.

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So let's pull down our water weighted

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coronal and our T1 coronal.

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I'm going to blow them up a little bit just

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to make it a little easier for you to see.

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Make them a little lighter.

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Now, admittedly,

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the patient has some meniscal pathology,

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quite a bit of meniscal pathology,

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but that's not why we're here,

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both on the lateral side and the medial side.

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And let's scroll.

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I want you to look at the scroll for a minute,

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and I'm sure you've recognized the

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tibial collateral ligament,

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which is the middle layer of the MCl.

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layer number two.

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Now follow the tibial collateral ligament forward.

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It's a little swollen. Now follow it backwards.

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There's the meniscofemoral attachment.

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There's the menisco tibial attachment.

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This is layer number three of the MCl.

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Let's keep going back because immediately

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behind the tibial collateral ligament,

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layer two of the MCl, you should run into the.

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You should be running into it right now.

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Might be a little pol.

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That might be a little pol.

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There's the broken pol. Let me make it bigger.

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That should be a sweeping, contiguous structure.

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There should not be a gap right here.

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That is a pol tear.

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So the trick is to follow the TCl,

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tibial collateral ligament,

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or middle layer of the MCl.

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Back becomes quite attenuated, then gone.

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So it's injured here, torn there.

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It helps anchor or check the movement of the

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meniscus along with the rest of

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the posteromedial corner.

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Now,

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I put the T1 up so that you could see the

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chondromalatia and some of the skeletal changes,

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but it's much harder to recognize the ligaments on

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the T1 than it is on the water weighted image.

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So this patient has a pol tear,

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a semimembranosus avulsion,

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and a meniscocapsular ligament injury or tear.

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Therefore,

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they have lost the stabilization of the postural

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medial meniscus and what we call the brake stop

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mechanism of the postural medial meniscus.

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But she has other problems that

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I put into the conclusion,

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including chronic chondromalacia meniscal pseudo

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extrusion, chronic meniscus tears.

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I described their length, their complexity,

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their shape, et cetera,

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and I would also add a pertinent

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neg in the body of the report,

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because the injury is pretty impressive

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that the ACL and the PCL are intact.

Report

Description

Faculty

Stephen J Pomeranz, MD

Chief Medical Officer, ProScan Imaging. Founder, MRI Online

ProScan Imaging

Tags

Vascular

Syndromes

Non-infectious Inflammatory

Musculoskeletal (MSK)

MRI

Knee

Idiopathic

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